C형 및 S형 단백 결핍에 의한 급성 동맥 폐쇄

• Published in: Annals of surgical treatment and research Vol. 64; no. 3; pp. 269 - 274
• Main Authors: 정은영(Eun Young Jung), 조용필(Yong Pil Cho), 장혁재(Hyuk Jai Jang), 김지수(Jee Soo Kim), 김용호(Yong Ho Kim), 한명식(Myoung Sik Han)
• Format: Journal Article
• Language: Korean
• Published: 대한외과학회 01.03.2003
• Subjects: 일반외과학
• ISSN: 2288-6575; 2288-6796

Sudden acute arterial occlusion can result from a multitude of pathological processes. Although the appearance and the secondary effects of an acute arterial occlusion are similar regardless of the underlying cause, the treatment and prognosis are different. Therefore, establishing a correct diagnosis is crucial. Acute arterial occlusion is most frequently a complication of ischemic cardiac disease, with an atrial fibrillation occurring in most patients. An identifiable noncardiac source of the acute arterial occlusion can be found in 5～10% of patients. Howerver, in these cases, the specific source of the occlusion cannot be determined clinically or even at autopsy. Possible hypercoagulable states should be suspected and appropriately evaluated, particularly in patients with no history of antecedent occlusive disease who present with sudden arterial occlusions, or in patients with malignant disease. Protein C and protein S deficiencies are frequently described as a cause of the hypercoagulable states. We reported 3 cases of acute arterial occlusion associated with protein C and S deficiencies. (J Korean Surg Soc 2003;64:269-274) KCI Citation Count: 0