Relationship among LRP1 expression, Pyk2 phosphorylation and MMP‐9 activation in left ventricular remodelling after myocardial infarction - Revuelta‐López, Elena; Soler‐Botija, Carol; Nasarre, Laura; Benitez‐Amaro, Aleyda; Gonzalo‐Calvo, David; Bayes‐Genis, Antoni; Llorente‐Cortés, Vicenta (Journal of cellular and molecular medicine, 2017)
Keywords: Animals; cardiac remodelling; Cell culture; Cell Hypoxia; Confocal microscopy; Enzyme Activation; Fibroblasts; Fibroblasts - metabolism; Fibroblasts - pathology; Focal Adhesion Kinase 2 - metabolism; Gelatinase A; Gelatinase B; Gene expression; Heart attacks; Heart diseases; Heart Ventricles - metabolism; Heart Ventricles - pathology; Hypoxia; Kinases; LRP1; Male; Matrix metalloproteinase; Matrix Metalloproteinase 2 - metabolism; Matrix Metalloproteinase 9 - metabolism; Metalloproteinase; Mice, Inbred C57BL; MMP‐9; mRNA; Myocardial infarction; Myocardial Infarction - metabolism; Myocardial Infarction - pathology; Original; Phosphorylation; Polymerase chain reaction; pPyk2; Proline; Proline-rich tyrosine kinase 2; Protein-tyrosine kinase; Proteins; Receptor density; Receptors, LDL - genetics; Receptors, LDL - metabolism; RNA, Messenger - genetics; RNA, Messenger - metabolism; Time Factors; Tumor Suppressor Proteins - genetics; Tumor Suppressor Proteins - metabolism; Up-Regulation; Ventricle; Ventricular Remodeling; Vimentin; United States--US; Indianapolis Indiana; pPyk2; cardiac remodelling; LRP1; MMP-9; myocardial infarction