Terminal deoxynucleotidyltransferase deficiency decreases autoimmune disease in diabetes-prone nonobese diabetic mice and lupus-prone MRL-Fas(lpr) mice - Robey, Ian F; Peterson, Melissa; Horwitz, Marc S; Kono, Dwight H; Stratmann, Thomas; Theofilopoulos, Argyrios N; Sarvetnick, Nora; Teyton, Luc; Feeney, Ann J (The Journal of immunology (1950), 2004)
Keywords: Animals; Animals, Newborn; Autoimmune Diseases - genetics; Autoimmune Diseases - mortality; Autoimmune Diseases - pathology; Autoimmune Diseases - prevention & control; CD4-Positive T-Lymphocytes - immunology; CD4-Positive T-Lymphocytes - metabolism; Clone Cells; Crosses, Genetic; Diabetes Mellitus, Type 1 - genetics; Diabetes Mellitus, Type 1 - mortality; Diabetes Mellitus, Type 1 - pathology; Diabetes Mellitus, Type 1 - prevention & control; DNA Nucleotidylexotransferase - deficiency; DNA Nucleotidylexotransferase - genetics; DNA Nucleotidylexotransferase - physiology; fas Receptor - genetics; Female; Islets of Langerhans - pathology; Male; Mice; Mice, Inbred MRL lpr; Mice, Inbred NOD; Mice, Knockout; Receptors, Interleukin-2 - biosynthesis; Survival Analysis; T-Lymphocyte Subsets - immunology; T-Lymphocyte Subsets - metabolism