ZNF768 loss amplifies p53 action and reduces lung tumorigenesis in mice - Poirier, Audrey; Utecht, Timon; Villot, Romain; Gélinas, Yves; Mouchiroud, Mathilde; Kordahi, Manal; Kolnohuz, Alona; Pasteur, Coline; Roy, Joanny; Beaulieu, Marie-Josée; Orain, Michèle; Samson, Nolwenn; et al. (Oncogene, 2025)
Keywords: 13/1; 13/51; 38/77; 631/67; 631/80/304; 631/80/509; 64/60; 82/80; 96/106; Animals; Apoptosis; Cancer; Carcinogenesis - genetics; Cell adhesion; Cell Biology; Cell division; Cell growth; Cell proliferation; Cell Proliferation - genetics; Cell surface receptors; Embryo fibroblasts; Embryos; Fibroblasts - metabolism; Genotoxicity; Human Genetics; Internal Medicine; Lung Neoplasms - genetics; Lung Neoplasms - metabolism; Lung Neoplasms - pathology; Medicine; Medicine & Public Health; Mice; Mice, Knockout; Oncology; p53 Protein; Radiosensitivity; Senescence; Tumor suppressor genes; Tumor Suppressor Protein p53 - genetics; Tumor Suppressor Protein p53 - metabolism; Tumorigenesis; Zinc finger proteins