Massive accumulation of N‐acylethanolamines after stroke. Cell signalling in acute cerebral ischemia? - Berger, Christian; Schmid, Patricia C.; Schabitz, Wolf‐Ruediger; Wolf, Margit; Schwab, Stefan; Schmid, Harald H. O. (Journal of neurochemistry, 2004)
Keywords: Acute Disease; Animals; Arachidonic Acids - metabolism; Biological and medical sciences; Brain Ischemia - metabolism; Cerebral Cortex - drug effects; Cerebral Cortex - metabolism; Corpus Striatum - drug effects; Corpus Striatum - metabolism; Disease Models, Animal; Dizocilpine Maleate - pharmacology; Endocannabinoids; Ethanolamines - metabolism; Excitatory Amino Acid Antagonists - pharmacology; Extracellular Fluid - metabolism; glutamate; ischemic stroke; Male; Medical sciences; Microdialysis; Neurology; N‐acylethanolamines; Phospholipids - metabolism; Piperidines - pharmacology; Polyunsaturated Alkamides; Pyrazoles - pharmacology; Rats; Rats, Wistar; Receptor, Cannabinoid, CB1 - antagonists & inhibitors; Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors; Rimonabant; Signal Transduction - drug effects; Signal Transduction - physiology; Stroke - metabolism; Vascular diseases and vascular malformations of the nervous system; Rat; Cardiovascular disease; Glutamate receptor; Encephalon; Vascular disease; Ischemia; Cerebrovascular disease; ischemic stroke; Nervous system diseases; Stroke; Pathophysiology; N-acylethanolamines; Rodentia; Basal ganglion; Corpus striatum; N-acylethanolamine; Glutamate; Cerebral disorder; N-acyl phosphatidylethanolamine; Vertebrata; Mammalia; Microdialysis; Animal; Central nervous system disease; Excitatory aminoacid; Neurotransmitter; endocannabinoids; NMDA receptor