Apobec3A maintains HIV-1 latency through recruitment of epigenetic silencing machinery to the long terminal repeat - Taura, Manabu; Song, Eric; Ho, Ya-Chi; Iwasaki, Akiko (Proceedings of the National Academy of Sciences - PNAS, 2019)
Keywords: Activation; Apolipoprotein B; Biological Sciences; Catalysis; CD4 antigen; CD4-Positive T-Lymphocytes - metabolism; CD4-Positive T-Lymphocytes - virology; Cell Line; Cell lines; Cytidine Deaminase - chemistry; Cytidine Deaminase - metabolism; Deoxyribonucleic acid; DNA; Epigenesis, Genetic; Gene Expression Regulation, Viral; Gene Silencing; Genomes; HIV; HIV Infections - metabolism; HIV Infections - virology; HIV Long Terminal Repeat; HIV-1 - physiology; Human immunodeficiency virus; Humans; Latency; Latent infection; Long terminal repeat; Lymphocytes; Lymphocytes T; mRNA; NF-kappa B - metabolism; PNAS Plus; Protein Binding; Protein Interaction Domains and Motifs; Proteins - chemistry; Proteins - metabolism; RNA editing; Sequence Deletion; Sp1 Transcription Factor - metabolism; Virus Activation - genetics; Virus Latency; reactivation; viral latency; TRIM28; T cells; epigenetic regulation