Statistical classifiers for diagnosing disease from immune repertoires: a case study using multiple sclerosis

Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire se...

Full description

Saved in:
Bibliographic Details
Published inBMC bioinformatics Vol. 18; no. 1; pp. 401 - 10
Main Authors Ostmeyer, Jared, Christley, Scott, Rounds, William H., Toby, Inimary, Greenberg, Benjamin M., Monson, Nancy L., Cowell, Lindsay G.
Format Journal Article
LanguageEnglish
Published London BioMed Central 07.09.2017
BioMed Central Ltd
Springer Nature B.V
BMC
Subjects
Online AccessGet full text
ISSN1471-2105
1471-2105
DOI10.1186/s12859-017-1814-6

Cover

Abstract Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose. Results We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data ( N  = 23) and 72% accuracy on unused data from a separate study ( N  = 102). Conclusions Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.
AbstractList Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose. We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data (N = 23) and 72% accuracy on unused data from a separate study (N = 102). Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.
Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose. We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data (N = 23) and 72% accuracy on unused data from a separate study (N = 102). Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.
Abstract Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose. Results We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data (N = 23) and 72% accuracy on unused data from a separate study (N = 102). Conclusions Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.
Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose.BACKGROUNDDeep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose.We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data (N = 23) and 72% accuracy on unused data from a separate study (N = 102).RESULTSWe use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data (N = 23) and 72% accuracy on unused data from a separate study (N = 102).Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.CONCLUSIONSOur method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.
Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose. Results We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data (N = 23) and 72% accuracy on unused data from a separate study (N = 102). Conclusions Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.
Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose. Results We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data (N = 23) and 72% accuracy on unused data from a separate study (N = 102). Conclusions Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process. Keywords: Antibody, Immune repertoire, CDR3, Machine learning, Multiple sclerosis, Statistical classifier
Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens the door for novel approaches to diagnose and prognosticate diseases with a driving immune component by identifying repertoire sequence patterns associated with clinical phenotypes. Indeed, recent studies support the feasibility of this, demonstrating an association between repertoire-level summary statistics (e.g., diversity) and patient outcomes for several diseases. In our own prior work, we have shown that six codons in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting multiple sclerosis (RRMS) have higher replacement mutation frequencies than observed in healthy controls or patients with other neurological diseases. However, prior methods to date have been limited to focusing on repertoire-level summary statistics, ignoring the vast amounts of information in the millions of individual immune receptors comprising a repertoire. We have developed a novel method that addresses this limitation by using innovative approaches for accommodating the extraordinary sequence diversity of immune receptors and widely used machine learning approaches. We applied our method to RRMS, an autoimmune disease that is notoriously difficult to diagnose. Results We use the biochemical features encoded by the complementarity determining region 3 of each B cell receptor heavy chain in every patient repertoire as input to a detector function, which is fit to give the correct diagnosis for each patient using maximum likelihood optimization methods. The resulting statistical classifier assigns patients to one of two diagnosis categories, RRMS or other neurological disease, with 87% accuracy by leave-one-out cross-validation on training data ( N  = 23) and 72% accuracy on unused data from a separate study ( N  = 102). Conclusions Our method is the first to apply statistical learning to immune repertoires to aid disease diagnosis, learning repertoire-level labels from the set of individual immune repertoire sequences. This method produced a repertoire-based statistical classifier for diagnosing RRMS that provides a high degree of diagnostic capability, rivaling the accuracy of diagnosis by a clinical expert. Additionally, this method points to a diagnostic biochemical motif in the antibodies of RRMS patients, which may offer insight into the disease process.
ArticleNumber 401
Audience Academic
Author Monson, Nancy L.
Rounds, William H.
Ostmeyer, Jared
Cowell, Lindsay G.
Greenberg, Benjamin M.
Toby, Inimary
Christley, Scott
Author_xml – sequence: 1
  givenname: Jared
  surname: Ostmeyer
  fullname: Ostmeyer, Jared
  organization: Department of Clinical Sciences, UT Southwestern Medical Center
– sequence: 2
  givenname: Scott
  surname: Christley
  fullname: Christley, Scott
  organization: Department of Clinical Sciences, UT Southwestern Medical Center
– sequence: 3
  givenname: William H.
  surname: Rounds
  fullname: Rounds, William H.
  organization: Department of Clinical Sciences, UT Southwestern Medical Center
– sequence: 4
  givenname: Inimary
  surname: Toby
  fullname: Toby, Inimary
  organization: Department of Clinical Sciences, UT Southwestern Medical Center
– sequence: 5
  givenname: Benjamin M.
  surname: Greenberg
  fullname: Greenberg, Benjamin M.
  organization: Department of Neurology and Neurotherapeutics, UT Southwestern Medical Center
– sequence: 6
  givenname: Nancy L.
  surname: Monson
  fullname: Monson, Nancy L.
  organization: Department of Neurology and Neurotherapeutics, UT Southwestern Medical Center
– sequence: 7
  givenname: Lindsay G.
  surname: Cowell
  fullname: Cowell, Lindsay G.
  email: lindsay.cowell@utsouthwestern.edu
  organization: Department of Clinical Sciences, UT Southwestern Medical Center
BackLink https://www.ncbi.nlm.nih.gov/pubmed/28882107$$D View this record in MEDLINE/PubMed
BookMark eNqNUk1v1DAQjVAR_YAfwAVF4gKHFNv5sMOhUlXxsVIlJApny-uMg1eOvdgJsP-eSXcpuxUg5IOtmffeeN7MaXbkg4cse0rJOaWieZUoE3VbEMoLKmhVNA-yE1pxWjBK6qO993F2mtKKIFCQ-lF2zIQQGOcn2XAzqtGm0Wrlcu1UStZYiCk3IeadVb0PyfoenwlUgtzEMOR2GCYPeYQ1xDHYCOl1rnI959M4dZt8uuUMkxvt2mFQO4iokx5nD41yCZ7s7rPs89s3n67eF9cf3i2uLq8LzUk5FqatTEWBgmopL43grFW8NszoigMHwgnryBJawlijGsqqctlWAkqC7TXAdHmWLba6XVAruY52UHEjg7LyNhBiL1XEnh1IzQiWYpQb1IFGoyivy05zwRrWcINabKs1-bXafFfO3QlSIuc5yO0cJNor5znIBkkXW9J6Wg7QafBjVO7gJ4cZb7_IPnyTdS2w3RoFXuwEYvg6QRrlYJMG55SHMCVJ25LXVJRcIPT5PegqTNGjv4iqqrZFV-hvVK-wa-tNwLp6FpWXNWkprdt6Lnv-BxSeDgarcfuMxfgB4eUBATEj_Bh7NaUkFzcfD7HP9k25c-PXNiKAbgEatyVFMP_lNL_H0XZe6TD7at0_mbvBJqzie4h7vv2V9BM0KBC2
CitedBy_id crossref_primary_10_1016_j_xcrm_2021_100192
crossref_primary_10_1186_s12859_019_3281_8
crossref_primary_10_1016_j_nrleng_2020_10_013
crossref_primary_10_1093_bioinformatics_btad426
crossref_primary_10_3389_fimmu_2021_627813
crossref_primary_10_1016_j_retram_2024_103439
crossref_primary_10_1038_s41435_021_00141_9
crossref_primary_10_3389_fimmu_2017_01418
crossref_primary_10_3389_fimmu_2018_00976
crossref_primary_10_3389_fimmu_2018_02913
crossref_primary_10_1016_j_nrl_2020_10_017
crossref_primary_10_1016_j_compbiomed_2021_104337
crossref_primary_10_1016_j_coisb_2020_10_010
crossref_primary_10_1101_gr_276683_122
crossref_primary_10_3389_fimmu_2021_574411
crossref_primary_10_1039_C9ME00071B
crossref_primary_10_3389_fimmu_2019_02533
crossref_primary_10_3390_diagnostics12071771
crossref_primary_10_1038_s41746_020_0229_3
crossref_primary_10_1111_imr_12664
crossref_primary_10_7554_eLife_38358
crossref_primary_10_3389_fonc_2023_1115361
crossref_primary_10_1038_s41467_021_25006_7
crossref_primary_10_1371_journal_pone_0265313
crossref_primary_10_7554_eLife_73111
crossref_primary_10_1146_annurev_chembioeng_101420_125021
crossref_primary_10_1038_s41467_021_21879_w
crossref_primary_10_4049_jimmunol_2200063
crossref_primary_10_1038_s41598_022_17741_8
crossref_primary_10_1158_0008_5472_CAN_19_0225
crossref_primary_10_1158_0008_5472_CAN_19_1457
crossref_primary_10_1074_jbc_REV120_010181
crossref_primary_10_1186_s12911_022_01985_5
crossref_primary_10_3389_fimmu_2021_624230
crossref_primary_10_1158_0008_5472_CAN_18_2292
crossref_primary_10_1186_s12859_019_2853_y
crossref_primary_10_1002_path_5592
crossref_primary_10_1093_bib_bbz095
crossref_primary_10_3389_fimmu_2018_00224
crossref_primary_10_1038_s41592_020_01020_3
crossref_primary_10_3389_fsysb_2022_918792
crossref_primary_10_1007_s40291_021_00513_x
crossref_primary_10_1371_journal_pcbi_1010681
crossref_primary_10_1111_imm_13165
crossref_primary_10_1080_21645515_2018_1475872
crossref_primary_10_1371_journal_pone_0229569
Cites_doi 10.1073/pnas.0408677102
10.1016/j.jneuroim.2009.05.014
10.1080/2162402X.2014.1001230
10.1016/j.gene.2015.07.011
10.1093/nar/gkt382
10.1158/1078-0432.CCR-13-3368
10.1371/journal.pone.0076546
10.4049/jimmunol.179.9.6343
10.1038/nrrheum.2014.220
10.1001/archneur.63.4.614
10.1111/j.1365-2249.2008.03770.x
10.1093/bioinformatics/btu138
10.1186/s40425-015-0070-4
10.1002/ana.22366
10.1177/1352458513491329
10.1016/j.autrev.2014.01.012
10.1016/j.jneuroim.2008.04.031
10.1186/s13073-015-0243-2
10.1186/1471-2164-15-110
ContentType Journal Article
Copyright The Author(s). 2017
COPYRIGHT 2017 BioMed Central Ltd.
Copyright BioMed Central 2017
Copyright_xml – notice: The Author(s). 2017
– notice: COPYRIGHT 2017 BioMed Central Ltd.
– notice: Copyright BioMed Central 2017
DBID C6C
AAYXX
CITATION
CGR
CUY
CVF
ECM
EIF
NPM
ISR
3V.
7QO
7SC
7X7
7XB
88E
8AL
8AO
8FD
8FE
8FG
8FH
8FI
8FJ
8FK
ABUWG
AEUYN
AFKRA
ARAPS
AZQEC
BBNVY
BENPR
BGLVJ
BHPHI
CCPQU
DWQXO
FR3
FYUFA
GHDGH
GNUQQ
HCIFZ
JQ2
K7-
K9.
L7M
LK8
L~C
L~D
M0N
M0S
M1P
M7P
P5Z
P62
P64
PHGZM
PHGZT
PIMPY
PJZUB
PKEHL
PPXIY
PQEST
PQGLB
PQQKQ
PQUKI
PRINS
Q9U
7X8
5PM
ADTOC
UNPAY
DOA
DOI 10.1186/s12859-017-1814-6
DatabaseName Springer Nature OA Free Journals
CrossRef
Medline
MEDLINE
MEDLINE (Ovid)
MEDLINE
MEDLINE
PubMed
Gale In Context: Science
ProQuest Central (Corporate)
Biotechnology Research Abstracts
Computer and Information Systems Abstracts
Health & Medical Collection
ProQuest Central (purchase pre-March 2016)
Medical Database (Alumni Edition)
Computing Database (Alumni Edition)
ProQuest Pharma Collection
Technology Research Database
ProQuest SciTech Collection
ProQuest Technology Collection
ProQuest Natural Science Journals
ProQuest Hospital Collection
Hospital Premium Collection (Alumni Edition)
ProQuest Central (Alumni) (purchase pre-March 2016)
ProQuest Central (Alumni)
ProQuest One Sustainability
ProQuest Central
Advanced Technologies & Computer Science Collection
ProQuest Central Essentials
Biological Science Collection
ProQuest Central
ProQuest Technology Collection
Natural Science Collection
ProQuest One
ProQuest Central Korea
Engineering Research Database
Health Research Premium Collection
Health Research Premium Collection (Alumni)
ProQuest Central Student
SciTech Premium Collection
ProQuest Computer Science Collection
Computer Science Database
ProQuest Health & Medical Complete (Alumni)
Advanced Technologies Database with Aerospace
ProQuest Biological Science Collection
Computer and Information Systems Abstracts – Academic
Computer and Information Systems Abstracts Professional
Computing Database
Health & Medical Collection (Alumni)
Medical Database
Biological Science Database
Advanced Technologies & Aerospace Database
ProQuest Advanced Technologies & Aerospace Collection
Biotechnology and BioEngineering Abstracts
ProQuest Central Premium
ProQuest One Academic (New)
Publicly Available Content Database (ProQuest)
ProQuest Health & Medical Research Collection
ProQuest One Academic Middle East (New)
ProQuest One Health & Nursing
ProQuest One Academic Eastern Edition (DO NOT USE)
ProQuest One Applied & Life Sciences
ProQuest One Academic
ProQuest One Academic UKI Edition
ProQuest Central China
ProQuest Central Basic
MEDLINE - Academic
PubMed Central (Full Participant titles)
Unpaywall for CDI: Periodical Content
Unpaywall
DOAJ Directory of Open Access Journals
DatabaseTitle CrossRef
MEDLINE
Medline Complete
MEDLINE with Full Text
PubMed
MEDLINE (Ovid)
Publicly Available Content Database
Computer Science Database
ProQuest Central Student
ProQuest Advanced Technologies & Aerospace Collection
ProQuest Central Essentials
ProQuest Computer Science Collection
Computer and Information Systems Abstracts
SciTech Premium Collection
ProQuest Central China
ProQuest One Applied & Life Sciences
ProQuest One Sustainability
Health Research Premium Collection
Natural Science Collection
Health & Medical Research Collection
Biological Science Collection
ProQuest Central (New)
ProQuest Medical Library (Alumni)
Advanced Technologies & Aerospace Collection
ProQuest Biological Science Collection
ProQuest One Academic Eastern Edition
ProQuest Hospital Collection
ProQuest Technology Collection
Health Research Premium Collection (Alumni)
Biological Science Database
ProQuest Hospital Collection (Alumni)
Biotechnology and BioEngineering Abstracts
ProQuest Health & Medical Complete
ProQuest One Academic UKI Edition
Engineering Research Database
ProQuest One Academic
ProQuest One Academic (New)
Technology Collection
Technology Research Database
Computer and Information Systems Abstracts – Academic
ProQuest One Academic Middle East (New)
ProQuest Health & Medical Complete (Alumni)
ProQuest Central (Alumni Edition)
ProQuest One Community College
ProQuest One Health & Nursing
ProQuest Natural Science Collection
ProQuest Pharma Collection
ProQuest Central
ProQuest Health & Medical Research Collection
Biotechnology Research Abstracts
Health and Medicine Complete (Alumni Edition)
ProQuest Central Korea
Advanced Technologies Database with Aerospace
ProQuest Computing
ProQuest Central Basic
ProQuest Computing (Alumni Edition)
ProQuest SciTech Collection
Computer and Information Systems Abstracts Professional
Advanced Technologies & Aerospace Database
ProQuest Medical Library
ProQuest Central (Alumni)
MEDLINE - Academic
DatabaseTitleList
MEDLINE


MEDLINE - Academic
Publicly Available Content Database


Database_xml – sequence: 1
  dbid: C6C
  name: SpringerOpen Free (Free internet resource, activated by CARLI)
  url: http://www.springeropen.com/
  sourceTypes: Publisher
– sequence: 2
  dbid: DOA
  name: DOAJ Directory of Open Access Journals
  url: https://www.doaj.org/
  sourceTypes: Open Website
– sequence: 3
  dbid: NPM
  name: PubMed
  url: https://proxy.k.utb.cz/login?url=http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 4
  dbid: EIF
  name: MEDLINE
  url: https://proxy.k.utb.cz/login?url=https://www.webofscience.com/wos/medline/basic-search
  sourceTypes: Index Database
– sequence: 5
  dbid: UNPAY
  name: Unpaywall
  url: https://proxy.k.utb.cz/login?url=https://unpaywall.org/
  sourceTypes: Open Access Repository
– sequence: 6
  dbid: 8FG
  name: ProQuest Technology Collection
  url: https://search.proquest.com/technologycollection1
  sourceTypes: Aggregation Database
DeliveryMethod fulltext_linktorsrc
Discipline Biology
Statistics
EISSN 1471-2105
EndPage 10
ExternalDocumentID oai_doaj_org_article_c2041e217f124e6c902753dc7826267f
10.1186/s12859-017-1814-6
PMC5588725
A509115955
28882107
10_1186_s12859_017_1814_6
Genre Journal Article
GrantInformation_xml – fundername: National Institute of Allergy and Infectious Diseases
  grantid: AI097403
  funderid: http://dx.doi.org/10.13039/100000060
– fundername: Cancer Prevention and Research Institute of Texas (US)
  grantid: RP160157
– fundername: NIAID NIH HHS
  grantid: R01 AI097403
– fundername: ;
  grantid: RP160157
– fundername: ;
  grantid: AI097403
GroupedDBID ---
0R~
23N
2WC
53G
5VS
6J9
7X7
88E
8AO
8FE
8FG
8FH
8FI
8FJ
AAFWJ
AAJSJ
AAKPC
AASML
ABDBF
ABUWG
ACGFO
ACGFS
ACIHN
ACIWK
ACPRK
ACUHS
ADBBV
ADMLS
ADRAZ
ADUKV
AEAQA
AENEX
AEUYN
AFKRA
AFPKN
AFRAH
AHBYD
AHMBA
AHYZX
ALMA_UNASSIGNED_HOLDINGS
AMKLP
AMTXH
AOIJS
ARAPS
AZQEC
BAPOH
BAWUL
BBNVY
BCNDV
BENPR
BFQNJ
BGLVJ
BHPHI
BMC
BPHCQ
BVXVI
C6C
CCPQU
CS3
DIK
DU5
DWQXO
E3Z
EAD
EAP
EAS
EBD
EBLON
EBS
EJD
EMB
EMK
EMOBN
ESX
F5P
FYUFA
GNUQQ
GROUPED_DOAJ
GX1
H13
HCIFZ
HMCUK
HYE
IAO
ICD
IHR
INH
INR
ISR
ITC
K6V
K7-
KQ8
LK8
M1P
M48
M7P
MK~
ML0
M~E
O5R
O5S
OK1
OVT
P2P
P62
PGMZT
PHGZM
PHGZT
PIMPY
PJZUB
PPXIY
PQGLB
PQQKQ
PROAC
PSQYO
PUEGO
RBZ
RNS
ROL
RPM
RSV
SBL
SOJ
SV3
TR2
TUS
UKHRP
W2D
WOQ
WOW
XH6
XSB
AAYXX
CITATION
-A0
3V.
ACRMQ
ADINQ
ALIPV
C24
CGR
CUY
CVF
ECM
EIF
M0N
NPM
7QO
7SC
7XB
8AL
8FD
8FK
AHSBF
FR3
JQ2
K9.
L7M
L~C
L~D
P64
PKEHL
PQEST
PQUKI
PRINS
Q9U
7X8
5PM
123
2VQ
4.4
ADTOC
AFFHD
C1A
IPNFZ
RIG
UNPAY
ID FETCH-LOGICAL-c703t-f94f41e1ea9173f8729a75f2fc47e7e0702d0be90226a61243b948e307806e2c3
IEDL.DBID DOA
ISSN 1471-2105
IngestDate Tue Oct 14 18:21:35 EDT 2025
Wed Oct 29 12:00:32 EDT 2025
Tue Sep 30 15:12:25 EDT 2025
Thu Sep 04 20:02:09 EDT 2025
Mon Oct 06 18:32:31 EDT 2025
Mon Oct 20 22:16:39 EDT 2025
Mon Oct 20 16:29:53 EDT 2025
Thu Oct 16 15:18:20 EDT 2025
Wed Feb 19 02:41:20 EST 2025
Wed Oct 01 04:15:29 EDT 2025
Thu Apr 24 23:10:04 EDT 2025
Sat Sep 06 07:27:16 EDT 2025
IsDoiOpenAccess true
IsOpenAccess true
IsPeerReviewed true
IsScholarly true
Issue 1
Keywords Multiple sclerosis
Antibody
CDR3
Immune repertoire
Statistical classifier
Machine learning
Language English
License Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
cc-by
LinkModel DirectLink
MergedId FETCHMERGED-LOGICAL-c703t-f94f41e1ea9173f8729a75f2fc47e7e0702d0be90226a61243b948e307806e2c3
Notes ObjectType-Case Study-2
SourceType-Scholarly Journals-1
content type line 14
ObjectType-Feature-4
ObjectType-Report-1
ObjectType-Article-3
ObjectType-Article-1
ObjectType-Feature-2
content type line 23
OpenAccessLink https://doaj.org/article/c2041e217f124e6c902753dc7826267f
PMID 28882107
PQID 1944997801
PQPubID 44065
PageCount 10
ParticipantIDs doaj_primary_oai_doaj_org_article_c2041e217f124e6c902753dc7826267f
unpaywall_primary_10_1186_s12859_017_1814_6
pubmedcentral_primary_oai_pubmedcentral_nih_gov_5588725
proquest_miscellaneous_1937518378
proquest_journals_1944997801
gale_infotracmisc_A509115955
gale_infotracacademiconefile_A509115955
gale_incontextgauss_ISR_A509115955
pubmed_primary_28882107
crossref_primary_10_1186_s12859_017_1814_6
crossref_citationtrail_10_1186_s12859_017_1814_6
springer_journals_10_1186_s12859_017_1814_6
ProviderPackageCode CITATION
AAYXX
PublicationCentury 2000
PublicationDate 2017-09-07
PublicationDateYYYYMMDD 2017-09-07
PublicationDate_xml – month: 09
  year: 2017
  text: 2017-09-07
  day: 07
PublicationDecade 2010
PublicationPlace London
PublicationPlace_xml – name: London
– name: England
PublicationSubtitle BMC series – open, inclusive and trusted
PublicationTitle BMC bioinformatics
PublicationTitleAbbrev BMC Bioinformatics
PublicationTitleAlternate BMC Bioinformatics
PublicationYear 2017
Publisher BioMed Central
BioMed Central Ltd
Springer Nature B.V
BMC
Publisher_xml – name: BioMed Central
– name: BioMed Central Ltd
– name: Springer Nature B.V
– name: BMC
References MA Postow (1814_CR6) 2015; 3
SS Kim (1814_CR14) 2014; 20
GP Owens (1814_CR15) 2007; 179
R Milo (1814_CR13) 2014; 13
M Abadi (1814_CR22) 2016; 1603
W Luo (1814_CR1) 2008; 154
WR Atchley (1814_CR10) 2005; 102
V Seitz (1814_CR21) 2015; 43
MD Iglesia (1814_CR4) 2014; 20
WH Robinson (1814_CR12) 2015; 11
J Ye (1814_CR19) 2013; 41
WH Rounds (1814_CR7) 2015; 572
CH Polman (1814_CR8) 2011; 69
EM Frohman (1814_CR9) 2006; 63
G Yaari (1814_CR17) 2015; 7
JL Bennett (1814_CR16) 2008; 199
D Kingma (1814_CR11) 2014; 1412
EM Cameron (1814_CR2) 2009; 213
Vander Heiden (1814_CR18) 2014; 30
MA Quail (1814_CR20) 2014; 15
Q Jia (1814_CR5) 2015; 4
I Marrero (1814_CR3) 2013; 8
23736535 - Mult Scler. 2014 Jan;20(1):57-63
23671333 - Nucleic Acids Res. 2013 Jul;41(Web Server issue):W34-40
26137399 - Oncoimmunology. 2015 Jan 22;4(4):e1001230
24424194 - Autoimmun Rev. 2014 Apr-May;13(4-5):518-24
26085931 - J Immunother Cancer. 2015 Jun 16;3:23
24146886 - PLoS One. 2013 Oct 17;8(10):e76546
16606781 - Arch Neurol. 2006 Apr;63(4):614-9
26152304 - Nucleic Acids Res. 2015 Nov 16;43(20):e135
26172868 - Gene. 2015 Nov 10;572(2):191-7
18811695 - Clin Exp Immunol. 2008 Dec;154(3):316-24
21387374 - Ann Neurol. 2011 Feb;69(2):292-302
17947712 - J Immunol. 2007 Nov 1;179(9):6343-51
24618469 - Bioinformatics. 2014 Jul 1;30(13):1930-2
24507442 - BMC Genomics. 2014 Feb 07;15:110
26589402 - Genome Med. 2015 Nov 20;7:121
25536486 - Nat Rev Rheumatol. 2015 Mar;11(3):171-82
24916698 - Clin Cancer Res. 2014 Jul 15;20(14):3818-29
15851683 - Proc Natl Acad Sci U S A. 2005 May 3;102(18):6395-400
18547652 - J Neuroimmunol. 2008 Aug 13;199(1-2):126-32
19631394 - J Neuroimmunol. 2009 Aug 18;213(1-2):123-30
References_xml – volume: 102
  start-page: 6395
  issue: 18
  year: 2005
  ident: 1814_CR10
  publication-title: Proc Natl Acad Sci U S A
  doi: 10.1073/pnas.0408677102
– volume: 213
  start-page: 123
  issue: 1–2
  year: 2009
  ident: 1814_CR2
  publication-title: J Neuroimmunol
  doi: 10.1016/j.jneuroim.2009.05.014
– volume: 4
  issue: 4
  year: 2015
  ident: 1814_CR5
  publication-title: Oncoimmunology
  doi: 10.1080/2162402X.2014.1001230
– volume: 572
  start-page: 191
  issue: 2
  year: 2015
  ident: 1814_CR7
  publication-title: Gene
  doi: 10.1016/j.gene.2015.07.011
– volume: 41
  start-page: W34
  issue: Web Server issu
  year: 2013
  ident: 1814_CR19
  publication-title: Nucleic Acids Res
  doi: 10.1093/nar/gkt382
– volume: 20
  start-page: 3818
  issue: 14
  year: 2014
  ident: 1814_CR4
  publication-title: Clin Cancer Res
  doi: 10.1158/1078-0432.CCR-13-3368
– volume: 8
  issue: 10
  year: 2013
  ident: 1814_CR3
  publication-title: PLoS One
  doi: 10.1371/journal.pone.0076546
– volume: 179
  start-page: 6343
  issue: 9
  year: 2007
  ident: 1814_CR15
  publication-title: J Immunol
  doi: 10.4049/jimmunol.179.9.6343
– volume: 11
  start-page: 171
  issue: 3
  year: 2015
  ident: 1814_CR12
  publication-title: Nat Rev Rheumatol
  doi: 10.1038/nrrheum.2014.220
– volume: 43
  start-page: e135
  issue: 20
  year: 2015
  ident: 1814_CR21
  publication-title: Nucleic Acids Res
– volume: 63
  start-page: 614
  issue: 4
  year: 2006
  ident: 1814_CR9
  publication-title: Arch Neurol
  doi: 10.1001/archneur.63.4.614
– volume: 154
  start-page: 316
  issue: 3
  year: 2008
  ident: 1814_CR1
  publication-title: Clin Exp Immunol
  doi: 10.1111/j.1365-2249.2008.03770.x
– volume: 30
  start-page: 1930
  issue: 13
  year: 2014
  ident: 1814_CR18
  publication-title: Bioinformatics
  doi: 10.1093/bioinformatics/btu138
– volume: 3
  start-page: 23
  year: 2015
  ident: 1814_CR6
  publication-title: J Immunother Cancer
  doi: 10.1186/s40425-015-0070-4
– volume: 69
  start-page: 292
  issue: 2
  year: 2011
  ident: 1814_CR8
  publication-title: Ann Neurol
  doi: 10.1002/ana.22366
– volume: 1412
  start-page: 6980
  year: 2014
  ident: 1814_CR11
  publication-title: Adam: A method for stochastic optimization arXiv preprint arXiv
– volume: 20
  start-page: 57
  issue: 1
  year: 2014
  ident: 1814_CR14
  publication-title: Mult Scler
  doi: 10.1177/1352458513491329
– volume: 13
  start-page: 518
  issue: 4–5
  year: 2014
  ident: 1814_CR13
  publication-title: Autoimmun Rev
  doi: 10.1016/j.autrev.2014.01.012
– volume: 1603
  start-page: 04467
  year: 2016
  ident: 1814_CR22
  publication-title: Tensorflow: Large-scale machine learning on heterogeneous distributed systems arXiv preprint arXiv
– volume: 199
  start-page: 126
  issue: 1–2
  year: 2008
  ident: 1814_CR16
  publication-title: J Neuroimmunol
  doi: 10.1016/j.jneuroim.2008.04.031
– volume: 7
  start-page: 121
  year: 2015
  ident: 1814_CR17
  publication-title: Genome Med
  doi: 10.1186/s13073-015-0243-2
– volume: 15
  start-page: 110
  year: 2014
  ident: 1814_CR20
  publication-title: BMC Genomics
  doi: 10.1186/1471-2164-15-110
– reference: 25536486 - Nat Rev Rheumatol. 2015 Mar;11(3):171-82
– reference: 26152304 - Nucleic Acids Res. 2015 Nov 16;43(20):e135
– reference: 19631394 - J Neuroimmunol. 2009 Aug 18;213(1-2):123-30
– reference: 23671333 - Nucleic Acids Res. 2013 Jul;41(Web Server issue):W34-40
– reference: 15851683 - Proc Natl Acad Sci U S A. 2005 May 3;102(18):6395-400
– reference: 24618469 - Bioinformatics. 2014 Jul 1;30(13):1930-2
– reference: 21387374 - Ann Neurol. 2011 Feb;69(2):292-302
– reference: 24146886 - PLoS One. 2013 Oct 17;8(10):e76546
– reference: 26589402 - Genome Med. 2015 Nov 20;7:121
– reference: 26137399 - Oncoimmunology. 2015 Jan 22;4(4):e1001230
– reference: 26172868 - Gene. 2015 Nov 10;572(2):191-7
– reference: 24424194 - Autoimmun Rev. 2014 Apr-May;13(4-5):518-24
– reference: 24916698 - Clin Cancer Res. 2014 Jul 15;20(14):3818-29
– reference: 18811695 - Clin Exp Immunol. 2008 Dec;154(3):316-24
– reference: 23736535 - Mult Scler. 2014 Jan;20(1):57-63
– reference: 18547652 - J Neuroimmunol. 2008 Aug 13;199(1-2):126-32
– reference: 24507442 - BMC Genomics. 2014 Feb 07;15:110
– reference: 17947712 - J Immunol. 2007 Nov 1;179(9):6343-51
– reference: 26085931 - J Immunother Cancer. 2015 Jun 16;3:23
– reference: 16606781 - Arch Neurol. 2006 Apr;63(4):614-9
SSID ssj0017805
Score 2.4706116
Snippet Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations....
Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations. This opens...
Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte populations....
Abstract Background Deep sequencing of lymphocyte receptor repertoires has made it possible to comprehensively profile the clonal composition of lymphocyte...
SourceID doaj
unpaywall
pubmedcentral
proquest
gale
pubmed
crossref
springer
SourceType Open Website
Open Access Repository
Aggregation Database
Index Database
Enrichment Source
Publisher
StartPage 401
SubjectTerms Algorithms
Amino Acid Sequence
Antibodies
Antibody
Area Under Curve
Autoimmune diseases
B-Lymphocytes - metabolism
Bioinformatics
Biomedical and Life Sciences
Case depth
CDR3
Cerebrospinal fluid
Classifiers
Coding
Codons
Complementarity
Complementarity Determining Regions - chemistry
Complementarity Determining Regions - metabolism
Complementarity-determining region 3
Computational biology
Computational Biology/Bioinformatics
Computer Appl. in Life Sciences
Datasets
Diagnosis
Diagnostic systems
Feasibility studies
High-Throughput Nucleotide Sequencing
Humans
Immune repertoire
Learning algorithms
Life Sciences
Lymphocytes
Lymphocytes B
Machine learning
Microarrays
Models, Statistical
Molecular diagnostic techniques
Multiple sclerosis
Multiple Sclerosis, Relapsing-Remitting - classification
Multiple Sclerosis, Relapsing-Remitting - diagnosis
Multiple Sclerosis, Relapsing-Remitting - immunology
Nervous System Diseases - classification
Nervous System Diseases - diagnosis
Nervous System Diseases - immunology
Neurological diseases
Ovarian cancer
Patients
Receptors
Research Article
ROC Curve
Sequence analysis (applications)
Statistical classifier
Statistical methods
Statistics
T cell receptors
SummonAdditionalLinks – databaseName: ProQuest Central
  dbid: BENPR
  link: http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwhV3daxNBEB9qilgfROvXaZVVBMFy9L73Ioi00lJ9CFIt9G3Z7M3WQLyLuQTpf-_M3d6ZU6yPye6S_ZiZ_U1m9jcAr2SREQjntPIosH6CMvZ1aCT5PBiHmMc2LRq2z0l2ep58ukgvtmDSvYXhtMrOJjaGuqgM_0d-QM42gXNJBvX94ofPVaM4utqV0NCutELxrqEYuwHbETNjjWD76Hjy-ayPKzCDv4tthnl2UIfM3-azpaabLvGzwe3UkPj_bao37qo_8yj7YOptuLUuF_rqp57PN-6rk7twxwFNcdhKxj3YwnIXbralJ692YYdRZkvSfB--9x9ohGE8PbNcIlsQohVFm4xHPyhcNEfwmxQx45clKJa4wOWqIstZvxVaGG5vOGvFuhnTZSyKmqZBi57VD-D85Pjrh1Pf1WHwDdmDlW_HiU1CDFGTbxfbnPC4lqmNrEkkSiSjERXBFMcEBzJNiCmJp-MkR7IeeZBhZOKHMCqrEh-DsJoZ-UyQ23GeoMVpUUwxNwazMJQhFh4E3f4r40jKuVbGXDXOSp6p9sgUHZniI1OZB2_6IYuWoeO6zkd8qH1HJtduvqiWl8rpqjJRQMslX83SWjAzYw7txoUhMEXun7QevGSRUEyfUXJ-zqVe17X6-OVMHTL-IoSYph68dp1sRSsw2j13oH1gxq1Bz71BT9JvM2zuJE85-1Kr39rgwYu-mUdyzlyJ1Zr7xBxTi2XuwaNWUPt1Rzl5VuT5eyAHIjzYmGFLOfvWsI-nKd1LEU1rvxP2jWn9e9_3e334_yk9uX7JT2EnYpXlsJ7cg9FqucZnBA1X0-dO338Bth5f5A
  priority: 102
  providerName: ProQuest
– databaseName: Scholars Portal Journals: Open Access
  dbid: M48
  link: http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwrV1Li9RAEC7WFVEP4tvoKq0Igks0j066I4is4rIKelAH9tYknep1YMyMkxl0_r1VeTnRVS9e09UkXe-iOl8BPFRlSkk4XyuPAudLVLGfh1ZRzYNxiDp2Sdmgfb5Pjyby7XFyvAP9eKuOgfWppR3Pk5osZ0--f928IIN_3hi8Tp_WIaOw-exvKV5JPz0DZylQZTzJ4Z382VRg-P6usXnqtlFoahD8f_fTW4Hq10uUQyf1IpxfV4t88y2fzbaC1eFluNRlmeKgVYsrsIPVVTjXzp3cXIMvnGI2CM1EZDl_njoeiS0ogxVle_mO3iG67o3gf1DElP8kQbHEBS5Xc_KU9TORC8vrDUatWDd7-huKoqY30zmn9XWYHL7-9OrI7-Yu-Jbsf-W7TDoZYog51XKx05R_5ypxkbNSoUJyElEZFJhR-E9zypBkXGRSI3kLHaQY2fgG7FbzCm-BcDkj8NlAu0xLdFiUZYHaWkzDUIVYehD0LDe2AyXn2Rgz0xQnOjWtlAxJybCUTOrB42HLokXk-BvxS5bjQMhg2s2D-fLEdLZpbBTQcak2c3QWTG3Grdy4tJQ8UbmnnAcPWAsMw2VUfB_nJF_XtXnz8YM54HyLMsIk8eBRR-TmdAKbd783EB8YYWtEuTeiJHu24-Ve2UxvDibMJJWmxN_Qg_vDMu_kO3IVztdME3MPLVbag5utbg7njjRVUlTpe6BGWjtizHilmn5u0MaThOJQRJ-13-v31mf9me_7gwn8W0q3_4eU7sCFiG2Zm31qD3ZXyzXepYRxVdxr3MAPHAdkTg
  priority: 102
  providerName: Scholars Portal
– databaseName: Springer Nature OA Free Journals
  dbid: C6C
  link: http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwlV3di9QwEA96IuqD-G31lCiC4FFs2jRJfTsPj9MHH9SDewvZdHIurN1lu0Xuv3cm7Zatn_jaTNom881MfmHsha4VBuHUVp5nIZWgi9QJrzHngUKAKUJZR7TPj-rkVH44K88GsGg6C7NbvxdGvW4FIaylZEvRF8lUXWZX0EepWJdVR2PBgKD5h6Llb6dN3E5E5__VBu84oZ8bJMcq6Q12rWtW7uK7Wyx2HNHxLXZziCD5Yc_y2-wSNHfY1f5OyYu77BuFjxF9GYk8xcbzQNddc4xOed031uE3-FCZ4XS-hM_plAjwNaxgvVmiFWzfcMc9jUf8Wd7FOdvuQ97il3Gd8_YeOz1-9-XoJB3uVEg96vYmDZUMUoAAh3laEQzG1k6XIQ9eatCABiCvsxlU6NqVw-hHFrNKGkBLYDIFuS_us71m2cBDxoMjdD2fmVAZCQFmdT0D4z0oIbSAOmHZdsutHwDH6d6LhY2Jh1G255JFLlniklUJezVOWfVoG38jfkt8HAkJKDs-QPmxg95Zn2e4XMy7Aq4FlK-oTFvUHgMjTOV0SNhzkgJLUBgN9dqcu65t7fvPn-whxVIY7ZVlwl4ORGGJK_BuOLqA-0DoWRPK_Qkl6qqfDm-FzQ62orWikph24v6KhD0bh2km9b81sOyIpqD6WKFNwh70sjmuOzeYJWEWnzA9kdrJxkxHmvnXiCReluhjcvytg6187_zWn_f9YFSBf3Pp0X-9-zG7npPSUsVO77O9zbqDJxj1bWZPo77_AIzjTYY
  priority: 102
  providerName: Springer Nature
– databaseName: Unpaywall
  dbid: UNPAY
  link: http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwrV3da9RAEB_qFVEf_KwarbKKIFhyzeZrE99OsVQfqqgH9WlJNrs19JoclwtS_3pn8sWlfiH4dtzOkuxkZ_Y3zMxvAZ6JLEQQTmXlrmNsXwvPTrgSGPNoj-vIM0HWsH0ehYdz_91xcLwF7_temPRMpXnZkYYSUfF0sw190fhu_KFO95eZaU0-CvcrTjxsNnlcPLF8O7wE22GA4HwC2_OjD7MvTY-R4DYGOEGX2_zlvNHp1JD4_-yqN86qi3WUQzL1Glypi2Vy_i1ZLDbOq4MbsOxX2papnE7rdTpV3y-QQP5HVdyE6x22ZbN2M96CLV3chsvtbZfnd-CMgG3DC41CilB7bugiboaPZllb8ofLYl3OiFHnC8upf0WzlV7q1bpE_1y9ZAlTNN4w47K6mdPXRbIKn4yqzasdmB-8-fz60O5ue7AVep21bWLf-FxznWAE6ZkIUX8iAuMa5QstNLomN3NSHSPoCBPEZb6Xxn6k0UdFTqhd5d2FSVEW-j4wkxDvn3IiE0e-NjrNslRHSumQc8F1ZoHTf2WpOip0upFjIZuQKAplq0WJWpSkRRla8GKYsmx5QP4k_Iq2ziBIFN7NH-XqRHYeQSrXweViRGhwLTpUMSWQvUwhZMMgUxgLntLGk0TSUVAV0ElSV5V8--mjnBHKQxwaBBY874RMSXsh6ZoqUA_E6zWS3B1JohdR4-F-f8vOi1WSxz4GxKhfbsGTYZhmUmVeocuaZDzK3HkisuBeaw7Dut0I4zfuCAvEyFBGihmPFPnXhuM8CPD0c_G19nqT2nit3-t9b7C6v3-lB_8k_RCuumRUlEsUuzBZr2r9CPHoOn3c-ZgfRuOD_w
  priority: 102
  providerName: Unpaywall
Title Statistical classifiers for diagnosing disease from immune repertoires: a case study using multiple sclerosis
URI https://link.springer.com/article/10.1186/s12859-017-1814-6
https://www.ncbi.nlm.nih.gov/pubmed/28882107
https://www.proquest.com/docview/1944997801
https://www.proquest.com/docview/1937518378
https://pubmed.ncbi.nlm.nih.gov/PMC5588725
https://bmcbioinformatics.biomedcentral.com/track/pdf/10.1186/s12859-017-1814-6
https://doaj.org/article/c2041e217f124e6c902753dc7826267f
UnpaywallVersion publishedVersion
Volume 18
hasFullText 1
inHoldings 1
isFullTextHit
isPrint
journalDatabaseRights – providerCode: PRVADU
  databaseName: BioMed Central Open Access Free
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: RBZ
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: https://www.biomedcentral.com/search/
  providerName: BioMedCentral
– providerCode: PRVAFT
  databaseName: Open Access Digital Library
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: KQ8
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: http://grweb.coalliance.org/oadl/oadl.html
  providerName: Colorado Alliance of Research Libraries
– providerCode: PRVAFT
  databaseName: Open Access Digital Library
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: KQ8
  dateStart: 20000701
  isFulltext: true
  titleUrlDefault: http://grweb.coalliance.org/oadl/oadl.html
  providerName: Colorado Alliance of Research Libraries
– providerCode: PRVAON
  databaseName: DOAJ Directory of Open Access Journals
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: DOA
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: https://www.doaj.org/
  providerName: Directory of Open Access Journals
– providerCode: PRVEBS
  databaseName: EBSCOhost Academic Search Ultimate
  customDbUrl: https://search.ebscohost.com/login.aspx?authtype=ip,shib&custid=s3936755&profile=ehost&defaultdb=asn
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: ABDBF
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: https://search.ebscohost.com/direct.asp?db=asn
  providerName: EBSCOhost
– providerCode: PRVEBS
  databaseName: Inspec with Full Text
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: ADMLS
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: https://www.ebsco.com/products/research-databases/inspec-full-text
  providerName: EBSCOhost
– providerCode: PRVBFR
  databaseName: Free Medical Journals
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: DIK
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: http://www.freemedicaljournals.com
  providerName: Flying Publisher
– providerCode: PRVFQY
  databaseName: GFMER Free Medical Journals
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: GX1
  dateStart: 0
  isFulltext: true
  titleUrlDefault: http://www.gfmer.ch/Medical_journals/Free_medical.php
  providerName: Geneva Foundation for Medical Education and Research
– providerCode: PRVHPJ
  databaseName: ROAD: Directory of Open Access Scholarly Resources
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: M~E
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: https://road.issn.org
  providerName: ISSN International Centre
– providerCode: PRVAQN
  databaseName: PubMed Central
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: RPM
  dateStart: 20000101
  isFulltext: true
  titleUrlDefault: https://www.ncbi.nlm.nih.gov/pmc/
  providerName: National Library of Medicine
– providerCode: PRVPQU
  databaseName: Health & Medical Collection
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: 7X7
  dateStart: 20090101
  isFulltext: true
  titleUrlDefault: https://search.proquest.com/healthcomplete
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: ProQuest Central
  customDbUrl: http://www.proquest.com/pqcentral?accountid=15518
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: BENPR
  dateStart: 20090101
  isFulltext: true
  titleUrlDefault: https://www.proquest.com/central
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: ProQuest Technology Collection
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: 8FG
  dateStart: 20090101
  isFulltext: true
  titleUrlDefault: https://search.proquest.com/technologycollection1
  providerName: ProQuest
– providerCode: PRVFZP
  databaseName: Scholars Portal Journals: Open Access
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 20250131
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: M48
  dateStart: 20000701
  isFulltext: true
  titleUrlDefault: http://journals.scholarsportal.info
  providerName: Scholars Portal
– providerCode: PRVAVX
  databaseName: HAS SpringerNature Open Access 2022
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: AAJSJ
  dateStart: 20001201
  isFulltext: true
  titleUrlDefault: https://www.springernature.com
  providerName: Springer Nature
– providerCode: PRVAVX
  databaseName: SpringerOpen Free (Free internet resource, activated by CARLI)
  customDbUrl:
  eissn: 1471-2105
  dateEnd: 99991231
  omitProxy: true
  ssIdentifier: ssj0017805
  issn: 1471-2105
  databaseCode: C6C
  dateStart: 20000112
  isFulltext: true
  titleUrlDefault: http://www.springeropen.com/
  providerName: Springer Nature
link http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwrV3di9NAEF_0RNQH8dvoWVYRBI9w2XzsbnzrlatnwXLcWahPS7qZ1UJNS9Mi9987k6ShUfRefEkgOyHZmdn5YGZ_y9hblUsMwqmtPAycH4OK_ExYhTkPRAJ05JK8Qvscy7NJPJom072jvqgnrIYHrhl3bMMgFoCBs0NPBNKmVGeLcoueDWNx5cj6BjrdJVNN_YCQ-psaptDyuBSE0-aTRUaPFvuy44UqsP4_TfKeT_q9X7Itmt5jd7bFKrv6mS0We35p-IDdbwJK3q8n8pDdgOIRu10fMXn1mP2gaLICY0YiS6Hy3NHp1xyDVZ7XfXb4Dd4UajhtN-Fz2jQCfA0rWG-WaBTLDzzjlsYrOFq-rd7ZNSPyEr-M85yXT9hkePplcOY3Ryz4Fpf6xndp7JC9AjJM2yKnMdTOVOJCZ2MFCtAehHkwA2R6KDMMhuJolsYa0DDoQEJoo6fsoFgW8JxxlxHYng20S3UMDmZ5PgNtLUghlIDcY8GO5cY2-ON0DMbCVHmIlqaWkkEpGZKSkR57376yqsE3_kV8QnJsCQk3u3qA2mQabTLXaZPH3pAWGELGKKj15lu2LUvz6fLC9Cm0wuAvSTz2riFyS5yBzZqdDMgHAtPqUB52KHHp2u7wTtlMYzpKI9IYs1Dkr_DY63aY3qR2uAKWW6KJqFwWKe2xZ7VutvMONSZNmNR7THW0tsOY7kgx_14BiycJupwQf-top997v_V3vh-1S-B6Kb34H1J6ye6GtJaprqcO2cFmvYVXGBtuZj12U00VXvXwY4_d6vdHlyO8n5yOzy_w6UAOepWhwOvnWOPIZHze__oL-jZkdw
linkProvider Directory of Open Access Journals
linkToHtml http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwtR3ZbtNAcFSKUMsDgnIZCiwIhNTKqu91kBAqR5XQ0gdopbwtznq2RAp2iBNV-Sm-kRlfxCDKUx-TnU08O7dndgbguUwjcsK5rNxzjB2g9O3E1ZJiHvRdjH0TpmW3z-Oofxp8HIbDNfjZ3IXhsspGJ5aKOs01vyPfo2CbnHNJCvXN9IfNU6M4u9qM0KjY4hCX5xSyFa8H74m-Lzzv4MPJu75dTxWwNXH33Da9wAQuuphQpOKbmLzLRIbGMzqQKJFEwEudEfbIuEUJ2f_AH_WCGEkWYidCT_v0u1fgauCTLiH5kcM2wHN5PkCdOXXjaK9wuTuczXaA7GhgRx3bV44I-NsQrFjCP6s021TtddhYZNNkeZ5MJivW8OAm3KjdWLFf8d0tWMNsC65Vgy2XW7DJPmzVAvo2fG8_0A7N3vrY8ABuQf6ySKtSP_pDUeeKBN94EWO-t4JihlOczXPSy8UrkQjN62VHXLEo9zT1kKKgxyCkx8UdOL0UetyF9SzP8D4Ik3C_P-3EphcHaHCUpiOMtcbIdaWLqQVOc_5K1y3QeRLHRJWhUBypimSKSKaYZCqyYKfdMq36f1wE_JaJ2gJy6-7yi3x2pmpNoLTnELoUCRrCBSPd48Sxn2py1Si4lMaCZ8wSiptzZFz9c5YsikINvnxW--zdkf8Zhha8rIFMThjopL5MQefA_bw6kNsdSNIeurvccJ6qtVehfsuaBU_bZd7JFXkZ5guG8Tlj58vYgnsVo7Z4ezHFba4jLZAdFu4cTHclG38re5uHIVk9jx5rt2H2lcf697nvtvLwfyo9uBjlJ7DRP_l0pI4Gx4cPYdNj8eUEotyG9flsgY_ICZ2PHpeSL-DrZauaX09clIc
linkToPdf http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwlV1Zb9QwELagiOsBcZZAAYOQkFpFzeHYDm9lYdUCqhBQqW9W1hmXlbbJarMR6r9nJpc2nOI1HiexPadm_A1jL1Uu0QmnsvIocL4AFftZaBXGPBCHoGOX5A3a57E8PBHvT5PTrs9p1Ve79ynJ9k4DoTQV6_1l7loR13K_Cgl3zScNixZK-PIyuyLQuFELg4mcDGkEAuzvUpm_nTYyRg1m_6-aecM0_Vw2OeROb7LrdbHMLr5ni8WGeZreZrc6v5IftIxwh12C4i672naavLjHzsmpbDCZkciSxzx31ASbo8_K87bcDr_Bu3wNp1snfE53R4CvYAmrdYm6sXrNM25pvEGl5XUzp69J5BV-Gdc5r-6zk-m7r5NDv-u04FuU-LXvUuFECCFkGL3FTqPHnanERc4KBQpQLUR5MIMUDb7M0CcS8SwVGlA_6EBCZOMHbKsoC3jIuMsIc88G2qVagINZns9AWwsyDFUIuceCfsuN7WDIqRvGwjThiJamPSWDp2TolIz02O4wZdlicPyN-A2d40BI8NnNg3J1ZjppNDYKcLkYjTlcC0ibUvI2zi26SxjgKeexF8QFhgAyCqrAOcvqqjJHXz6bA_Kw0AdMEo-96ohciSuwWXehAfeBMLVGlDsjSpRgOx7umc10GqQyYSowGMX9DT32fBimmVQVV0BZE01MWbNYaY9tt7w5rDvSGDthbO8xNeLa0caMR4r5twZfPEnQ8kT4W3s9f2_81p_3fW8QgX-f0qP_evczdu3T26n5eHT84TG7EZH8UkpP7bCt9aqGJ-gWrmdPG9H_AbZyWLw
linkToUnpaywall http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwrV3da9RAEB_qFVEf_KwarbKKIFhyzeZrE99OsVQfqqgH9WlJNrs19JoclwtS_3pn8sWlfiH4dtzOkuxkZ_Y3zMxvAZ6JLEQQTmXlrmNsXwvPTrgSGPNoj-vIM0HWsH0ehYdz_91xcLwF7_temPRMpXnZkYYSUfF0sw190fhu_KFO95eZaU0-CvcrTjxsNnlcPLF8O7wE22GA4HwC2_OjD7MvTY-R4DYGOEGX2_zlvNHp1JD4_-yqN86qi3WUQzL1Glypi2Vy_i1ZLDbOq4MbsOxX2papnE7rdTpV3y-QQP5HVdyE6x22ZbN2M96CLV3chsvtbZfnd-CMgG3DC41CilB7bugiboaPZllb8ofLYl3OiFHnC8upf0WzlV7q1bpE_1y9ZAlTNN4w47K6mdPXRbIKn4yqzasdmB-8-fz60O5ue7AVep21bWLf-FxznWAE6ZkIUX8iAuMa5QstNLomN3NSHSPoCBPEZb6Xxn6k0UdFTqhd5d2FSVEW-j4wkxDvn3IiE0e-NjrNslRHSumQc8F1ZoHTf2WpOip0upFjIZuQKAplq0WJWpSkRRla8GKYsmx5QP4k_Iq2ziBIFN7NH-XqRHYeQSrXweViRGhwLTpUMSWQvUwhZMMgUxgLntLGk0TSUVAV0ElSV5V8--mjnBHKQxwaBBY874RMSXsh6ZoqUA_E6zWS3B1JohdR4-F-f8vOi1WSxz4GxKhfbsGTYZhmUmVeocuaZDzK3HkisuBeaw7Dut0I4zfuCAvEyFBGihmPFPnXhuM8CPD0c_G19nqT2nit3-t9b7C6v3-lB_8k_RCuumRUlEsUuzBZr2r9CPHoOn3c-ZgfRuOD_w
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Statistical+classifiers+for+diagnosing+disease+from+immune+repertoires%3A+a+case+study+using+multiple+sclerosis&rft.jtitle=BMC+bioinformatics&rft.au=Jared+Ostmeyer&rft.au=Scott+Christley&rft.au=William+H.+Rounds&rft.au=Inimary+Toby&rft.date=2017-09-07&rft.pub=BMC&rft.eissn=1471-2105&rft.volume=18&rft.issue=1&rft.spage=1&rft.epage=10&rft_id=info:doi/10.1186%2Fs12859-017-1814-6&rft.externalDBID=DOA&rft.externalDocID=oai_doaj_org_article_c2041e217f124e6c902753dc7826267f
thumbnail_l http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=1471-2105&client=summon
thumbnail_m http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=1471-2105&client=summon
thumbnail_s http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=1471-2105&client=summon