Truncation of Tau selectively facilitates its pathological activities - Gu, Jianlan; Xu, Wen; Jin, Nana; Li, Longfei; Zhou, Yan; Chu, Dandan; Gong, Cheng-Xin; Iqbal, Khalid; Liu, Fei (The Journal of biological chemistry, 2020)
Keywords: aggregation; Alzheimer Disease - genetics; Alzheimer Disease - metabolism; Alzheimer's disease; Amino Acid Sequence; Animals; HEK293 Cells; HeLa Cells; Humans; Mice; Molecular Bases of Disease; pathogenesis; phosphorylation; prion-like activity; protein aggregation; Rats; Sequence Deletion; Tau protein (Tau); tau Proteins - genetics; tau Proteins - metabolism; truncation; pathogenesis; Tau protein (Tau); protein aggregation; aggregation; Alzheimer's disease; truncation; phosphorylation; prion-like activity