Pancreatic RECK inactivation promotes cancer formation, epithelial-mesenchymal transition, and metastasis - Masuda, Tomonori; Fukuda, Akihisa; Yamakawa, Go; Omatsu, Mayuki; Namikawa, Mio; Sono, Makoto; Fukunaga, Yuichi; Nagao, Munemasa; Araki, Osamu; Yoshikawa, Takaaki; Ogawa, Satoshi; Masuo, Kenji; et al. (The Journal of clinical investigation, 2023)
Keywords: Acute phase proteins; Adenocarcinoma; Age; Analysis; Animals; Biomedical research; Cadherins - genetics; Carcinogenesis; Carcinoma, Pancreatic Ductal - genetics; Development and progression; DNA binding proteins; E-cadherin; Epithelial-Mesenchymal Transition - genetics; Gastroenterology; Gelatinase A; Genetic aspects; Genotype & phenotype; GPI-Linked Proteins - genetics; Health aspects; Humans; Inflammation; Liver; Liver cancer; Liver Neoplasms - genetics; Medical prognosis; Metastases; Metastasis; Mice; Morphology; Oncology; Pancreas; Pancreatic cancer; Pancreatic Neoplasms; Pancreatic Neoplasms - genetics; Phenotypes; Physiological aspects; Proteins; Scientific equipment and supplies industry; Stem cells; Transcription factors; Transplantation; Tumor suppressor genes; Tumors; Japan; United States; Oncology; Mouse models; Tumor suppressors; Gastroenterology; Cancer