Celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathy
Celiac disease is usually associated with autoimmune disorders and has occasionally been reported in patients with inflammatory myopathies. Our aim was to determine the presence of celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathies and to investigat...
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| Published in | Muscle & nerve Vol. 35; no. 1; pp. 49 - 54 |
|---|---|
| Main Authors | , , , , , |
| Format | Journal Article |
| Language | English |
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Hoboken
Wiley Subscription Services, Inc., A Wiley Company
01.01.2007
Wiley |
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| Online Access | Get full text |
| ISSN | 0148-639X 1097-4598 1097-4598 |
| DOI | 10.1002/mus.20652 |
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| Abstract | Celiac disease is usually associated with autoimmune disorders and has occasionally been reported in patients with inflammatory myopathies. Our aim was to determine the presence of celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathies and to investigate their relationship. Serum antigliadin, anti–tissue transglutaminase, and antiendomysial antibodies were determined in 51 patients with inflammatory myopathies. HLA‐DQ2 and ‐DQ8 alleles were studied to assess their complementary diagnostic value. Jejunal biopsy was performed in patients with moderate to high levels of antigliadin antibodies. Patients with jejunal histology consistent with celiac disease initiated a gluten‐free diet. Seventeen patients (31%) were positive for antigliadin antibodies, which were significantly more frequent in patients with inclusion‐body myositis than dermatomyositis (P < 0.001). Positive status to HLA‐DQ2 and/or ‐DQ8 did not differ between antigliadin‐positive (75% and 12.5%) or ‐negative (60% and 15%) patients. Three of five jejunal biopsies were diagnostic for celiac disease with histological normalization after a gluten‐free diet. Thus, celiac disease is more prevalent in patients with inflammatory myopathies than in the general population. Positive status to HLA‐DQ2 allele, which is known to be more frequent in patients with inflammatory myopathies, could explain the high prevalence of antigliadin antibodies in this population. The diagnostic value of HLA‐DQ2 or ‐DQ8 haplotypes to detect celiac disease in patients with inflammatory myopathy is limited. Muscle Nerve, 2006 |
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| AbstractList | Celiac disease is usually associated with autoimmune disorders and has occasionally been reported in patients with inflammatory myopathies. Our aim was to determine the presence of celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathies and to investigate their relationship. Serum antigliadin, anti-tissue transglutaminase, and antiendomysial antibodies were determined in 51 patients with inflammatory myopathies. HLA-DQ2 and -DQ8 alleles were studied to assess their complementary diagnostic value. Jejunal biopsy was performed in patients with moderate to high levels of antigliadin antibodies. Patients with jejunal histology consistent with celiac disease initiated a gluten-free diet. Seventeen patients (31%) were positive for antigliadin antibodies, which were significantly more frequent in patients with inclusion-body myositis than dermatomyositis (P < 0.001). Positive status to HLA-DQ2 and/or -DQ8 did not differ between antigliadin-positive (75% and 12.5%) or -negative (60% and 15%) patients. Three of five jejunal biopsies were diagnostic for celiac disease with histological normalization after a gluten-free diet. Thus, celiac disease is more prevalent in patients with inflammatory myopathies than in the general population. Positive status to HLA-DQ2 allele, which is known to be more frequent in patients with inflammatory myopathies, could explain the high prevalence of antigliadin antibodies in this population. The diagnostic value of HLA-DQ2 or -DQ8 haplotypes to detect celiac disease in patients with inflammatory myopathy is limited. Celiac disease is usually associated with autoimmune disorders and has occasionally been reported in patients with inflammatory myopathies. Our aim was to determine the presence of celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathies and to investigate their relationship. Serum antigliadin, anti–tissue transglutaminase, and antiendomysial antibodies were determined in 51 patients with inflammatory myopathies. HLA‐DQ2 and ‐DQ8 alleles were studied to assess their complementary diagnostic value. Jejunal biopsy was performed in patients with moderate to high levels of antigliadin antibodies. Patients with jejunal histology consistent with celiac disease initiated a gluten‐free diet. Seventeen patients (31%) were positive for antigliadin antibodies, which were significantly more frequent in patients with inclusion‐body myositis than dermatomyositis ( P < 0.001). Positive status to HLA‐DQ2 and/or ‐DQ8 did not differ between antigliadin‐positive (75% and 12.5%) or ‐negative (60% and 15%) patients. Three of five jejunal biopsies were diagnostic for celiac disease with histological normalization after a gluten‐free diet. Thus, celiac disease is more prevalent in patients with inflammatory myopathies than in the general population. Positive status to HLA‐DQ2 allele, which is known to be more frequent in patients with inflammatory myopathies, could explain the high prevalence of antigliadin antibodies in this population. The diagnostic value of HLA‐DQ2 or ‐DQ8 haplotypes to detect celiac disease in patients with inflammatory myopathy is limited. Muscle Nerve, 2006 Celiac disease is usually associated with autoimmune disorders and has occasionally been reported in patients with inflammatory myopathies. Our aim was to determine the presence of celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathies and to investigate their relationship. Serum antigliadin, anti–tissue transglutaminase, and antiendomysial antibodies were determined in 51 patients with inflammatory myopathies. HLA‐DQ2 and ‐DQ8 alleles were studied to assess their complementary diagnostic value. Jejunal biopsy was performed in patients with moderate to high levels of antigliadin antibodies. Patients with jejunal histology consistent with celiac disease initiated a gluten‐free diet. Seventeen patients (31%) were positive for antigliadin antibodies, which were significantly more frequent in patients with inclusion‐body myositis than dermatomyositis (P < 0.001). Positive status to HLA‐DQ2 and/or ‐DQ8 did not differ between antigliadin‐positive (75% and 12.5%) or ‐negative (60% and 15%) patients. Three of five jejunal biopsies were diagnostic for celiac disease with histological normalization after a gluten‐free diet. Thus, celiac disease is more prevalent in patients with inflammatory myopathies than in the general population. Positive status to HLA‐DQ2 allele, which is known to be more frequent in patients with inflammatory myopathies, could explain the high prevalence of antigliadin antibodies in this population. The diagnostic value of HLA‐DQ2 or ‐DQ8 haplotypes to detect celiac disease in patients with inflammatory myopathy is limited. Muscle Nerve, 2006 Celiac disease is usually associated with autoimmune disorders and has occasionally been reported in patients with inflammatory myopathies. Our aim was to determine the presence of celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathies and to investigate their relationship. Serum antigliadin, anti-tissue transglutaminase, and antiendomysial antibodies were determined in 51 patients with inflammatory myopathies. HLA-DQ2 and -DQ8 alleles were studied to assess their complementary diagnostic value. Jejunal biopsy was performed in patients with moderate to high levels of antigliadin antibodies. Patients with jejunal histology consistent with celiac disease initiated a gluten-free diet. Seventeen patients (31%) were positive for antigliadin antibodies, which were significantly more frequent in patients with inclusion-body myositis than dermatomyositis (P < 0.001). Positive status to HLA-DQ2 and/or -DQ8 did not differ between antigliadin-positive (75% and 12.5%) or -negative (60% and 15%) patients. Three of five jejunal biopsies were diagnostic for celiac disease with histological normalization after a gluten-free diet. Thus, celiac disease is more prevalent in patients with inflammatory myopathies than in the general population. Positive status to HLA-DQ2 allele, which is known to be more frequent in patients with inflammatory myopathies, could explain the high prevalence of antigliadin antibodies in this population. The diagnostic value of HLA-DQ2 or -DQ8 haplotypes to detect celiac disease in patients with inflammatory myopathy is limited.Celiac disease is usually associated with autoimmune disorders and has occasionally been reported in patients with inflammatory myopathies. Our aim was to determine the presence of celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathies and to investigate their relationship. Serum antigliadin, anti-tissue transglutaminase, and antiendomysial antibodies were determined in 51 patients with inflammatory myopathies. HLA-DQ2 and -DQ8 alleles were studied to assess their complementary diagnostic value. Jejunal biopsy was performed in patients with moderate to high levels of antigliadin antibodies. Patients with jejunal histology consistent with celiac disease initiated a gluten-free diet. Seventeen patients (31%) were positive for antigliadin antibodies, which were significantly more frequent in patients with inclusion-body myositis than dermatomyositis (P < 0.001). Positive status to HLA-DQ2 and/or -DQ8 did not differ between antigliadin-positive (75% and 12.5%) or -negative (60% and 15%) patients. Three of five jejunal biopsies were diagnostic for celiac disease with histological normalization after a gluten-free diet. Thus, celiac disease is more prevalent in patients with inflammatory myopathies than in the general population. Positive status to HLA-DQ2 allele, which is known to be more frequent in patients with inflammatory myopathies, could explain the high prevalence of antigliadin antibodies in this population. The diagnostic value of HLA-DQ2 or -DQ8 haplotypes to detect celiac disease in patients with inflammatory myopathy is limited. |
| Author | de Torres, Ines Grau-Junyent, Josep M. Selva-O'Callaghan, Albert Vilardell-Tarrés, Miquel Casellas, Francesc Palou, Eduard |
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| Keywords | Human Immunopathology Connective tissue disease Skin disease Prevalence Dermatomyositis Autoimmune disease antigliadin antibodies Coeliac disease Inflammatory disease Striated muscle disease Polymyositis anti-transglutaminase antibodies Intestinal malabsorption Biopsy Systemic disease celiac disease Digestive diseases Intestinal disease Myositis Serum Inclusion body inflammatory myopathy Myopathy |
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enteropathy publication-title: Lancet – volume: 19 start-page: 757 year: 2001 end-page: 758 article-title: Dermatomyositis and celiac disease association: a further case publication-title: Clin Exp Rheumatol – volume: 19 start-page: 201 year: 2001 end-page: 203 article-title: An uncommon association: celiac disease and dermatomyositis in adults publication-title: Clin Exp Rheumatol – volume: 126 start-page: 137 year: 2006 end-page: 142 article-title: Celiac disease publication-title: Med Clin (Barc) – volume: 65 start-page: 301 year: 1982 end-page: 319 article-title: Polymyositis and adult coeliac disease publication-title: Acta Neurol Scand – volume: 325 start-page: 1487 year: 1991 end-page: 1498 article-title: Polymyositis, dermatomyositis, and inclusion body myositis publication-title: N Engl J Med – volume: 126 start-page: 685 year: 2003 end-page: 691 article-title: Gluten ataxia in perspective: epidemiology, genetic susceptibility and clinical characteristics publication-title: Brain – volume: 96 start-page: 1113 year: 2001 end-page: 1115 article-title: The prevalence of celiac disease autoantibodies in patients with systemic lupus erythematosus publication-title: Am J Gastroenterol – ident: e_1_2_7_17_2 doi: 10.1093/brain/awg050 – ident: e_1_2_7_20_2 doi: 10.1016/S0140-6736(76)90772-8 – ident: e_1_2_7_13_2 doi: 10.1016/j.imlet.2004.11.017 – ident: e_1_2_7_6_2 doi: 10.1074/jbc.275.12.8703 – ident: e_1_2_7_4_2 doi: 10.1097/00005176-199707000-00018 – ident: e_1_2_7_30_2 doi: 10.1111/j.1572-0241.2001.03753.x – ident: e_1_2_7_25_2 doi: 10.1097/01.bor.0000198008.11439.c9 – volume: 19 start-page: 201 year: 2001 ident: e_1_2_7_26_2 article-title: An uncommon association: celiac disease and dermatomyositis in adults publication-title: Clin Exp Rheumatol – ident: e_1_2_7_14_2 doi: 10.1002/ana.410380504 – volume: 126 start-page: 137 year: 2006 ident: e_1_2_7_5_2 article-title: Celiac disease publication-title: Med Clin (Barc) – ident: e_1_2_7_28_2 doi: 10.1016/0950-3528(95)90032-2 – ident: e_1_2_7_18_2 doi: 10.1212/01.wnl.0000196480.55601.3a – ident: e_1_2_7_33_2 doi: 10.1097/01.SMJ.0000051148.97720.69 – volume: 26 start-page: 1419 year: 1999 ident: e_1_2_7_10_2 article-title: Juvenile dermatomyositis and celiac disease publication-title: J Rheumatol – ident: e_1_2_7_22_2 doi: 10.1111/j.1572-0241.1999.01011.x – ident: e_1_2_7_34_2 – ident: e_1_2_7_3_2 doi: 10.1056/NEJM197502132920706 – ident: e_1_2_7_16_2 doi: 10.1016/S0140-6736(98)05342-2 – ident: e_1_2_7_15_2 doi: 10.1136/jnnp.63.6.770 – volume: 85 start-page: 9 year: 1995 ident: e_1_2_7_27_2 article-title: The natural history of gluten sensitivity: defining, refining and re‐defining publication-title: Q J Med – volume: 17 start-page: 1333 ident: e_1_2_7_12_2 article-title: Predisposing HLA‐DQ2 and HLA‐DQ8 haplotypes of coeliac disease and associated enteropathy in microscopic colitis publication-title: Eur J Gastroenterol Hepatol doi: 10.1097/00042737-200512000-00011 – ident: 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| Title | Celiac disease and antibodies associated with celiac disease in patients with inflammatory myopathy |
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