Suppression of SHIP2 contributes to tumorigenesis and proliferation of gastric cancer cells via activation of Akt - Ye, Yan; Ge, Yan Mei; Xiao, Miao Miao; Guo, Li Mei; Li, Qun; Hao, Ji Qing; Da, Jie; Hu, Wang Lai; Zhang, Xu Dong; Xu, Jiegou; Zhang, Lin Jie (Journal of gastroenterology, 2016)
Keywords: Abdominal Surgery; Analysis; Animals; Apoptosis; Cancer; Cancer cells; Cell Proliferation; Cell Survival - physiology; Cell Transformation, Neoplastic - metabolism; Cell Transformation, Neoplastic - pathology; Colorectal Surgery; Down-Regulation; Gastric Mucosa - cytology; Gastric Mucosa - metabolism; Gastroenterology; Gene Expression Regulation, Neoplastic; Gene Knockdown Techniques; Hepatology; Heterografts; Humans; Immunohistochemistry; Inositol; Medicine; Medicine & Public Health; Mice, Nude; Neoplasm Invasiveness; Neoplasm Transplantation; Oncology, Experimental; Original Article—Alimentary Tract; Phosphatases; Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases; Phosphoric Monoester Hydrolases - genetics; Phosphoric Monoester Hydrolases - metabolism; Phosphoric Monoester Hydrolases - physiology; Proto-Oncogene Proteins c-akt - physiology; Signal Transduction - physiology; Stomach cancer; Stomach Neoplasms - genetics; Stomach Neoplasms - metabolism; Stomach Neoplasms - pathology; Surgical Oncology; Tumor Cells, Cultured; Tumorigenesis; Akt; Proliferation; SHIP2; Gastric cancer