急性间歇性卟啉病并发肝细胞癌的发病机制与预防策略

R735.7; 随着遗传学、分子生物学的发展,急性间歇性卟啉病(AIP)的相关研究不断深入,其并发肝细胞癌(HCC)也日益引起关注.主要总结了AIP并发HCC的发病机制及预防研究进展.AIP并发HCC的机制包括氧化应激、p53突变、B淋巴细胞瘤-2下调、致炎细胞因子增多、铁超载等有关.其预防策略包括他汀类药物的应用、严格管理、肝移植和基因及酶替代治疗.对AIP并发HCC的发病机制及预防新进展作一综述....

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Published inLinchuang gandanbing zazhi Vol. 37; no. 5; pp. 1208 - 1211
Main Authors 曹茜茜, 任毅, 刘思敏, 杨静
Format Journal Article
LanguageChinese
Published Changchun Journal of Clinical Hepatology 01.01.2021
山西医科大学第一临床医学院,太原030001%山西医科大学第一医院内分泌科,太原030001%山西医科大学麻醉学院,太原030001
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ISSN1001-5256
2097-3497
DOI10.3969/j.issn.1001-5256.2021.05.049

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Abstract R735.7; 随着遗传学、分子生物学的发展,急性间歇性卟啉病(AIP)的相关研究不断深入,其并发肝细胞癌(HCC)也日益引起关注.主要总结了AIP并发HCC的发病机制及预防研究进展.AIP并发HCC的机制包括氧化应激、p53突变、B淋巴细胞瘤-2下调、致炎细胞因子增多、铁超载等有关.其预防策略包括他汀类药物的应用、严格管理、肝移植和基因及酶替代治疗.对AIP并发HCC的发病机制及预防新进展作一综述.
AbstractList R735.7; 随着遗传学、分子生物学的发展,急性间歇性卟啉病(AIP)的相关研究不断深入,其并发肝细胞癌(HCC)也日益引起关注.主要总结了AIP并发HCC的发病机制及预防研究进展.AIP并发HCC的机制包括氧化应激、p53突变、B淋巴细胞瘤-2下调、致炎细胞因子增多、铁超载等有关.其预防策略包括他汀类药物的应用、严格管理、肝移植和基因及酶替代治疗.对AIP并发HCC的发病机制及预防新进展作一综述.
随着遗传学、分子生物学的发展,急性间歇性卟啉病(AIP)的相关研究不断深入,其并发肝细胞癌(HCC)也日益引起关注。主要总结了AIP并发HCC的发病机制及预防研究进展。AIP并发HCC的机制包括氧化应激、p53突变、B淋巴细胞瘤-2下调、致炎细胞因子增多、铁超载等有关。其预防策略包括他汀类药物的应用、严格管理、肝移植和基因及酶替代治疗。对AIP并发HCC的发病机制及预防新进展作一综述。
Author 刘思敏
任毅
曹茜茜
杨静
AuthorAffiliation 山西医科大学第一临床医学院,太原030001%山西医科大学第一医院内分泌科,太原030001%山西医科大学麻醉学院,太原030001
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LIU Simin
YANG Jing
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山西医科大学第一临床医学院,太原030001%山西医科大学第一医院内分泌科,太原030001%山西医科大学麻醉学院,太原030001
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Snippet 随着遗传学、分子生物学的发展,急性间歇性卟啉病(AIP)的相关研究不断深入,其并发肝细胞癌(HCC)也日益引起关注。主要总结了AIP并发HCC的发病机制及预防研究进展。AIP并发HCC...
R735.7; 随着遗传学、分子生物学的发展,急性间歇性卟啉病(AIP)的相关研究不断深入,其并发肝细胞癌(HCC)也日益引起关注.主要总结了AIP并发HCC的发病机制及预防研究进展.AIP...
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