Human T H 9 differentiation is dependent on signal transducer and activator of transcription (STAT) 3 to restrain STAT1-mediated inhibition - Zhang, Yuan; Siegel, Andrea M; Sun, Guangping; Dimaggio, Tom; Freeman, Alexandra F; Milner, Joshua D (Journal of allergy and clinical immunology, 2019)
Keywords: Autoimmunity; Candidiasis; CD4 antigen; Cell Differentiation; Cytokines; Deoxyribonucleic acid; Dermatitis; Disease; DNA; Human subjects; Humans; Hypersensitivity (immediate); Immunoglobulin E; Infections; Inflammation; Interleukin 21; Interleukin 9; Interleukins - immunology; Lymphocytes; Lymphocytes T; Lymphoma; Mutation; Ostomy; Pathogenesis; Phenotypes; Recurrent infection; RNA-mediated interference; Scoliosis; SOCS-3 protein; Squamous cell carcinoma; Stat1 protein; STAT1 Transcription Factor - genetics; STAT1 Transcription Factor - immunology; Stat3 protein; STAT3 Transcription Factor - genetics; STAT3 Transcription Factor - immunology; T-Lymphocytes, Helper-Inducer - immunology; T-Lymphocytes, Helper-Inducer - physiology; Transcription activation; allergy; Human T(H)9; signal transducer and activator of transcription 3; signal transducer and activator of transcription 1; IL-21; anticancer