Cannabinoid signaling modulation through JZL184 restores key phenotypes of a mouse model for Williams–Beuren syndrome - Lorena Galera-López; Alba Navarro-Romero; Paula Ortiz-Romero; Alberto Llorente-Ovejero; Lucía de los Reyes-Ramírez; Iker Bengoetxea de Tena; Anna Garcia-Elias; Aleksandra Mas-Stachurska; Marina Reixachs-Solé; Antoni Pastor; Rafael de la Torre; Rafael Maldonado; et al. (eLife, 2022)
Keywords: Animals; Behavior; Benzodioxoles; Biology (General); Brain; Cannabinoid receptors; cannabinoid type-1 receptor; Cannabinoids; Cannabis; Cognition & reasoning; Cognitive ability; Cànnabis; Disease Models, Animal; Drug therapy; Endocannabinoid system; Endocannabinoids; Enzymes; Gene expression; Genotype & phenotype; Hypertension; Intellectual disabilities; Intellectual disability; Ligands; Male; Medicine; Memory; Mice; Monoacylglycerol Lipases; Mouse; Parkinson's disease; People with mental disabilities; Persones amb discapacitat mental; Phenotype; Phenotypes; Piperidines; Q; QH301-705.5; R; Ratolins; Rodents; Science; Statistical significance; Síndrome de Williams; Variance analysis; Williams Syndrome; Williams-Beuren syndrome