Bone Morphogenetic Protein 9 Reduces Cardiac Fibrosis and Improves Cardiac Function in Heart Failure - Morine, Kevin J.; Qiao, Xiaoying; York, Sam; Natov, Peter S.; Paruchuri, Vikram; Zhang, Yali; Aronovitz, Mark J.; Karas, Richard H.; Kapur, Navin K. (Circulation (New York, N.Y.), 2018)
Keywords: Animals; Disease Models, Animal; Endoglin - deficiency; Endoglin - genetics; Endoglin - metabolism; Fibroblasts - metabolism; Fibroblasts - pathology; Fibrosis; Growth Differentiation Factor 2 - deficiency; Growth Differentiation Factor 2 - genetics; Growth Differentiation Factor 2 - metabolism; Growth Differentiation Factors - genetics; Growth Differentiation Factors - metabolism; Haploinsufficiency; Heart Failure - genetics; Heart Failure - metabolism; Heart Failure - pathology; Heart Failure - physiopathology; Humans; Mice, Inbred C57BL; Mice, Knockout; Myocardium - metabolism; Myocardium - pathology; Phosphorylation; Recovery of Function; Signal Transduction; Smad1 Protein - metabolism; Smad3 Protein - metabolism; Ventricular Function, Left; Ventricular Remodeling; heart failure; fibrosis; endoglin; transforming growth factor beta; growth differentiation factor 2