Hyper-N-glycosylated SEL1L3 as auto-antigenic B-cell receptor target of primary vitreoretinal lymphomas - Elbert, Michelle; Neumann, Frank; Kiefer, Maximilian; Christofyllakis, Konstantinos; Balensiefer, Benedikt; Kos, Igor; Carbon, Gabi; Kaddu-Mulindwa, Dominic; Bittenbring, Joerg Thomas; Fadle, Natalie; Regitz, Evi; Fend, Falko; et al. (Scientific reports, 2024)
Keywords: 631/67/1484; 631/67/1922; 631/67/1990; 631/67/1990/291/1621; 692/308/575; Aged; Antigens; Auto-antigens; Autoantigens; Autoantigens - immunology; Autoantigens - metabolism; B-cell receptor; B-cell receptor antigens; Biopsy; Cell activation; Cell Line, Tumor; Central nervous system; Complementarity-determining region 3; Female; Glycosylation; Humanities and Social Sciences; Humans; Lymphocytes B; Lymphoma; Lymphoma, Large B-Cell, Diffuse - genetics; Lymphoma, Large B-Cell, Diffuse - immunology; Lymphoma, Large B-Cell, Diffuse - metabolism; Lymphoma, Large B-Cell, Diffuse - pathology; Male; Middle Aged; multidisciplinary; Post-translation; Primary CNS lymphoma; Primary vitreoretinal lymphoma; Receptors, Antigen, B-Cell - metabolism; Retinal Neoplasms - genetics; Retinal Neoplasms - immunology; Retinal Neoplasms - metabolism; Retinal Neoplasms - pathology; SAMD14/neurabin-I; Science; Science (multidisciplinary); SEL1L3; Toxins; Tumor cell lines; Vitreous Body - metabolism; Vitreous Body - pathology; Primary CNS lymphoma; B-cell receptor antigens; SAMD14/neurabin-I; Auto-antigens; Primary vitreoretinal lymphoma; SEL1L3