Invariant natural killer T cell deficiency leads to the development of spontaneous liver inflammation dependent on γδT cells in mice - Nishio, Kumiko; Miyagi, Takuya; Tatsumi, Tomohide; Mukai, Kaori; Yokoyama, Yoshinobu; Yoshioka, Teppei; Sakamori, Ryotaro; Hikita, Hayato; Kodama, Takahiro; Shimizu, Satoshi; Shigekawa, Minoru; Nawa, Takatoshi; et al. (Journal of gastroenterology, 2015)
Keywords: Abdominal Surgery; Adult; Aged; Aged, 80 and over; Animals; Autoimmune Diseases - immunology; Biliary Tract; Colorectal Surgery; Cytokines - biosynthesis; Female; Gastroenterology; Hepatitis - immunology; Hepatitis, Animal - immunology; Hepatology; Humans; Immune Tolerance - immunology; Interleukin-17 - biosynthesis; Liver - immunology; Male; Medicine; Medicine & Public Health; Mice, Inbred BALB C; Mice, Knockout; Middle Aged; Natural Killer T-Cells - immunology; Original Article—Liver; Pancreas; Receptors, Antigen, T-Cell, gamma-delta - analysis; Surgical Oncology; T-Lymphocyte Subsets - immunology; Invariant natural killer T cells; Autoimmune hepatitis; Interleukin-17; γδT cells