Human plasma vitamin E kinetics demonstrate rapid recycling of plasma RRR-alpha-tocopherol

A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR-and SRR-). In normal subjects, both alpha-tocopherols are present at similar concentrations in chylomicrons, but by 24 hr, RRR-alpha-tocopherol is a...

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Published inProceedings of the National Academy of Sciences - PNAS Vol. 91; no. 21; pp. 10005 - 10008
Main Authors Traber, M.G. (University of California, Berkeley, CA.), Ramakrishnan, R, Kayden, H.J
Format Journal Article
LanguageEnglish
Published United States National Academy of Sciences of the United States of America 11.10.1994
National Acad Sciences
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Online AccessGet full text
ISSN0027-8424
1091-6490
1091-6490
DOI10.1073/pnas.91.21.10005

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Abstract A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR-and SRR-). In normal subjects, both alpha-tocopherols are present at similar concentrations in chylomicrons, but by 24 hr, RRR-alpha-tocopherol is at higher plasma concentrations because RRR-alpha-tocopherol is preferentially incorporated into very low density lipoproteins, which are then secreted into plasma. In three nondiscriminator patients with familial isolated vitamin E deficiency, the fractional disappearance rates (mean +/-SD) of deuterium-labeled RRR- and SRR-alpha-tocopherols in plasma were 1.4 +/- 0.6 and 1.3 +/- 0.3 pools per day, respectively (difference, 0.1 +/- 0.3). In these patients, plasma concentrations of both RRR- and SRR-alpha-tocopherols decreased similarly to SRR-alpha-tocopherol in controls. In six controls, fractional disappearance rates of deuterium-labeled RRR-alpha-tocopherol (0.4 +/- 0.1 pool per day) were significantly (P 0.01) slower than for SRR- (1.2 +/-0.6). The differences (0.8 +/- 0.6 pool per day) between these two rates in controls estimate the rate at which RRR-alpha-tocopherol, which had left the plasma, was returned to the plasma. Although plasma labeled RRR-alpha-tocopherol concentrations in controls appear to change slowly, these data show that both RRR- and SRR-alpha-tocopherols leave the plasma rapidly, but only RRR-alpha-tocopherol is returned to the plasma, likely in nascent very low density lipoproteins. This recycling of RRR-alpha-tocopherol accounts for nearly 1 pool of alpha-tocopherol per day
AbstractList A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR- and SRR-). In normal subjects, both alpha-tocopherols are present at similar concentrations in chylomicrons, but by 24 hr, RRR-alpha-tocopherol is at higher plasma concentrations because RRR-alpha-tocopherol is preferentially incorporated into very low density lipoproteins, which are then secreted into plasma. In three nondiscriminator patients with familial isolated vitamin E deficiency, the fractional disappearance rates (mean +/- SD) of deuterium-labeled RRR- and SRR-alpha-tocopherols in plasma were 1.4 +/- 0.6 and 1.3 +/- 0.3 pools per day, respectively (difference, 0.1 +/- 0.3). In these patients, plasma concentrations of both RRR- and SRR-alpha-tocopherols decreased similarly to SRR-alpha-tocopherol in controls. In six controls, fractional disappearance rates of deuterium-labeled RRR-alpha-tocopherol (0.4 +/- 0.1 pool per day) were significantly (P < 0.01) slower than for SRR- (1.2 +/- 0.6). The differences (0.8 +/- 0.6 pool per day) between these two rates in controls estimate the rate at which RRR-alpha-tocopherol, which had left the plasma, was returned to the plasma. Although plasma labeled RRR-alpha-tocopherol concentrations in controls appear to change slowly, these data show that both RRR- and SRR-alpha-tocopherols leave the plasma rapidly, but only RRR-alpha-tocopherol is returned to the plasma, likely in nascent very low density lipoproteins. This recycling of RRR-alpha-tocopherol accounts for nearly 1 pool of alpha-tocopherol per day.
A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of α-tocopherol (RRR- and SRR-). In normal subjects, both α-tocopherols are present at similar concentrations in chylomicrons, but by 24 hr, RRR-α-tocopherol is at higher plasma concentrations because RRR-α-tocopherol is preferentially incorporated into very low density lipoproteins, which are then secreted into plasma. In three nondiscriminator patients with familial isolated vitamin E deficiency, the fractional disappearance rates (mean ± SD) of deuterium-labeled RRR- and SRR-α-tocopherols in plasma were 1.4 ± 0.6 and 1.3 ± 0.3 pools per day, respectively (difference, 0.1 ± 0.3). In these patients, plasma concentrations of both RRR- and SRR-α-tocopherols decreased similarly to SRR-α-tocopherol in controls. In six controls, fractional disappearance rates of deuterium-labeled RRR-α-tocopherol (0.4 ± 0.1 pool per day) were significantly (P < 0.01) slower than for SRR- (1.2 ± 0.6). The differences (0.8 ± 0.6 pool per day) between these two rates in controls estimate the rate at which RRR-α-tocopherol, which had left the plasma, was returned to the plasma. Although plasma labeled RRR-α-tocopherol concentrations in controls appear to change slowly, these data show that both RRR- and SRR-α-tocopherols leave the plasma rapidly, but only RRR-α-tocopherol is returned to the plasma, likely in nascent very low density lipoproteins. This recycling of RRR-α-tocopherol accounts for nearly 1 pool of α-tocopherol per day.
A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR-and SRR-). In normal subjects, both alpha-tocopherols are present at similar concentrations in chylomicrons, but by 24 hr, RRR-alpha-tocopherol is at higher plasma concentrations because RRR-alpha-tocopherol is preferentially incorporated into very low density lipoproteins, which are then secreted into plasma. In three nondiscriminator patients with familial isolated vitamin E deficiency, the fractional disappearance rates (mean +/-SD) of deuterium-labeled RRR- and SRR-alpha-tocopherols in plasma were 1.4 +/- 0.6 and 1.3 +/- 0.3 pools per day, respectively (difference, 0.1 +/- 0.3). In these patients, plasma concentrations of both RRR- and SRR-alpha-tocopherols decreased similarly to SRR-alpha-tocopherol in controls. In six controls, fractional disappearance rates of deuterium-labeled RRR-alpha-tocopherol (0.4 +/- 0.1 pool per day) were significantly (P 0.01) slower than for SRR- (1.2 +/-0.6). The differences (0.8 +/- 0.6 pool per day) between these two rates in controls estimate the rate at which RRR-alpha-tocopherol, which had left the plasma, was returned to the plasma. Although plasma labeled RRR-alpha-tocopherol concentrations in controls appear to change slowly, these data show that both RRR- and SRR-alpha-tocopherols leave the plasma rapidly, but only RRR-alpha-tocopherol is returned to the plasma, likely in nascent very low density lipoproteins. This recycling of RRR-alpha-tocopherol accounts for nearly 1 pool of alpha-tocopherol per day
A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR- and SRR-). In normal subjects, both alpha-tocopherols are present at similar concentrations in chylomicrons, but by 24 hr, RRR-alpha-tocopherol is at higher plasma concentrations because RRR-alpha-tocopherol is preferentially incorporated into very low density lipoproteins, which are then secreted into plasma. In three nondiscriminator patients with familial isolated vitamin E deficiency, the fractional disappearance rates (mean +/- SD) of deuterium-labeled RRR- and SRR-alpha-tocopherols in plasma were 1.4 +/- 0.6 and 1.3 +/- 0.3 pools per day, respectively (difference, 0.1 +/- 0.3). In these patients, plasma concentrations of both RRR- and SRR-alpha-tocopherols decreased similarly to SRR-alpha-tocopherol in controls. In six controls, fractional disappearance rates of deuterium-labeled RRR-alpha-tocopherol (0.4 +/- 0.1 pool per day) were significantly (P < 0.01) slower than for SRR- (1.2 +/- 0.6). The differences (0.8 +/- 0.6 pool per day) between these two rates in controls estimate the rate at which RRR-alpha-tocopherol, which had left the plasma, was returned to the plasma. Although plasma labeled RRR-alpha-tocopherol concentrations in controls appear to change slowly, these data show that both RRR- and SRR-alpha-tocopherols leave the plasma rapidly, but only RRR-alpha-tocopherol is returned to the plasma, likely in nascent very low density lipoproteins. This recycling of RRR-alpha-tocopherol accounts for nearly 1 pool of alpha-tocopherol per day.A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR- and SRR-). In normal subjects, both alpha-tocopherols are present at similar concentrations in chylomicrons, but by 24 hr, RRR-alpha-tocopherol is at higher plasma concentrations because RRR-alpha-tocopherol is preferentially incorporated into very low density lipoproteins, which are then secreted into plasma. In three nondiscriminator patients with familial isolated vitamin E deficiency, the fractional disappearance rates (mean +/- SD) of deuterium-labeled RRR- and SRR-alpha-tocopherols in plasma were 1.4 +/- 0.6 and 1.3 +/- 0.3 pools per day, respectively (difference, 0.1 +/- 0.3). In these patients, plasma concentrations of both RRR- and SRR-alpha-tocopherols decreased similarly to SRR-alpha-tocopherol in controls. In six controls, fractional disappearance rates of deuterium-labeled RRR-alpha-tocopherol (0.4 +/- 0.1 pool per day) were significantly (P < 0.01) slower than for SRR- (1.2 +/- 0.6). The differences (0.8 +/- 0.6 pool per day) between these two rates in controls estimate the rate at which RRR-alpha-tocopherol, which had left the plasma, was returned to the plasma. Although plasma labeled RRR-alpha-tocopherol concentrations in controls appear to change slowly, these data show that both RRR- and SRR-alpha-tocopherols leave the plasma rapidly, but only RRR-alpha-tocopherol is returned to the plasma, likely in nascent very low density lipoproteins. This recycling of RRR-alpha-tocopherol accounts for nearly 1 pool of alpha-tocopherol per day.
Author Ramakrishnan, R
Traber, M.G. (University of California, Berkeley, CA.)
Kayden, H.J
AuthorAffiliation Department of Medicine, New York University School of Medicine, NY 10016
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BackLink https://www.ncbi.nlm.nih.gov/pubmed/7937827$$D View this record in MEDLINE/PubMed
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Snippet A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR-and...
A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of α-tocopherol (RRR- and SRR-)....
A kinetic model of vitamin E transport in humans is described using data from our studies with deuterium-labeled stereoisomers of alpha-tocopherol (RRR- and...
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SubjectTerms administration & dosage
Administration, Oral
alpha-tocopherol
blood
Blood plasma
CARENCE EN VITAMINE
Carrier proteins
CATABOLISME
CATABOLISMO
CHYLOMICRON
chylomicrons
DEFICIENCIA DE VITAMINAS
Deuterium
enantiomers
ESTRUCTURA QUIMICA
GENERO HUMANO
GENRE HUMAIN
Humans
Intestinal Absorption
Kinetics
Lipids
LIPOPROTEINAS
LIPOPROTEINE
Lipoproteins
Liver
Liver - metabolism
mathematical models
metabolism
METABOLISME
METABOLISMO
MODELE MATHEMATIQUE
MODELOS MATEMATICOS
Models, Biological
pharmacokinetics
PLASMA SANGUIN
PLASMA SANGUINEO
QUILOMICRONES
Radioisotope Dilution Technique
Secretion
STRUCTURE CHIMIQUE
Time Factors
TOCOFEROLES
TOCOPHEROL
Tocopherols
very low density lipoprotein
vitamin deficiencies
Vitamin E
Vitamin E - administration & dosage
Vitamin E - blood
Vitamin E - pharmacokinetics
VITAMINA E
VITAMINE E
Title Human plasma vitamin E kinetics demonstrate rapid recycling of plasma RRR-alpha-tocopherol
URI https://www.jstor.org/stable/2365751
http://www.pnas.org/content/91/21/10005.abstract
https://www.ncbi.nlm.nih.gov/pubmed/7937827
https://www.proquest.com/docview/47960167
https://www.proquest.com/docview/76768378
https://pubmed.ncbi.nlm.nih.gov/PMC44946
https://pnas.org/doi/full/10.1073/pnas.91.21.10005
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Volume 91
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