Contributions of DNA repair, cell cycle checkpoints and cell death to suppressing the DNA damage-induced tumorigenic behavior of Drosophila epithelial cells - Dekanty, A; Barrio, L; Milán, M (Oncogene, 2015)
Keywords: 631/337/1427; 631/67; 631/80/641; 631/80/82/23; Animal models; Animals; Animals, Genetically Modified; Apoptosis; Cancer; Carcinogenesis; Carcinogenesis - genetics; Carcinogenesis - radiation effects; Cell Biology; Cell culture; Cell cycle; Cell Cycle Checkpoints - physiology; Cell death; Cell Death - physiology; Cellular biology; Deoxyribonucleic acid; DNA; DNA Breaks, Double-Stranded; DNA damage; DNA Damage - physiology; DNA repair; DNA Repair - physiology; Drosophila; Drosophila melanogaster - genetics; E-cadherin; Epithelial cells; Epithelial Cells - metabolism; Epithelial Cells - pathology; Epithelial Cells - radiation effects; G2 Phase; Genetic aspects; Genetic research; Homologous recombination; Human Genetics; Insects; Internal Medicine; Ionizing radiation; LIG4 protein; Medicine; Medicine & Public Health; Membrane degradation; Mutation; Oncology; original-article; Radiation, Ionizing; Studies; Tissues; Tumor suppressor genes; Vitaceae