NF-κB-responsive miR-155 induces functional impairment of vascular smooth muscle cells by downregulating soluble guanylyl cyclase - Park, Minsik; Choi, Seunghwan; Kim, Suji; Kim, Joohwan; Lee, Dong-Keon; Park, Wonjin; Kim, Taesam; Jung, Jiwon; Hwang, Jong Yun; Won, Moo-Ho; Ryoo, Sungwoo; Kang, Seung Goo; et al. (Experimental & molecular medicine, 2019)
Keywords: 13/89; 14; 14/19; 631/136/1425; 631/337/505; 631/80/84; 64/60; 82/80; 96/21; Animals; Aorta; Apolipoprotein E; Arteriosclerosis; Atherosclerosis; Biomedical and Life Sciences; Biomedicine; Cell migration; Cell proliferation; Cyclic GMP; Cyclic GMP - metabolism; Cytology; Gene Expression Regulation - drug effects; Guanylate cyclase; High fat diet; Humans; Hyperplasia; Male; Medical Biochemistry; Mice; Mice, Knockout; MicroRNAs - genetics; miRNA; Molecular Medicine; Muscle contraction; Muscle, Smooth, Vascular - cytology; Myocytes, Smooth Muscle - metabolism; NF-kappa B - metabolism; NF-κB protein; Nitric oxide; Nitric Oxide - metabolism; Phenotype; Phenotypes; Pre-eclampsia; Preeclampsia; RNA Interference; Smooth muscle; Soluble Guanylyl Cyclase - genetics; Soluble Guanylyl Cyclase - metabolism; Stem Cells; Therapeutic applications; Transcription; Tumor Necrosis Factor-alpha - metabolism; Tumor Necrosis Factor-alpha - pharmacology; Tumor necrosis factor-TNF; Tumor necrosis factor-α; Vasodilation; 생화학