2. 動脈硬化のハイリスク病態とその分子基盤
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| Published in | 日本内科学会雑誌 Vol. 95; no. Suppl; pp. 61a - 63a |
|---|---|
| Main Author | |
| Format | Journal Article |
| Language | Japanese |
| Published |
一般社団法人 日本内科学会
2006
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| Subjects | |
| Online Access | Get full text |
| ISSN | 0021-5384 1883-2083 |
| DOI | 10.2169/naika.95.61a |
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| Author | 山田, 信博 |
|---|---|
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| DOI | 10.2169/naika.95.61a |
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| Discipline | Medicine |
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| EndPage | 63a |
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| References | 2) Abe H, et al : Hypertension, hypertriglyceridemia, and impaired endothelium-dependent vascular relaxation in mice lacking insulin receptor substrate-1. J Clin Invest 101 : 1784-1788, 1998. 4) Ide T, et al : SREBPs suppress IRS-2-mediated insulin signalling in the liver. Nature Cell Biology 6 (4) : 351-357, 2004. 5) Yoshikawa T, et al : Identification of Liver X Receptor-Retinoid X Receptor as an Activator of the Sterol Regulatory Element-Binding Protein 1c Gene Promoter. Mol Cell Biol 21 : 2991-3000, 2001. 6) Ide T, et al : Cross-talk between peroxisome proliferator-activated receptor (PPAR) alpha and liver X receptor (LXR) in nutritional regulation of fatty acid metabolism. II. LXRs suppress lipid degradation gene promoters through inhibition of PPAR signaling. Mol Endocrinol 17 : 1255-1267, 2003. 3) Chen Z, et al : Troglitazone inhibits atherosclerosis in apolipoprotein E-knockout mice : pleiotropic effects on CD36 expression and HDL. Arterioscler Thromb Vasc Biol 21 : 372-375, 2001. 7) Matsuzaka T, et al : Insulin-Independent Induction of Sterol Regulatory Element-Binding Protein-1c Expression in the Livers of Streptozotocin-Treated Mice. Diabetes 53 : 560-569, 2004. 1) Yamada N, et al : Increased risk factors for coronary artery disease in Japanese subjects with hyperinsulinemia or glucose intolerance. Diabetes Care 17 : 107, 1994. |
| References_xml | – reference: 4) Ide T, et al : SREBPs suppress IRS-2-mediated insulin signalling in the liver. Nature Cell Biology 6 (4) : 351-357, 2004. – reference: 6) Ide T, et al : Cross-talk between peroxisome proliferator-activated receptor (PPAR) alpha and liver X receptor (LXR) in nutritional regulation of fatty acid metabolism. II. LXRs suppress lipid degradation gene promoters through inhibition of PPAR signaling. Mol Endocrinol 17 : 1255-1267, 2003. – reference: 1) Yamada N, et al : Increased risk factors for coronary artery disease in Japanese subjects with hyperinsulinemia or glucose intolerance. Diabetes Care 17 : 107, 1994. – reference: 2) Abe H, et al : Hypertension, hypertriglyceridemia, and impaired endothelium-dependent vascular relaxation in mice lacking insulin receptor substrate-1. J Clin Invest 101 : 1784-1788, 1998. – reference: 3) Chen Z, et al : Troglitazone inhibits atherosclerosis in apolipoprotein E-knockout mice : pleiotropic effects on CD36 expression and HDL. Arterioscler Thromb Vasc Biol 21 : 372-375, 2001. – reference: 5) Yoshikawa T, et al : Identification of Liver X Receptor-Retinoid X Receptor as an Activator of the Sterol Regulatory Element-Binding Protein 1c Gene Promoter. Mol Cell Biol 21 : 2991-3000, 2001. – reference: 7) Matsuzaka T, et al : Insulin-Independent Induction of Sterol Regulatory Element-Binding Protein-1c Expression in the Livers of Streptozotocin-Treated Mice. Diabetes 53 : 560-569, 2004. |
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| SubjectTerms | インスリン抵抗性 メタボリックシンドローム 動脈硬化 糖尿病 転写調節 |
| Title | 2. 動脈硬化のハイリスク病態とその分子基盤 |
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