Serotonin signals through a gut-liver axis to regulate hepatic steatosis - Choi, Wonsuk; Namkung, Jun; Hwang, Inseon; Kim, Hyeongseok; Lim, Ajin; Park, Hye Jung; Lee, Hye Won; Han, Kwang-Hyub; Park, Seongyeol; Jeong, Ji-Seon; Bang, Geul; Kim, Young Hwan; et al. (Nature communications, 2018)
Keywords: 14; 14/63; 38; 38/77; 38/91; 631/443/319/1642; 64; 64/60; 692/4020/4021/1607/2750; Animals; Diet, High-Fat - adverse effects; Disease Models, Animal; Energy balance; Epidemics; Fatty liver; Gene Expression Profiling; Gene Expression Regulation; High fat diet; Homeostasis; Humanities and Social Sciences; Humans; Hypolipidemic Agents - pharmacology; Insulin Resistance; Intestinal Mucosa - metabolism; Intestinal Mucosa - pathology; Lipid Metabolism; Liver; Liver - metabolism; Liver - pathology; Liver diseases; Male; Mice; Mice, Knockout; multidisciplinary; Non-alcoholic Fatty Liver Disease - etiology; Non-alcoholic Fatty Liver Disease - genetics; Non-alcoholic Fatty Liver Disease - pathology; Non-alcoholic Fatty Liver Disease - prevention & control; Pathogenesis; Receptor, Serotonin, 5-HT2A - deficiency; Receptor, Serotonin, 5-HT2A - genetics; Rodents; Science; Science (multidisciplinary); Serotonin; Serotonin - metabolism; Serotonin Antagonists - pharmacology; Signal Transduction; Steatosis; Succinates - pharmacology; Tryptophan Hydroxylase - deficiency; Tryptophan Hydroxylase - genetics