Increased α2-6 sialylation of endometrial cells contributes to the development of endometriosis - Choi, Hee-Jin; Chung, Tae-Wook; Choi, Hee-Jung; Han, Jung Ho; Choi, Jung-Hye; Kim, Cheorl-Ho; Ha, Ki-Tae (Experimental & molecular medicine, 2018)
Keywords: 13/109; 13/21; 13/31; 13/89; 38/1; 38/77; 38/90; 631/337/458/1524; 631/80/304; 631/80/79; 692/699/2732/2730; Animals; Biomedical and Life Sciences; Biomedicine; Cell adhesion; Cell adhesion & migration; Cell Line, Tumor; Cytokines; Disease Models, Animal; Endometriosis; Endometriosis - metabolism; Endometrium; Endometrium - pathology; Epithelium - pathology; Epitopes; Female; Focal Adhesions; Humans; Infertility; Medical Biochemistry; Mesothelium; Mice; Mice, Inbred C57BL; Molecular Medicine; N-Acetylneuraminic Acid - metabolism; Outbreaks; Peritoneum; Peritoneum - pathology; RNA, Small Interfering - genetics; Sialic Acid Binding Immunoglobulin-like Lectins - metabolism; Sialyltransferases - genetics; Sialyltransferases - metabolism; Signal Transduction; Smad2 protein; Smad2 Protein - metabolism; Smad3 Protein - metabolism; Stem Cells; Transforming growth factor; Transforming Growth Factor beta1 - metabolism; Transforming growth factor-b1; Uterus; 생화학