尿路結石症の発生原因に対する内分泌学的検討 第IV報 カルシウム制限および負荷試験による高カルシウム尿症における病態生理の研究
前報のごとくCa摂取量がほぼ日常通りの条件下で, 尿路結石症のCa代謝について知り得ることには限界があつたため, Ca制限及びCa負荷によるCa摂取量に条件を加え検討をした. (1) 1日200mg以下のCa制限食を4日間したCa制限試験では, 尿中Ca排泄量 (U-Ca) は高Ca尿症群 (HC) においてのみ著明に低下, 尿中 cyclic AMP (U-cAMP) もHCにのみ有意な低下をみた. (2) 1g相当のCa負荷試験ではU-Ca, 血清Ca値は対照群 (CG), 正常Ca尿症群 (NC), HCの3群共に極めて大きい上昇をみたが, HCの上昇は他の2群よりも有意に大きかつた....
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| Published in | 日本泌尿器科學會雑誌 Vol. 71; no. 11; pp. 1349 - 1363 |
|---|---|
| Main Author | |
| Format | Journal Article |
| Language | Japanese |
| Published |
社団法人 日本泌尿器科学会
20.11.1980
|
| Online Access | Get full text |
| ISSN | 0021-5287 1884-7110 |
| DOI | 10.5980/jpnjurol1928.71.11_1349 |
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| Abstract | 前報のごとくCa摂取量がほぼ日常通りの条件下で, 尿路結石症のCa代謝について知り得ることには限界があつたため, Ca制限及びCa負荷によるCa摂取量に条件を加え検討をした. (1) 1日200mg以下のCa制限食を4日間したCa制限試験では, 尿中Ca排泄量 (U-Ca) は高Ca尿症群 (HC) においてのみ著明に低下, 尿中 cyclic AMP (U-cAMP) もHCにのみ有意な低下をみた. (2) 1g相当のCa負荷試験ではU-Ca, 血清Ca値は対照群 (CG), 正常Ca尿症群 (NC), HCの3群共に極めて大きい上昇をみたが, HCの上昇は他の2群よりも有意に大きかつた. この事からHCの原因は日頃のCa摂取量が多い他に, 腸管Ca吸収率が異常に多いことが推察された. Ca値の上昇と共にCGやNCではU-cAMPやPTHは低下したがHCでは低下せず, この理由はHCでは前報のごとく上皮小体機能が抑制されているため, たとえCa値が上昇しても余り低下しないものと考えられた. またその代償として, 血清Ca値を下げる目的で血清カルチトニン値が, HCにおいてのみ有意に上昇したものと推察される. (4) HCの中にはCa制限にてもU-Caが低下せず, U-cAMPも上昇しない症例がみられた. この症例は他のHCに比べ, U-cAMPやPTHが高く, 血清Caが低かつた. またCa負荷ではU-cAMPが著しく低下した. この事からこの症例では腎尿細管Ca再吸収量が高Ca尿症の原因で, いわゆる「腎Ca喪失型」と考えられ, その結果続発性上皮小体機能亢進症の状態を呈し, 一方他の大半のHC症例は「腸管吸収型」と考えられた. |
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| AbstractList | 前報のごとくCa摂取量がほぼ日常通りの条件下で, 尿路結石症のCa代謝について知り得ることには限界があつたため, Ca制限及びCa負荷によるCa摂取量に条件を加え検討をした. (1) 1日200mg以下のCa制限食を4日間したCa制限試験では, 尿中Ca排泄量 (U-Ca) は高Ca尿症群 (HC) においてのみ著明に低下, 尿中 cyclic AMP (U-cAMP) もHCにのみ有意な低下をみた. (2) 1g相当のCa負荷試験ではU-Ca, 血清Ca値は対照群 (CG), 正常Ca尿症群 (NC), HCの3群共に極めて大きい上昇をみたが, HCの上昇は他の2群よりも有意に大きかつた. この事からHCの原因は日頃のCa摂取量が多い他に, 腸管Ca吸収率が異常に多いことが推察された. Ca値の上昇と共にCGやNCではU-cAMPやPTHは低下したがHCでは低下せず, この理由はHCでは前報のごとく上皮小体機能が抑制されているため, たとえCa値が上昇しても余り低下しないものと考えられた. またその代償として, 血清Ca値を下げる目的で血清カルチトニン値が, HCにおいてのみ有意に上昇したものと推察される. (4) HCの中にはCa制限にてもU-Caが低下せず, U-cAMPも上昇しない症例がみられた. この症例は他のHCに比べ, U-cAMPやPTHが高く, 血清Caが低かつた. またCa負荷ではU-cAMPが著しく低下した. この事からこの症例では腎尿細管Ca再吸収量が高Ca尿症の原因で, いわゆる「腎Ca喪失型」と考えられ, その結果続発性上皮小体機能亢進症の状態を呈し, 一方他の大半のHC症例は「腸管吸収型」と考えられた. |
| Author | 郡, 健二郎 |
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| References | 20) Broadus, A. E., Dominguez, M. and Bartter, F. C.: Pathophysiological studies in idiopathic hypercalciuria: Use of an oral calcium tolerance test to characterize distinctive hypercalciuric subgroups. J. Clin. Endocrinol. Metab., 47, 751-760, 1978. 32) Albright, F. and Ellisworth, R.: Studies on the physiology of the parathyroid glands. J. Clin. Invest., 7, 183-188, 1929. 13) Parry, E. S. and Lister, I. S.: Sunlight and hypercalciuria. Lancet, 1063-1065, 1975. 15) Munson, P. L. and Gvay, T. K.: Function of thyrocalcitonin in normal physiology. Fed. Proc., 29, 1206-1208, 1970. 17) Pak, C. Y. C.: Diagnostic criteria for hypercalciuriar. Calcium Urolithiasis, p. 44-49, Plenum Medical Book Co., New York and London, 1978. 6) Ljunghall, S.: Incidence and natural history of renal stone disease and its relationship to calcium metabolism. Eur. Urol., 4, 424-430, 1978. 1) 郡健二郎: 尿路結石症の発生原因に対する内分泌学的検討. 第III報. カルシウム結石症におけるカルシウム代謝と上皮小体機能について. 日泌尿会誌投稿中. 3) Albright, F., Henneman, P., Benedict, PH. and Forbes, AP.: Idiopathic hypercalciuria. Proc. Roy. Soc. Med., 46, 1077-1087, 1953. 24) Bordier, P., Ryckewart, A., Gueris, J. and Rasmussen, H.: On the pathogenesis of socalled idiopathic hypercalciuria. Am. J. Med., 63, 398-409, 1977. 22) Fujita, T. and Barttcr, F. C.: Personal communication. 30) Barker, E. S.: Physiologic and clinical aspects of magnesium metabolism. J. Chron. Dis., 11, 278-282, 1960. 31) Agus, Z. S., Gardner, L. B., Beck, L. H. and Goldberg, M.: Effects of parathyroid hormone on renal tubular reabsorption of calcium, sodium and phosphate. Amer. J. Physiol., 224, 1143-1148, 1973. 16) Pak, C. Y. C., Kaplan, R., Bone, H., Townsend, J. and Waters, O.: A simple test for the diagnosis of absorptive, resorptive and renal hypercalciurias. N. Engl. J. Med., 292, 497-500, 1975. 5) Benabe, J. and Martinez-Maldonado, M. Some characteristics of recurrent calcium stone formers in Puerto Rico. Urology., 14, 13-17, 1979. 11) Schachter, D., Dowdle, E. B. and Schenker, H.: Active transport of calcium by the small intestine of the rat. Am. J. Physiol., 198, 263-268, 1960. 21) Coe, F. L., Canterbury, J. M., Firpo, J. J. and Reiss, E.: Evidence for secondary hyperparathyroidism. J. Clin. Invest., 52, 134-142, 1973. 28) Samiy, A. H., Brown, J. L., Globus, D. L., Kessler, R. H. and Tompson, D. D.: Interrelation between renal transport systems of magnesium and calcium. Am. J. Physiol., 198, 599-602, 1960. 8) Nordin, B. E. C., Peacock, M. and Wilkinson, R.: Hypercalciuria and calcium stone disease. Clin. Endocrinol. Metab., 1, 169-183, 1972. 4) Coe, F. L., Parks, J. H. and Moore, E. S.: Familial idiopathic hypercalciuria. N. Eng. J. Med., 300, 337-340, 1979. 2) 八竹直, 古武敏彦, 西井易穂, 清水トシ子: 尿中蓚酸に関する検討. 第1報. 尿中蓚酸の新しい測定法について. 日泌尿会誌, 70, 286-290, 1979. 7) Blacklock, N. J. and Macleod, M. A.: Calcium-47 absorption in urolithiasis. Br. J. Urol., 46, 377-384, 1974. 9) Coe, F. L.: Idiopathic hypercalciuria. Nephrolithiasis pathogenesis and treatment. P. 86-94 Year Book Medical Publishers Inc., Chicago, London, 1978. 18) Pak, C. Y. C. and Galosy, R. A.: Fasting urinary calcium and adenosine 3′,5′-monophosphate: A discriminant analysis for the identification of renal and absorptive hypercalciurias. J. Clin. Endocrino. Metab., 48, 260-265, 1979. 19) Sakhaee, K., Peterson, R., Northcutt, C. and Pak, C. Y. C.: Critical appraisal of oral calcium load test for indirect assessment of intestinal calcium absorption. Urology, 14, 251-255, 1979. 27) Massry, S. G.: Renal handling of magnesium, urolithiasis research, p. 57-72, Plenum Press, New York and London, 1976. 26) Pak, C. Y. C.: Idiopathic hypercalciuria. Adv. Exp. Biol., 81, 309-317, 1977. 10) Wasserman, R. H. and Kallfelz, F. A.: Vitamin D and unidirectional calcium fluxes across the rachitic chick duodenum. Am. J. Physiol., 203, 221-229, 1962. 23) Fuss, M., Verbeelen, D., Geurts, J., Simon, J., Gergans, A., De Backer, M., Six, R. and Corvilain, J.: The use of a test for the differential diagnosis of hypercalciuria. Eur. Urol., 4, 324-329, 1978. 29) Chesley, L. C. and Tepper, I.: Some effects of magnesium loading upon renal excretion of magnesium and certain other electrolytes. J. Clin. Invest., 37, 1362-1368, 1958. 12) Moore, E. S., Coe, F. L., McMann, B. J. and Favus, M. J.: Idiopathic hypercalciuria in children: Prevalence and metabolic characteristics. J. Pediatr., 92, 906-910, 1978. 14) DeLuca, H. F.: Recent advances in the metabolism and function of vitamin D. Fed. Proc., 28, 1678-1689, 1969. 25) Shen, F. H., Baylink, D. J., Nielsen, R. L., Sherrard, D. J., Ivey, J. L. and Haussler, M. R.: Increased serum 1,25-dihydroxyvitamin D in idiopathic hypercalciuria. J. Lab. Clin. Med., 90, 955-962, 1977. |
| References_xml | – reference: 11) Schachter, D., Dowdle, E. B. and Schenker, H.: Active transport of calcium by the small intestine of the rat. Am. J. Physiol., 198, 263-268, 1960. – reference: 25) Shen, F. H., Baylink, D. J., Nielsen, R. L., Sherrard, D. J., Ivey, J. L. and Haussler, M. R.: Increased serum 1,25-dihydroxyvitamin D in idiopathic hypercalciuria. J. Lab. Clin. Med., 90, 955-962, 1977. – reference: 6) Ljunghall, S.: Incidence and natural history of renal stone disease and its relationship to calcium metabolism. Eur. Urol., 4, 424-430, 1978. – reference: 16) Pak, C. Y. C., Kaplan, R., Bone, H., Townsend, J. and Waters, O.: A simple test for the diagnosis of absorptive, resorptive and renal hypercalciurias. N. Engl. J. Med., 292, 497-500, 1975. – reference: 17) Pak, C. Y. C.: Diagnostic criteria for hypercalciuriar. Calcium Urolithiasis, p. 44-49, Plenum Medical Book Co., New York and London, 1978. – reference: 12) Moore, E. S., Coe, F. L., McMann, B. J. and Favus, M. J.: Idiopathic hypercalciuria in children: Prevalence and metabolic characteristics. J. Pediatr., 92, 906-910, 1978. – reference: 26) Pak, C. Y. C.: Idiopathic hypercalciuria. Adv. Exp. Biol., 81, 309-317, 1977. – reference: 8) Nordin, B. E. C., Peacock, M. and Wilkinson, R.: Hypercalciuria and calcium stone disease. Clin. Endocrinol. Metab., 1, 169-183, 1972. – reference: 30) Barker, E. S.: Physiologic and clinical aspects of magnesium metabolism. J. Chron. Dis., 11, 278-282, 1960. – reference: 20) Broadus, A. E., Dominguez, M. and Bartter, F. C.: Pathophysiological studies in idiopathic hypercalciuria: Use of an oral calcium tolerance test to characterize distinctive hypercalciuric subgroups. J. Clin. Endocrinol. Metab., 47, 751-760, 1978. – reference: 18) Pak, C. Y. C. and Galosy, R. A.: Fasting urinary calcium and adenosine 3′,5′-monophosphate: A discriminant analysis for the identification of renal and absorptive hypercalciurias. J. Clin. Endocrino. Metab., 48, 260-265, 1979. – reference: 5) Benabe, J. and Martinez-Maldonado, M. Some characteristics of recurrent calcium stone formers in Puerto Rico. Urology., 14, 13-17, 1979. – reference: 32) Albright, F. and Ellisworth, R.: Studies on the physiology of the parathyroid glands. J. Clin. Invest., 7, 183-188, 1929. – reference: 13) Parry, E. S. and Lister, I. S.: Sunlight and hypercalciuria. Lancet, 1063-1065, 1975. – reference: 21) Coe, F. L., Canterbury, J. M., Firpo, J. J. and Reiss, E.: Evidence for secondary hyperparathyroidism. J. Clin. Invest., 52, 134-142, 1973. – reference: 1) 郡健二郎: 尿路結石症の発生原因に対する内分泌学的検討. 第III報. カルシウム結石症におけるカルシウム代謝と上皮小体機能について. 日泌尿会誌投稿中. – reference: 28) Samiy, A. H., Brown, J. L., Globus, D. L., Kessler, R. H. and Tompson, D. D.: Interrelation between renal transport systems of magnesium and calcium. Am. J. Physiol., 198, 599-602, 1960. – reference: 7) Blacklock, N. J. and Macleod, M. A.: Calcium-47 absorption in urolithiasis. Br. J. Urol., 46, 377-384, 1974. – reference: 15) Munson, P. L. and Gvay, T. K.: Function of thyrocalcitonin in normal physiology. Fed. Proc., 29, 1206-1208, 1970. – reference: 2) 八竹直, 古武敏彦, 西井易穂, 清水トシ子: 尿中蓚酸に関する検討. 第1報. 尿中蓚酸の新しい測定法について. 日泌尿会誌, 70, 286-290, 1979. – reference: 31) Agus, Z. S., Gardner, L. B., Beck, L. H. and Goldberg, M.: Effects of parathyroid hormone on renal tubular reabsorption of calcium, sodium and phosphate. Amer. J. Physiol., 224, 1143-1148, 1973. – reference: 3) Albright, F., Henneman, P., Benedict, PH. and Forbes, AP.: Idiopathic hypercalciuria. Proc. Roy. Soc. Med., 46, 1077-1087, 1953. – reference: 27) Massry, S. G.: Renal handling of magnesium, urolithiasis research, p. 57-72, Plenum Press, New York and London, 1976. – reference: 22) Fujita, T. and Barttcr, F. C.: Personal communication. – reference: 19) Sakhaee, K., Peterson, R., Northcutt, C. and Pak, C. Y. C.: Critical appraisal of oral calcium load test for indirect assessment of intestinal calcium absorption. Urology, 14, 251-255, 1979. – reference: 14) DeLuca, H. F.: Recent advances in the metabolism and function of vitamin D. Fed. Proc., 28, 1678-1689, 1969. – reference: 4) Coe, F. L., Parks, J. H. and Moore, E. S.: Familial idiopathic hypercalciuria. N. Eng. J. Med., 300, 337-340, 1979. – reference: 10) Wasserman, R. H. and Kallfelz, F. A.: Vitamin D and unidirectional calcium fluxes across the rachitic chick duodenum. Am. J. Physiol., 203, 221-229, 1962. – reference: 24) Bordier, P., Ryckewart, A., Gueris, J. and Rasmussen, H.: On the pathogenesis of socalled idiopathic hypercalciuria. Am. J. Med., 63, 398-409, 1977. – reference: 29) Chesley, L. C. and Tepper, I.: Some effects of magnesium loading upon renal excretion of magnesium and certain other electrolytes. J. Clin. Invest., 37, 1362-1368, 1958. – reference: 23) Fuss, M., Verbeelen, D., Geurts, J., Simon, J., Gergans, A., De Backer, M., Six, R. and Corvilain, J.: The use of a test for the differential diagnosis of hypercalciuria. Eur. Urol., 4, 324-329, 1978. – reference: 9) Coe, F. L.: Idiopathic hypercalciuria. Nephrolithiasis pathogenesis and treatment. P. 86-94 Year Book Medical Publishers Inc., Chicago, London, 1978. |
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| Snippet | 前報のごとくCa摂取量がほぼ日常通りの条件下で, 尿路結石症のCa代謝について知り得ることには限界があつたため, Ca制限及びCa負荷によるCa摂取量に条件を加え検討をした.... |
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| Subtitle | 第IV報 カルシウム制限および負荷試験による高カルシウム尿症における病態生理の研究 |
| Title | 尿路結石症の発生原因に対する内分泌学的検討 |
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