Selenoprotein T Promotes Proliferation and G1-to-S Transition in SK-N-SH Cells: Implications in Parkinson's Disease - Shao, Zi-Qiang; Zhang, Xiong; Fan, Hui-Hui; Wang, Xiao-Shuang; Wu, Hong-Mei; Zhang, Li; Cheng, Wen-Hsing; Zhu, Jian-Hong (The Journal of nutrition, 2019)
Keywords: 6-Hydroxydopamine; acetylcysteine; Aged; Animals; Annexin V; Antioxidants; Blood; brain; Calcium; Calcium - metabolism; Calcium channel blockers; Calcium channels; Caspase-3; Cell cycle; cell cycle checkpoints; Cell Line; Cell proliferation; Cell Proliferation - physiology; Cyclin-dependent kinase; Cyclin-dependent kinase 4; Cyclin-dependent kinase inhibitor p27; Cysteine; Dopamine receptors; Female; Flow cytometry; Forkhead protein; G1 Phase - physiology; Humans; Hydroxylase; Immunoblotting; Iodides; Kinases; Leukocytes (mononuclear); Male; males; Mice; Mice, Inbred C57BL; Middle Aged; Movement disorders; mRNA; Neurodegenerative diseases; Neurons; Nimodipine; nutrition; oxidative stress; Parkinson disease; Parkinson Disease - metabolism; Parkinson Disease - pathology; Parkinson's disease; Proteins; Reactive oxygen species; Reactive Oxygen Species - metabolism; S Phase - physiology; Selenium; selenoprotein; selenoproteins; Selenoproteins - physiology; Tyrosine; Up-Regulation; Vitamins; Parkinson's disease; CDKN1B; 6-OHDA; CDK; FOXO3; MPP; SELENOH; selenium; CCND1; siSELT; CDKN2D; calcium; CDKN2C; GPX1; GPX4; TXNRD1; siRNA; PBMC; selenoprotein; NAC; PD; CCNE1; EdU; ROS; SELENOP; SELENOT; oxidative stress