Mitochondrial dysfunction enhances the migration of vascular smooth muscles cells via suppression of Akt phosphorylation - Ahn, Sun Young; Choi, Yon-Sik; Koo, Hyun-Jung; Jeong, Jae Hoon; Park, Wook Ha; Kim, Minseok; Piao, Ying; Pak, Youngmi Kim (Biochimica et biophysica acta, 2010)
Keywords: Adenosine Triphosphate - metabolism; Akt/PKB; Animals; Atherosclerosis; Atherosclerosis - pathology; Atherosclerosis - physiopathology; Cardiovascular Diseases - physiopathology; Cell Movement; DNA Primers; Electron Transport Complex IV - genetics; Humans; Insulin - pharmacology; Insulin - physiology; Insulin signaling; Lipoproteins, LDL - pharmacology; Liver - physiology; Mitochondria, Muscle - drug effects; Mitochondria, Muscle - metabolism; Mitochondria, Muscle - physiology; Mitochondrial dysfunction; Muscle, Smooth, Vascular - cytology; Muscle, Smooth, Vascular - drug effects; Muscle, Smooth, Vascular - physiology; Oxidative Phosphorylation - drug effects; oxLDL; Oxygen Consumption; Proto-Oncogene Proteins c-akt - genetics; Proto-Oncogene Proteins c-akt - metabolism; Rats; Reverse Transcriptase Polymerase Chain Reaction; Signal Transduction - physiology; Transfection; VSMC; ddC; Mitochondrial dysfunction; Akt/PKB; OXPHOS; Atherosclerosis; oxLDL; AMPK; AICAR; VSMC; SFM; TCN; OCR; Insulin signaling