E2F1 interacts with BCL‐xL and regulates its subcellular localization dynamics to trigger cell death - Vuillier, Céline; Lohard, Steven; Fétiveau, Aurélie; Allègre, Jennifer; Kayaci, Cémile; King, Louise E; Braun, Frédérique; Barillé‐Nion, Sophie; Gautier, Fabien; Dubrez, Laurence; Gilmore, Andrew P; Juin, Philippe P; et al. (EMBO reports, 2018)
Keywords: Apoptosis; bcl-2 Homologous Antagonist-Killer Protein - metabolism; Bcl-x protein; bcl-X Protein - chemistry; bcl-X Protein - metabolism; BCL‐2 family; BCL‐xL mobility; Binding; Cancer; Cell Death; Cell Line, Tumor; E2F1; E2F1 Transcription Factor - chemistry; E2F1 Transcription Factor - metabolism; EMBO07; Extracellular Space - metabolism; Gene Expression Regulation - drug effects; Humans; Life Sciences; Localization; Mitochondria; Mitochondria - metabolism; Mobility; Mortality; Protein Binding; Protein Transport; Proteins; Proto-Oncogene Proteins c-bcl-2 - genetics; Proto-Oncogene Proteins c-bcl-2 - metabolism; Scientific Report; Scientific Reports; Transcription; Transcription, Genetic; Tumor suppressor genes; E2F1; BCL‐2 family; apoptosis; BCL‐xL mobility; BCL-xL mobility; BCL-2 family; E2F1 Subject Category Autophagy & Cell Death