Eukaryotic Elongation Factor 2 Kinase Activity Is Controlled by Multiple Inputs from Oncogenic Signaling - Wang, Xuemin; Regufe da Mota, Sergio; Liu, Rui; Moore, Claire E.; Xie, Jianling; Lanucara, Francesco; Agarwala, Usha; Pyr dit Ruys, Sébastien; Vertommen, Didier; Rider, Mark H.; Eyers, Claire E.; Proud, Christopher G. (Molecular and cellular biology, 2014)
Keywords: active sites; Animals; cell growth; Cell Line; Cell Line, Tumor; cell viability; Elongation Factor 2 Kinase - genetics; Elongation Factor 2 Kinase - metabolism; Enzyme Activation; Extracellular Signal-Regulated MAP Kinases - metabolism; Humans; MAP Kinase Signaling System; Mechanistic Target of Rapamycin Complex 1; Mice; mitogen-activated protein kinase; Multiprotein Complexes - metabolism; Neoplasms - genetics; Neoplasms - metabolism; peptide elongation factors; Phosphorylation; Point Mutation; protein synthesis; raf Kinases - metabolism; ras Proteins - metabolism; Rats; Signal Transduction; starvation; tau-protein kinase; TOR Serine-Threonine Kinases - metabolism