A single 1-h bout of evening exercise increases basal FFA flux without affecting VLDL-triglyceride and VLDL-apolipoprotein B-100 kinetics in untrained lean men
1 Washington University School of Medicine, St. Louis, Missouri; 2 Department of Nutrition and Dietetics, Harokopio University, Athens, Greece Submitted 22 November 2006 ; accepted in final form 29 January 2007 Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol E...
Saved in:
Published in | American journal of physiology: endocrinology and metabolism Vol. 292; no. 6; pp. E1568 - E1574 |
---|---|
Main Authors | , , , |
Format | Journal Article |
Language | English |
Published |
United States
American Physiological Society
01.06.2007
|
Subjects | |
Online Access | Get full text |
ISSN | 0193-1849 1522-1555 |
DOI | 10.1152/ajpendo.00636.2006 |
Cover
Abstract | 1 Washington University School of Medicine, St. Louis, Missouri; 2 Department of Nutrition and Dietetics, Harokopio University, Athens, Greece
Submitted 22 November 2006
; accepted in final form 29 January 2007
Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were 55% greater ( P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1 ) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB-100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2 ) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3 ) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver.
hepatic lipid metabolism; lipoprotein; stable isotope; tracer
Address for reprint requests and other correspondence: B. Mittendorfer, Washington Univ. School of Medicine, Division of Geriatrics and Nutritional Science, 660 South Euclid Ave.; Campus Box 8031, St. Louis, MO 63110 (e-mail: mittendb{at}wustl.edu ) |
---|---|
AbstractList | 1 Washington University School of Medicine, St. Louis, Missouri; 2 Department of Nutrition and Dietetics, Harokopio University, Athens, Greece
Submitted 22 November 2006
; accepted in final form 29 January 2007
Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were 55% greater ( P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1 ) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB-100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2 ) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3 ) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver.
hepatic lipid metabolism; lipoprotein; stable isotope; tracer
Address for reprint requests and other correspondence: B. Mittendorfer, Washington Univ. School of Medicine, Division of Geriatrics and Nutritional Science, 660 South Euclid Ave.; Campus Box 8031, St. Louis, MO 63110 (e-mail: mittendb{at}wustl.edu ) Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355-E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were approximately 55% greater (P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB-100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver.Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355-E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were approximately 55% greater (P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB-100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver. Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355–E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were ∼55% greater ( P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB-100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver. Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355-E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were ...55% greater (P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB- 100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver. (ProQuest-CSA LLC: ... denotes formulae/symbols omitted.) Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355-E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were similar to 55% greater (P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB-100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver. Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355-E362, 2006) has recently demonstrated that a single, prolonged bout of moderate-intensity cycling (2 h at 60% of peak oxygen consumption) in the evening increases basal whole-body free fatty acid (FFA) flux and fat oxidation, decreases hepatic VLDL-apolipoprotein B-100 (apoB-100) secretion, and enhances removal efficiency of VLDL-triglyceride (TG) from the circulation the following day in untrained, healthy, lean men. In the present study, we investigated the effect of a single, shorter-duration bout of the same exercise (1 h cycling at 60% of peak oxygen consumption) on basal FFA, VLDL-TG, and VLDL-apoB-100 kinetics in seven untrained, healthy, lean men by using stable isotope-labeled tracer techniques. Basal FFA rate of appearance in plasma and plasma FFA concentration were approximately 55% greater (P < 0.05) the morning after exercise than rest, whereas resting metabolic rate and whole-body substrate oxidation rates were not different after rest and exercise. Exercise had no effect on plasma VLDL-TG and VLDL-apoB-100 concentrations, hepatic VLDL-TG and VLDL-apoB-100 secretion rates, and VLDL-TG and VLDL-apoB-100 plasma clearance rates (all P > 0.05). We conclude that in untrained, healthy, lean men 1) the exercise-induced changes in basal whole-body fat oxidation, VLDL-TG, and VLDL-apoB-100 metabolism during the late phase of recovery from exercise are related to the duration of the exercise bout; 2) single sessions of typical recreational activities appear to have little effect on basal, fasting plasma TG homeostasis; and 3) there is a dissociation between systemic FFA availability and VLDL-TG and VLDL-apoB-100 secretion by the liver. |
Author | Mohammed, B. Selma Magkos, Faidon Patterson, Bruce W Mittendorfer, Bettina |
Author_xml | – sequence: 1 fullname: Magkos, Faidon – sequence: 2 fullname: Patterson, Bruce W – sequence: 3 fullname: Mohammed, B. Selma – sequence: 4 fullname: Mittendorfer, Bettina |
BackLink | https://www.ncbi.nlm.nih.gov/pubmed/17264219$$D View this record in MEDLINE/PubMed |
BookMark | eNqFks9uEzEQxleoiKaFF-CALA7cNvjPrneXW1qaghSJS-Fqeb2ziYNjL7aXJk_Dq9ZpEoQqAaeRx99vZjz-LrIz6yxk2WuCp4SU9L1cD2A7N8WYMz6lKTzLJumC5qQsy7NsgknDclIXzXl2EcIaY1yVBX2RnZOK8oKSZpL9mqGg7dIAIvkKtW6MyPUIfoJNWQRb8EoHQNoqDzJAQK0M0qD5fIZ6M27RvY6rPST7HlTcM98WHxd59Hppdgq87gBJ2x2ycnBGD27wLoK26ConGKPv2kLUKqQeaLTRy3TukAFp0Qbsy-x5L02AV8d4mX2d39xdf8oXX24_X88WuSqKJuZQAZeyahkvcaMaUmCSVtSzuiVdjdsaCOOFLFuFawoFaYnilOMa94yRiveUXWbvDnXTcD9GCFFsdFBgjLTgxiAqXFYUU_ZfIWl4as-bJHz7RLh2o7fpEYIyynDRYJxEb46isd1AJwavN9LvxOmDkqA-CJR3IXjohdJRRu0eN2UEwWLvBXH0gnj0gth7IaH0Cfq7-r-gDwdopZere-1BDKtd0M645U7MR2PuYBtPIG2o4OKGlLwWQ9cnePp3-NTsD4g9AJTv3Y8 |
CODEN | AJPMD9 |
CitedBy_id | crossref_primary_10_1152_japplphysiol_00956_2013 crossref_primary_10_1017_S0007114508012233 crossref_primary_10_1042_CS20070134 crossref_primary_10_1007_s00421_010_1409_7 crossref_primary_10_1152_ajpendo_90545_2008 crossref_primary_10_1139_H08_126 crossref_primary_10_1042_CS20080078 crossref_primary_10_1530_EJE_09_0756 crossref_primary_10_1016_j_metabol_2007_03_012 crossref_primary_10_1016_j_metabol_2009_03_023 crossref_primary_10_1016_j_ogc_2009_03_001 crossref_primary_10_1139_apnm_2015_0314 crossref_primary_10_1186_1758_5996_2_7 crossref_primary_10_3389_fendo_2014_00162 crossref_primary_10_1002_cbf_1495 crossref_primary_10_1155_2012_809576 crossref_primary_10_3389_fphys_2017_00293 crossref_primary_10_1249_MSS_0b013e318278183e crossref_primary_10_1002_phy2_262 crossref_primary_10_1186_1758_5996_2_31 crossref_primary_10_1113_jphysiol_2007_137331 crossref_primary_10_1186_s12937_016_0134_4 crossref_primary_10_1371_journal_pone_0013318 crossref_primary_10_1042_CS20080220 crossref_primary_10_1007_s00421_009_1115_5 crossref_primary_10_1186_1476_511X_10_164 crossref_primary_10_2217_clp_09_9 crossref_primary_10_1194_jlr_M074062 crossref_primary_10_1111_j_1365_2362_2008_01999_x crossref_primary_10_1152_japplphysiol_90761_2008 crossref_primary_10_1152_japplphysiol_00623_2013 crossref_primary_10_1139_apnm_2012_0468 |
Cites_doi | 10.1016/0002-9149(64)90004-9 10.1152/jappl.2001.90.4.1239 10.1016/0009-8981(81)90320-X 10.1093/ajcn/30.5.716 10.1152/jappl.1997.83.6.2019 10.1152/ajpendo.2000.278.6.E1144 10.1016/S0022-2275(20)32435-4 10.1152/jappl.1992.72.3.914 10.1152/ajpendo.2000.279.5.E1020 10.1194/jlr.M600175-JLR200 10.2337/diabetes.52.7.1641 10.1016/0026-0495(87)90040-0 10.1093/ajcn/71.2.465 10.1152/jappl.1984.56.4.845 10.1007/BF00280883 10.1152/ajpendo.1998.275.2.E332 10.1097/00008483-200211000-00002 10.1007/s00421-004-1190-6 10.1007/BF00634303 10.1080/026404102321011715 10.1152/jappl.1998.85.4.1516 10.1172/JCI106264 10.1016/S0022-2275(20)30164-4 10.1079/PNS2004350 10.1152/jappl.1998.84.6.1895 10.1113/jphysiol.2002.022392 10.1152/ajpendo.00379.2002 10.1152/ajpendo.2001.280.3.E540 10.1016/0026-0495(82)90085-3 10.1016/0026-0495(86)90108-3 10.1007/BF00576264 10.1249/01.MSS.0000135793.43808.6C 10.1152/ajpendo.1999.276.3.E427 10.1097/00005768-199610000-00005 10.1111/j.1440-1681.1995.tb01982.x 10.1097/00043798-199604000-00014 10.2337/diab.38.12.1595 10.2337/diabetes.51.6.1867 10.1016/S1043-2760(03)00143-7 10.1210/jc.2003-030226 10.1016/0026-0495(80)90111-0 10.1152/jappl.1998.85.3.1169 10.1007/s005920070035 10.1152/ajpendo.00567.2001 10.1152/jappl.2000.89.2.472 10.1046/j.1365-201X.2003.01167.x 10.1152/ajpendo.00268.2002 10.1007/BF00334418 10.1152/jappl.1995.79.1.279 10.1152/ajpendo.2000.279.2.E348 10.1152/ajpendo.00259.2005 10.2337/diabetes.47.5.779 10.1016/S0025-7125(16)31054-9 10.1152/japplphysiol.00368.2004 10.1172/JCI117633 10.1152/jappl.2000.89.5.2049 10.1097/00041433-199706000-00004 10.1097/00005768-199806000-00032 10.2165/00007256-200131150-00002 10.1152/jappl.1993.75.1.108 10.1249/01.MSS.0000126498.04454.C0 10.1017/S0007114598001779 10.1152/jappl.1983.55.2.628 |
ContentType | Journal Article |
Copyright | Copyright American Physiological Society Jun 2007 |
Copyright_xml | – notice: Copyright American Physiological Society Jun 2007 |
DBID | AAYXX CITATION CGR CUY CVF ECM EIF NPM 7QP 7TS 7U7 C1K 7X8 |
DOI | 10.1152/ajpendo.00636.2006 |
DatabaseName | CrossRef Medline MEDLINE MEDLINE (Ovid) MEDLINE MEDLINE PubMed Calcium & Calcified Tissue Abstracts Physical Education Index Toxicology Abstracts Environmental Sciences and Pollution Management MEDLINE - Academic |
DatabaseTitle | CrossRef MEDLINE Medline Complete MEDLINE with Full Text PubMed MEDLINE (Ovid) Toxicology Abstracts Calcium & Calcified Tissue Abstracts Physical Education Index Environmental Sciences and Pollution Management MEDLINE - Academic |
DatabaseTitleList | MEDLINE - Academic CrossRef Toxicology Abstracts Physical Education Index MEDLINE |
Database_xml | – sequence: 1 dbid: NPM name: PubMed url: https://proxy.k.utb.cz/login?url=http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed sourceTypes: Index Database – sequence: 2 dbid: EIF name: MEDLINE url: https://proxy.k.utb.cz/login?url=https://www.webofscience.com/wos/medline/basic-search sourceTypes: Index Database |
DeliveryMethod | fulltext_linktorsrc |
Discipline | Medicine Anatomy & Physiology |
EISSN | 1522-1555 |
EndPage | E1574 |
ExternalDocumentID | 1284655731 17264219 10_1152_ajpendo_00636_2006 ajpendo_292_6_E1568 |
Genre | Research Support, Non-U.S. Gov't Journal Article Research Support, N.I.H., Extramural Feature |
GrantInformation_xml | – fundername: NCRR NIH HHS grantid: RR-00954 – fundername: NIDDK NIH HHS grantid: DK-56341 – fundername: NIAMS NIH HHS grantid: AR-49869 – fundername: NIDDK NIH HHS grantid: P30 DK056341 – fundername: NCRR NIH HHS grantid: RR-00036 |
GroupedDBID | - 23M 2WC 39C 4.4 53G 5GY 5VS 8M5 ABPTK ACPRK ADACO ADBBV AENEX AFFNX AFRAH ALMA_UNASSIGNED_HOLDINGS BAWUL BKOMP C1A DIK DL E3Z EBS EJD F5P GX1 H13 KQ8 O0- OK1 P2P PQEST PQQKQ RAP RHF RHI RPL WH7 WOQ --- 6J9 AAYXX ABJNI BKKCC BTFSW CITATION EMOBN ITBOX P6G RPRKH TR2 W8F XSW YSK AAFWJ CGR CUY CVF ECM EIF NPM 7QP 7TS 7U7 C1K 7X8 |
ID | FETCH-LOGICAL-c449t-e7e6aa7b36509c91401115f38b1d80b8e1364a5bc082e41b1c626080f33176f23 |
ISSN | 0193-1849 |
IngestDate | Thu Jul 10 19:34:04 EDT 2025 Thu Jul 10 18:09:20 EDT 2025 Mon Jun 30 10:34:34 EDT 2025 Fri May 30 10:49:33 EDT 2025 Tue Jul 01 03:18:08 EDT 2025 Thu Apr 24 22:55:10 EDT 2025 Tue Jan 05 17:54:18 EST 2021 Mon May 06 11:42:23 EDT 2019 |
IsPeerReviewed | true |
IsScholarly | true |
Issue | 6 |
Language | English |
LinkModel | OpenURL |
MergedId | FETCHMERGED-LOGICAL-c449t-e7e6aa7b36509c91401115f38b1d80b8e1364a5bc082e41b1c626080f33176f23 |
Notes | SourceType-Scholarly Journals-1 ObjectType-Feature-1 content type line 14 ObjectType-Article-1 ObjectType-Feature-2 content type line 23 |
PMID | 17264219 |
PQID | 232304900 |
PQPubID | 48583 |
ParticipantIDs | proquest_journals_232304900 pubmed_primary_17264219 proquest_miscellaneous_70572023 highwire_physiology_ajpendo_292_6_E1568 proquest_miscellaneous_19691469 crossref_citationtrail_10_1152_ajpendo_00636_2006 crossref_primary_10_1152_ajpendo_00636_2006 |
ProviderPackageCode | CITATION AAYXX |
PublicationCentury | 2000 |
PublicationDate | 20070601 2007-06-00 2007-Jun |
PublicationDateYYYYMMDD | 2007-06-01 |
PublicationDate_xml | – month: 06 year: 2007 text: 20070601 day: 01 |
PublicationDecade | 2000 |
PublicationPlace | United States |
PublicationPlace_xml | – name: United States – name: Bethesda |
PublicationTitle | American journal of physiology: endocrinology and metabolism |
PublicationTitleAlternate | Am J Physiol Endocrinol Metab |
PublicationYear | 2007 |
Publisher | American Physiological Society |
Publisher_xml | – name: American Physiological Society |
References | R61 R60 R63 R62 R21 R65 R20 R64 R23 R22 R25 R24 R27 R26 R29 R28 R1 R2 R3 R4 R5 R6 R7 R8 R9 R30 R32 R31 R34 R33 R36 R35 R38 R37 R39 R41 R40 R43 R42 R45 R44 R47 R46 R49 R48 R50 R52 R51 R10 R54 R53 R12 R56 R11 R55 R14 R58 R13 R57 R16 R15 R59 R18 R17 R19 |
References_xml | – ident: R32 doi: 10.1016/0002-9149(64)90004-9 – ident: R57 doi: 10.1152/jappl.2001.90.4.1239 – ident: R9 doi: 10.1016/0009-8981(81)90320-X – ident: R25 doi: 10.1093/ajcn/30.5.716 – ident: R7 doi: 10.1152/jappl.1997.83.6.2019 – ident: R34 doi: 10.1152/ajpendo.2000.278.6.E1144 – ident: R52 doi: 10.1016/S0022-2275(20)32435-4 – ident: R11 doi: 10.1152/jappl.1992.72.3.914 – ident: R44 doi: 10.1152/ajpendo.2000.279.5.E1020 – ident: R23 doi: 10.1194/jlr.M600175-JLR200 – ident: R48 doi: 10.2337/diabetes.52.7.1641 – ident: R3 doi: 10.1016/0026-0495(87)90040-0 – ident: R20 doi: 10.1093/ajcn/71.2.465 – ident: R46 doi: 10.1152/jappl.1984.56.4.845 – ident: R47 doi: 10.1007/BF00280883 – ident: R40 doi: 10.1152/ajpendo.1998.275.2.E332 – ident: R14 doi: 10.1097/00008483-200211000-00002 – ident: R12 doi: 10.1007/s00421-004-1190-6 – ident: R18 doi: 10.1007/BF00634303 – ident: R21 doi: 10.1080/026404102321011715 – ident: R65 doi: 10.1152/jappl.1998.85.4.1516 – ident: R4 doi: 10.1172/JCI106264 – ident: R51 doi: 10.1016/S0022-2275(20)30164-4 – ident: R55 doi: 10.1079/PNS2004350 – ident: R27 doi: 10.1152/jappl.1998.84.6.1895 – ident: R61 doi: 10.1113/jphysiol.2002.022392 – ident: R49 doi: 10.1152/ajpendo.00379.2002 – ident: R63 doi: 10.1152/ajpendo.2001.280.3.E540 – ident: R10 doi: 10.1016/0026-0495(82)90085-3 – ident: R13 doi: 10.1016/0026-0495(86)90108-3 – ident: R6 – ident: R26 doi: 10.1007/BF00576264 – ident: R1 doi: 10.1249/01.MSS.0000135793.43808.6C – ident: R54 doi: 10.1152/ajpendo.1999.276.3.E427 – ident: R60 doi: 10.1097/00005768-199610000-00005 – ident: R36 doi: 10.1111/j.1440-1681.1995.tb01982.x – ident: R31 doi: 10.1097/00043798-199604000-00014 – ident: R39 doi: 10.2337/diab.38.12.1595 – ident: R5 doi: 10.2337/diabetes.51.6.1867 – ident: R28 – ident: R33 doi: 10.1016/S1043-2760(03)00143-7 – ident: R19 doi: 10.1210/jc.2003-030226 – ident: R58 doi: 10.1016/0026-0495(80)90111-0 – ident: R16 doi: 10.1152/jappl.1998.85.3.1169 – ident: R56 doi: 10.1007/s005920070035 – ident: R64 doi: 10.1152/ajpendo.00567.2001 – ident: R24 doi: 10.1152/jappl.2000.89.2.472 – ident: R37 doi: 10.1046/j.1365-201X.2003.01167.x – ident: R38 doi: 10.1152/ajpendo.00268.2002 – ident: R59 doi: 10.1007/BF00334418 – ident: R8 doi: 10.1152/jappl.1995.79.1.279 – ident: R35 doi: 10.1152/ajpendo.2000.279.2.E348 – ident: R43 doi: 10.1152/ajpendo.00259.2005 – ident: R45 doi: 10.2337/diabetes.47.5.779 – ident: R22 doi: 10.1016/S0025-7125(16)31054-9 – ident: R62 doi: 10.1152/japplphysiol.00368.2004 – ident: R42 doi: 10.1172/JCI117633 – ident: R30 doi: 10.1152/jappl.2000.89.5.2049 – ident: R41 doi: 10.1097/00041433-199706000-00004 – ident: R2 doi: 10.1097/00005768-199806000-00032 – ident: R15 doi: 10.2165/00007256-200131150-00002 – ident: R53 doi: 10.1152/jappl.1993.75.1.108 – ident: R50 doi: 10.1249/01.MSS.0000126498.04454.C0 – ident: R29 doi: 10.1017/S0007114598001779 – ident: R17 doi: 10.1152/jappl.1983.55.2.628 |
SSID | ssj0007542 |
Score | 2.073228 |
Snippet | 1 Washington University School of Medicine, St. Louis, Missouri; 2 Department of Nutrition and Dietetics, Harokopio University, Athens, Greece
Submitted 22... Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355–E362, 2006) has recently demonstrated that a... Our group (Magkos F, Wright DC, Patterson BW, Mohammed BS, Mittendorfer B, Am J Physiol Endocrinol Metab 290: E355-E362, 2006) has recently demonstrated that a... |
SourceID | proquest pubmed crossref highwire |
SourceType | Aggregation Database Index Database Enrichment Source Publisher |
StartPage | E1568 |
SubjectTerms | Adult Apolipoprotein B-100 - blood Chemical compounds Circadian Rhythm Exercise Exercise - physiology Fatty acids Fatty Acids, Nonesterified - blood Humans Insulin - blood Isotopes Kinetics Lipoproteins, VLDL - blood Liver Male Men Metabolism Oils & fats Osmolar Concentration Oxidation Oxygen Consumption Proteins Stable isotopes Thinness - blood Time Factors Tracer techniques Triglycerides - blood |
Title | A single 1-h bout of evening exercise increases basal FFA flux without affecting VLDL-triglyceride and VLDL-apolipoprotein B-100 kinetics in untrained lean men |
URI | http://ajpendo.physiology.org/cgi/content/abstract/292/6/E1568 https://www.ncbi.nlm.nih.gov/pubmed/17264219 https://www.proquest.com/docview/232304900 https://www.proquest.com/docview/19691469 https://www.proquest.com/docview/70572023 |
Volume | 292 |
hasFullText | 1 |
inHoldings | 1 |
isFullTextHit | |
isPrint | |
journalDatabaseRights | – providerCode: PRVAFT databaseName: Colorado Digital library customDbUrl: eissn: 1522-1555 dateEnd: 99991231 omitProxy: true ssIdentifier: ssj0007542 issn: 0193-1849 databaseCode: KQ8 dateStart: 19971001 isFulltext: true titleUrlDefault: http://grweb.coalliance.org/oadl/oadl.html providerName: Colorado Alliance of Research Libraries – providerCode: PRVFQY databaseName: GFMER Free Medical Journals customDbUrl: eissn: 1522-1555 dateEnd: 20241005 omitProxy: true ssIdentifier: ssj0007542 issn: 0193-1849 databaseCode: GX1 dateStart: 19971001 isFulltext: true titleUrlDefault: http://www.gfmer.ch/Medical_journals/Free_medical.php providerName: Geneva Foundation for Medical Education and Research |
link | http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwnV1bb9MwFLbGkBAvE2zAyrj4AcFDlJJ7k8cOVk2woiF1sDfLSRxWlibVmkqMP8Nf5Rw7TjLaIsZL1Lqxm-R8OTcffybkVQomwIqDxLQj4Zqel7lmBFbYBFsZe8guk4W4dnj8KTg-8z6c--dbd3Y6VUvLKu4nP9euK_kfqUIbyBVXyd5Css2g0ACfQb5wBAnD8Z9kPDQw0s-FYZsXhqwwBtcPKZkw_tebKRnTAj3DhVgYYLJAIqPR0Mjy5Q-Zg8VOXNZ0YJ8vJ-9PzAoC9vw6gRtI1dyCbOW4ncO8lLwO08I4BM1qGZfgo0qeZ2hZFnK7CXBgc8zuz-o1ZprhVs8MdagqZFZFrZbB_YGKtAQVVrSkUDNRAURzTXIo8-bfLlVd4IhP07aC4FSxhKoKAglX42u_6VReYHpeQbkP2jFvbdF4WuEkQHmVKegeCqwD5zdyIYO2ZkunRyPXhJhVKWFRq3QIt8Fr8rs634mcDri7GvwIAtqw4w7Ad7WN0Kqt8ZG7ln_HvYpLrA90ZcnLGmLvPwxuUwYpAzDfYfUYTI7BFIX8XWcQeJhN-Pi5pb_H7YrV-n91m3oVmO-8Xb2Om56WZr_eHElJj2rygOzUoRAdKlw_JFui2CV7w4JX5eyavqanDTx2yb1xXQOyR34NqUI9BdRTRD0tM1qjnmrU0wb1VKKeAuopop7WqKcN6ukK6imgj65BPZWopxr18B-0QT1F1FNA_SNyNjqavDs2621GzMTzosoUAxFwPohdJJNMIsw4wBPN3DC209CKQ2G7gcf9OAFvWXh2bCeYBAitzAXfO8gc9zHZLspC7BMKwZKd2UGQZLHlJUHARRJDj8izeJSkKe8RW4uEJTUHP15lzjZDoUeMps9cMdD89WxXS5q1bzHDfNYEwKd7AP5ZwCTW2TzNeuTNul56-M7ZPXKg4cNqhbFgEJXhbL1l9cjL5lcwUzj3yAtRLhcMWbjAKYs2nzGAyNGBCKJHnihUtnc7cHA5fvT0Vk_igNxvVcQzsl1dLcVzCCCq-IV8qX4D0IsfgA |
linkProvider | Colorado Alliance of Research Libraries |
openUrl | ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=A+single+1-h+bout+of+evening+exercise+increases+basal+FFA+flux+without+affecting+VLDL-triglyceride+and+VLDL-apolipoprotein+B-100+kinetics+in+untrained+lean+men&rft.jtitle=American+journal+of+physiology%3A+endocrinology+and+metabolism&rft.au=Magkos%2C+Faidon&rft.au=Patterson%2C+Bruce+W.&rft.au=Mohammed%2C+B.+Selma&rft.au=Mittendorfer%2C+Bettina&rft.date=2007-06-01&rft.issn=0193-1849&rft.eissn=1522-1555&rft.volume=292&rft.issue=6&rft.spage=E1568&rft.epage=E1574&rft_id=info:doi/10.1152%2Fajpendo.00636.2006&rft.externalDBID=n%2Fa&rft.externalDocID=10_1152_ajpendo_00636_2006 |
thumbnail_l | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=0193-1849&client=summon |
thumbnail_m | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=0193-1849&client=summon |
thumbnail_s | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=0193-1849&client=summon |