Disruption of KMT2D perturbs germinal center B cell development and promotes lymphomagenesis - Zhang, Jiyuan; Dominguez-Sola, David; Hussein, Shafinaz; Lee, Ji-Eun; Holmes, Antony B; Bansal, Mukesh; Vlasevska, Sofija; Mo, Tongwei; Tang, Hongyan; Basso, Katia; Ge, Kai; Dalla-Favera, Riccardo; et al. (Nature medicine, 2015)
Keywords: 13/106; 13/31; 13/51; 14/63; 38/22; 38/23; 38/39; 38/61; 38/70; 38/71; 38/77; 38/90; 49; 49/105; 49/39; 49/90; 631/337/100/2285; 631/67/1990/291/1621/1915; 64/60; 96/109; Animals; B cells; B-Lymphocytes - pathology; Biomedicine; Cancer; Cancer Research; Cell Proliferation; Development and progression; DNA Methylation; DNA-Binding Proteins - genetics; Enzymatic activity; Epigenesis, Genetic; Gene mutations; Gene Silencing; Genes; Genetic aspects; Germinal Center - cytology; Humans; Infectious Diseases; Lymphoma; Lymphoma, Large B-Cell, Diffuse - etiology; Lymphoma, Large B-Cell, Diffuse - genetics; Lymphomas; Metabolic Diseases; Methylation; Mice; Molecular Medicine; Mutation; Mutation, Missense; Neoplasm Proteins - genetics; Neurosciences; Physiological aspects; Transcription, Genetic; Tumors; United States