Polymorphisms of the ADAM33 gene are associated with accelerated lung function decline in asthma - Jongepier, H.; Boezen, H. M.; Dijkstra, A.; Howard, T. D.; Vonk, J. M.; Koppelman, G. H.; Zheng, S. L.; Meyers, D. A.; Bleecker, E. R.; Postma, D. S. (Clinical and experimental allergy, 2004)
Keywords: ADAM Proteins; Adolescent; Adult; Aging - physiology; Allergic diseases; Asthma - genetics; Asthma - physiopathology; Biological and medical sciences; bronchial asthma; Chronic obstructive pulmonary disease, asthma; Disease Progression; FEV1; Forced Expiratory Volume; Genetic Predisposition to Disease; Humans; Immunopathology; Longitudinal Studies; Lung - physiopathology; Medical sciences; Metalloendopeptidases - genetics; Pneumology; Polymorphism, Genetic; Polymorphism, Single Nucleotide; SNP; Lung disease; Allergy; Immunopathology; Respiratory disease; Genotype; disease progression; Respiratory system; Asthma; Respiratory tract; Lung function; Immunology; Gene; SNP; Bronchus disease; Genetics; bronchial asthma; FEV1; Obstructive pulmonary disease; longitudinal studies