The hexosamine signaling pathway: O-GlcNAc cycling in feast or famine - Hanover, John A.; Krause, Michael W.; Love, Dona C. (Biochimica et biophysica acta, 2010)
Keywords: Acetylglucosamine - physiology; Acetylglucosaminidase - metabolism; AKT; AMPK; Animals; Caenorhabditis elegans; Cancer; Cardiovascular disease; Catalytic Domain; Diabetes; Diabetes Mellitus, Type 2 - physiopathology; Evolution, Molecular; Food; Gene Expression Regulation - physiology; Growth factor; Hexosamine; Humans; Insulin; Insulin Resistance - physiology; MAP Kinase Signaling System - physiology; MAPK; Models, Animal; Models, Molecular; mTOR; N-Acetylglucosaminyltransferases - metabolism; Neurodegeneration; O-GlcNAc; Proteasome Endopeptidase Complex - physiology; Proto-Oncogene Proteins c-akt - physiology; Sirtuins - physiology; Starvation; Neurodegeneration; mTOR; Cardiovascular disease; AMPK; Hexosamine; AKT; Diabetes; MAPK; Insulin; O-GlcNAc; Growth factor; Cancer