Resolvin E1, an endogenous lipid mediator derived from eicosapentaenoic acid, prevents dextran sulfate sodium–induced colitis - Ishida, Tsukasa; Yoshida, Masaru; Arita, Makoto; Nishitani, Yosuke; Nishiumi, Shin; Masuda, Atsuhiro; Mizuno, Shigeto; Takagawa, Tetsuya; Morita, Yoshinori; Kutsumi, Hiromu; Inokuchi, Hideto; Serhan, Charles N.; et al. (Inflammatory bowel diseases, 2010)
Keywords: Animals; Cell Nucleus - metabolism; Cells, Cultured; Colitis - chemically induced; Colitis - immunology; Colitis - prevention & control; Cytokines; Dextran Sulfate - toxicity; DSS‐induced colitis; Eicosapentaenoic Acid - analogs & derivatives; Eicosapentaenoic Acid - pharmacology; Female; Flow Cytometry; Humans; Immunoenzyme Techniques; Inflammation; Inflammation - immunology; Inflammation - pathology; Inflammation - prevention & control; Inflammation Mediators - metabolism; Inflammatory bowel disease; Kidney - cytology; Kidney - metabolism; macrophage; Macrophages, Peritoneal - drug effects; Macrophages, Peritoneal - immunology; Macrophages, Peritoneal - pathology; Mice; Mice, Inbred C57BL; NF-kappa B - genetics; NF-kappa B - metabolism; NF‐κB; Protein Transport; Receptors, Chemokine - metabolism; resolvin E1; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger - genetics; RNA, Messenger - metabolism; NF-κB; macrophage; DSS-induced colitis; resolvin E1