PAI‐1 gain‐of‐function genotype, factors increasing PAI‐1 levels, and airway obstruction: The GALA II Cohort - Sherenian, M. G.; Cho, S. H.; Levin, A.; Min, J‐Y.; Oh, S. S.; Hu, D.; Galanter, J.; Sen, S.; Huntsman, S.; Eng, C.; Rodriguez‐Santana, J. R.; Serebrisky, D.; et al. (Clinical and experimental allergy, 2017)
Keywords: Adolescent; Adult; Airway management; Airway Obstruction - epidemiology; Airway Obstruction - genetics; Airway Obstruction - metabolism; Airway Obstruction - physiopathology; Alleles; Asthma; Asthma, Occupational - epidemiology; Asthma, Occupational - genetics; Asthma, Occupational - metabolism; Asthma, Occupational - physiopathology; Child; Children; Cohort Studies; epidemiology; Ethnicity; Female; Fibrosis; Gain of Function Mutation; Gene polymorphism; Genetic Association Studies; Genetic Predisposition to Disease; Genotype; Genotype & phenotype; Humans; Male; Odds Ratio; PAI‐1; pediatrics; Plasminogen Activator Inhibitor 1 - genetics; Plasminogen Activator Inhibitor 1 - metabolism; Plasminogen activator inhibitors; Polymorphism, Single Nucleotide; Respiratory function; Respiratory Function Tests; Respiratory tract; Respiratory tract diseases; Tobacco; Young Adult; epidemiology; PAI-1; asthma; pediatrics