Inhibition of myostatin in mice improves insulin sensitivity via irisin-mediated cross talk between muscle and adipose tissues - Dong, J; Dong, Y; Dong, Y; Chen, F; Mitch, W E; Zhang, L (International Journal of Obesity, 2016)
Keywords: 13/1; 13/100; 13/109; 13/51; 13/89; 13/95; 14/1; 14/28; 14/63; 38/77; 631/443/319/1642/393; 631/45/127/1219; 692/163; 692/699; 692/700/2814; 692/700/565/1331; 96/21; Abnormalities; Adipocytes; Adipose tissue; Adipose tissue (brown); Adipose Tissue - metabolism; Adipose tissues; AKT protein; Animals; Body composition; Body fat; Browning; Care and treatment; Crosstalk; Cytokines; Development and progression; Diabetes mellitus; Diet, High-Fat; Disease Models, Animal; Energy expenditure; Epidemiology; Fatty acids; Genetic aspects; Glucose; Glucose tolerance; Glucose transporter; Health aspects; Health Promotion and Disease Prevention; High fat diet; Inflammation; Insulin; Insulin - metabolism; Insulin Resistance; Internal Medicine; Lentivirus; Life sciences; Lipid Metabolism; Macrophages; Male; Medicine; Medicine & Public Health; Membrane proteins; Metabolic Diseases; Metabolic pathways; Mice; Mice, Inbred C57BL; Molecular Targeted Therapy; Muscle proteins; Muscle, Skeletal - metabolism; Muscles; Myostatin; Myostatin - antagonists & inhibitors; Myostatin - metabolism; Obesity; original-article; Oxidation; Physiological aspects; Proteins; Proto-Oncogene Proteins c-akt - metabolism; Public Health; Receptor Cross-Talk; Stem cell transplantation; Stem cells; Therapeutic targets; Tolerances; China; Beijing China; United States--US