A Case Control Study of Risk Factors for Japanese Cedar Pollinosis
Risk factors for Japanese cedar pollinosis including past or family history of allergic diseases, smoking and passive smoking, dwelling conditions, and life events were analyzed by a case control method. Patients with Japanese cedar pollinosis (22 males and 67 females) were matched with a correspond...
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| Published in | Nippon Eiseigaku Zasshi (Japanese Journal of Hygiene) Vol. 50; no. 2; pp. 622 - 630 |
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| Main Authors | , , , , |
| Format | Journal Article |
| Language | English Japanese |
| Published |
Japan
The Japanese Society for Hygiene
1995
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| Subjects | |
| Online Access | Get full text |
| ISSN | 0021-5082 1882-6482 1882-6482 |
| DOI | 10.1265/jjh.50.622 |
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| Abstract | Risk factors for Japanese cedar pollinosis including past or family history of allergic diseases, smoking and passive smoking, dwelling conditions, and life events were analyzed by a case control method. Patients with Japanese cedar pollinosis (22 males and 67 females) were matched with a corresponding number of patients without potential symptoms of pollinosis according to sex and age (±5 years). The mean age was 39 years in both groups. The odds ratio (OR) was calculated by McNemar's method and the conditional logistic regression model. The design and methodology in this study were somewhat inadequate so that the validity of the results is limited. The most important problem was no-matching according to exposure to pollen. Significantly high OR for past history of allergic disease (8.80, 95% confidence interval (CI); 3.49-22.2), atopic sermatitis (9.00, 95% CI; 1.14-71.0), and a sibling history of allergic disease (3.25, 95% CI; 1.06-9.97) were consistent with former genetical studies. ORs were lower than unity for current smokers (0.36, 95% CI; 0.11-1.13) and those smoking 10 cigarettes/day or more (0.20, 95% CI; 0.04-0.91) relative to nonsmokers. The OR for passive smoking from 7-15 years of age as a result of the father's smoking habit (0.38, 95% CI; 0.17-0.86) was also significantly low. Smoking was suggested to increase the level of total and antigen-specific IgE in serum by former studies, so that sensitization and symptoms should be studied separately. The high OR of residents in a business or light industrial area (5.00, 95% CI; 1.45-17.3) suggested an association with air pollution. The findings such as a high OR for using a gas stove (3.52, 95% CI; 1.34-9.24), low OR for using an airconditioner (0.38, 95% CI; 0.16-0.87), and a not significant OR for other housing environments, were controversial by comparison with former studies of allergic responses to indoor antigens. “Moving” was the only item with a significantly high OR among the 37 life events. Therefore, it may represent an environmental change rather than mental stress. |
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| AbstractList | Risk factors for Japanese cedar pollinosis including past or family history of allergic diseases, smoking and passive smoking, dwelling conditions, and life events were analyzed by a case control method. Patients with Japanese cedar pollinosis (22 males and 67 females) were matched with a corresponding number of patients without potential symptoms of pollinosis according to sex and age (+/- 5 years). The mean age was 39 years in both groups. The odds ratio (OR) was calculated by McNemar's method and the conditional logistic regression model. The design and methodology in this study were somewhat inadequate so that the validity of the results is limited. The most important problem was no-matching according to exposure to pollen. Significantly high OR for past history of allergic disease (8.80, 95% confidence interval (CI); 3.49-22.2), atopic sermatitis (9.00, 95% CI; 1.14-71.0), and a sibling history of allergic disease (3.25, 95% CI; 1.06-9.97) were consistent with former genetical studies. ORs were lower than unity for current smokers (0.36, 95% CI; 0.11-1.13) and those smoking 10 cigarettes/day or more (0.20, 95% CI; 0.04-0.91) relative to nonsmokers. The OR for passive smoking from 7-15 years of age as a result of the father's smoking habit (0.38, 95% CI; 0.17-0.86) was also significantly low. Smoking was suggested to increase the level of total and antigen-specific IgE in serum by former studies, so that sensitization and symptoms should be studied separately. The high OR of residents in a business or light industrial area (5.00, 95% CI; 1.45-17.3) suggested an association with air pollution.(ABSTRACT TRUNCATED AT 250 WORDS) Risk factors for Japanese cedar pollinosis including past or family history of allergic diseases, smoking and passive smoking, dwelling conditions, and life events were analyzed by a case control method. Patients with Japanese cedar pollinosis (22 males and 67 females) were matched with a corresponding number of patients without potential symptoms of pollinosis according to sex and age (±5 years). The mean age was 39 years in both groups. The odds ratio (OR) was calculated by McNemar's method and the conditional logistic regression model. The design and methodology in this study were somewhat inadequate so that the validity of the results is limited. The most important problem was no-matching according to exposure to pollen. Significantly high OR for past history of allergic disease (8.80, 95% confidence interval (CI); 3.49-22.2), atopic sermatitis (9.00, 95% CI; 1.14-71.0), and a sibling history of allergic disease (3.25, 95% CI; 1.06-9.97) were consistent with former genetical studies. ORs were lower than unity for current smokers (0.36, 95% CI; 0.11-1.13) and those smoking 10 cigarettes/day or more (0.20, 95% CI; 0.04-0.91) relative to nonsmokers. The OR for passive smoking from 7-15 years of age as a result of the father's smoking habit (0.38, 95% CI; 0.17-0.86) was also significantly low. Smoking was suggested to increase the level of total and antigen-specific IgE in serum by former studies, so that sensitization and symptoms should be studied separately. The high OR of residents in a business or light industrial area (5.00, 95% CI; 1.45-17.3) suggested an association with air pollution. The findings such as a high OR for using a gas stove (3.52, 95% CI; 1.34-9.24), low OR for using an airconditioner (0.38, 95% CI; 0.16-0.87), and a not significant OR for other housing environments, were controversial by comparison with former studies of allergic responses to indoor antigens. “Moving” was the only item with a significantly high OR among the 37 life events. Therefore, it may represent an environmental change rather than mental stress. Risk factors for Japanese cedar pollinosis including past or family history of allergic diseases, smoking and passive smoking, dwelling conditions, and life events were analyzed by a case control method. Patients with Japanese cedar pollinosis (22 males and 67 females) were matched with a corresponding number of patients without potential symptoms of pollinosis according to sex and age (+/- 5 years). The mean age was 39 years in both groups. The odds ratio (OR) was calculated by McNemar's method and the conditional logistic regression model. The design and methodology in this study were somewhat inadequate so that the validity of the results is limited. The most important problem was no-matching according to exposure to pollen. Significantly high OR for past history of allergic disease (8.80, 95% confidence interval (CI); 3.49-22.2), atopic sermatitis (9.00, 95% CI; 1.14-71.0), and a sibling history of allergic disease (3.25, 95% CI; 1.06-9.97) were consistent with former genetical studies. ORs were lower than unity for current smokers (0.36, 95% CI; 0.11-1.13) and those smoking 10 cigarettes/day or more (0.20, 95% CI; 0.04-0.91) relative to nonsmokers. The OR for passive smoking from 7-15 years of age as a result of the father's smoking habit (0.38, 95% CI; 0.17-0.86) was also significantly low. Smoking was suggested to increase the level of total and antigen-specific IgE in serum by former studies, so that sensitization and symptoms should be studied separately. The high OR of residents in a business or light industrial area (5.00, 95% CI; 1.45-17.3) suggested an association with air pollution.(ABSTRACT TRUNCATED AT 250 WORDS)Risk factors for Japanese cedar pollinosis including past or family history of allergic diseases, smoking and passive smoking, dwelling conditions, and life events were analyzed by a case control method. Patients with Japanese cedar pollinosis (22 males and 67 females) were matched with a corresponding number of patients without potential symptoms of pollinosis according to sex and age (+/- 5 years). The mean age was 39 years in both groups. The odds ratio (OR) was calculated by McNemar's method and the conditional logistic regression model. The design and methodology in this study were somewhat inadequate so that the validity of the results is limited. The most important problem was no-matching according to exposure to pollen. Significantly high OR for past history of allergic disease (8.80, 95% confidence interval (CI); 3.49-22.2), atopic sermatitis (9.00, 95% CI; 1.14-71.0), and a sibling history of allergic disease (3.25, 95% CI; 1.06-9.97) were consistent with former genetical studies. ORs were lower than unity for current smokers (0.36, 95% CI; 0.11-1.13) and those smoking 10 cigarettes/day or more (0.20, 95% CI; 0.04-0.91) relative to nonsmokers. The OR for passive smoking from 7-15 years of age as a result of the father's smoking habit (0.38, 95% CI; 0.17-0.86) was also significantly low. Smoking was suggested to increase the level of total and antigen-specific IgE in serum by former studies, so that sensitization and symptoms should be studied separately. The high OR of residents in a business or light industrial area (5.00, 95% CI; 1.45-17.3) suggested an association with air pollution.(ABSTRACT TRUNCATED AT 250 WORDS) |
| Author | TAKENAKA, Hiroshi KAWAI, Keiichi OZASA, Kotaro AOIKE, Akira TAKAGI, Nobuo |
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| References | 30) 雨皿亮,鵜飼幸太郎,坂倉康夫:モルモット鼻粘膜非特異性過敏性および反応性に及ぼす上気道ウイルス(HVJ)感染の影響,アレルギー,37,140-146(1988). 36) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清ダニIgE抗体と居住環境との関係の研究(第2報)室内環境との関係について,日本公衛誌,33,3-11(1986). 12) Rothman, K. J.: Modern epidemiology, pp. 7-21, pp. 250-274, Little, Brown and Company, Boston (1986). 28) 中村晋:大学生の入学時と4年次における杉花粉症有病率の推移に関する調査成績,アレルギー,42,101-106(1993). 10) 奥田稔:鼻アレルギー(第2版),pp.138-208,金原出版,東京(1992). 37) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清総IgE抗体と暖房器具との関係について,日衛誌,41,593-600(1986). 19) 松下祥:花粉症の遺伝要因,Pharma Medica, 12, 19-23 (1994). 5) 井上栄,坂口雅弘,森田盛大,庄司俊雄,金田誠一,木村英二,山本保男,井上博雄,小野哲郎,道家直,平川浩資:一般住民のスギ花粉特異IgE抗体保有率の地域差,医学のあゆみ,145,121-122(1988). 25) Bloom, J. W., Halonen, M., Dunn, A. M., Pinnas, J. L. and Burrows, B.: Pneumococcus-specific immunoglobulin E in cigarette smokers, Clin. Allergy, 16, 25-32 (1986). 21) 宮崎恭一,野上浩志,大島明,中村正和:わが国の職場の分煙・禁煙状況,日本公衛誌,39(10,特別付録),44(1992). 8) Ishizaki, T., Koizumi, K., Ikemori, R., Ishiyama, Y. and Koshibiki, E: Studies of prevalence of Japanese cedar pollinosis among the residents in a densely cultivated area, Ann. Allergy, 58, 265-270 (1987). 29) 雨皿亮,鵜飼幸太郎,坂倉康夫:亜硫酸ガス暴露モルモット鼻粘膜のヒスタミンに対する過敏性および反応性の検討,アレルギー,37,40-46(1988). 14) SAS Institute Inc. SAS/STAT User's Guide, Version 6, Fourth edition, Volume 2, pp. 1122-1125, SAS Institute Inc., Cary (1990). 11) 宗像恒次:新版行動科学からみた健康と病気,p.9,メヂカルフレンド社,東京(1990). 6) 鈴木修二,千葉響子,玉川鐡雄,西田〓太郎,坂口雅弘,井上栄,宮本昭正:都内事業所3集団における抗スギ花粉IgE抗体値と花粉症症状,アレルギー,36,587(1987). 17) 小川保,石井譲治,三谷一憲,今井昌雄,今岡浩一,井上栄:スギ花粉症患者における血中特異抗体濃度の経時変化,Bull. Inst. Public Health, 41, 404-406 (1992). 26) Arshad, S. H. and Hide, D. W.: Effect of environmental factors on the development of allergic disorders in infancy, J. Allergy Clin. Immunol., 90, 235-241 (1992). 33) 高木学:小児期におけるスギ花粉感作の研究,第1編小児気管支喘息におけるスギ花粉感作に影響を及ぼす環境因子の検討,アレルギー,37,1065-1071(1988). 20) 厚生省編:喫煙と健康(第2版),pp.268-269,保健同人社,東京(1993). 13) Schlesselman, J. J.: Case-control studies-design, conduct, analysis, Oxford University Press, New York (1982).(重松逸造監訳:疫学・臨床医学のための患者対照研究,pp.198-202,ソフトサイエンス社,東京,1983). 16) 前田裕二,安枝浩,剱田幸子,信太隆夫:飛散アレルゲンの量とアレルギー患者の感作状態との関連について,アレルギー,37,919-925(1988). 1) 堀口申作,斉藤洋三:栃木県日光地方におけるスギ花粉症Japanese cedar pollinosisの発見,アレルギー,13,16-18(1964). 35) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清スギIgE抗体と居住環境との関係について,アレルギー,36,72-80(1987). 2) 奥田稔:鼻アレルギー(第2版),pp.113-137,金原出版,東京(1992). 31) 横山榮二,山内巌雄:自動車排出ガスの健康リスク,公衆衛生研究(Bull. Inst. Public Health), 41, 324-334 (1992). 34) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清ダニIgE抗体と居住環境との関係の研究(第1報)住宅環境との関係について,日本公衛誌,32,731-737(1985). 27) Ownby, D. R. and McCullough, J.: Passive exposure to cigarette smoke does not increase allergic sensitization in children, J. Allergy Clin. Immunol., 82, 634-638 (1988). 3) 中村晋:大学生並びに大学職員における杉花粉症の頻度調査成績,アレルギー,39,476-482(1990). 18) 白川太郎:アトピーの遺伝子,細胞工学,11,906-913(1992). 32) 石山康子,池森享介,小泉一弘,石崎達:大気汚染のスギ花粉症に及ぼす影響,環境別花粉数測定との関係,アレルギー,35,892(1986). 4) 斉藤洋三:花粉症治療の最近の話題,内科,59,677-681(1987). 7) 宮沢博,坂口雅弘,井上栄,鈴木修二:一般成人におけるスギ花粉特異的IgE抗体保有と症状との関係,アレルギー,38,890(1989). 15) 斉藤憲治:スギ花粉飛散と症状について,耳鼻臨,76,補冊(印刷中). 23) Ronchetti, R., Bonci, E., Cutrera, R., DeCastro, G., Indinnimeo, L., Midulla, F., Tancredi, G. and Martinez, F. D.: Enhanced allergic sensitisation related to parental smoking, Arch. Dis. Child, 67, 496-500 (1992). 22) 白川太郎,森本兼曩:ライフスタイルとアレルギー反応,(森本兼曩:ライフスタイルと健康),pp.83-100,医学書院,東京(1991). 24) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童の血清ダニIgEと母の喫煙習慣との関係の研究,日衛誌,40,1789-1794(1985). 9) 九嶋敦,大橋裕二,小泉一裕,井上栄,坂口雅弘:栃木県日光地区におけるスギ花粉症の出現頻度,アレルギー,36,588(1987). |
| References_xml | – reference: 13) Schlesselman, J. J.: Case-control studies-design, conduct, analysis, Oxford University Press, New York (1982).(重松逸造監訳:疫学・臨床医学のための患者対照研究,pp.198-202,ソフトサイエンス社,東京,1983). – reference: 32) 石山康子,池森享介,小泉一弘,石崎達:大気汚染のスギ花粉症に及ぼす影響,環境別花粉数測定との関係,アレルギー,35,892(1986). – reference: 4) 斉藤洋三:花粉症治療の最近の話題,内科,59,677-681(1987). – reference: 18) 白川太郎:アトピーの遺伝子,細胞工学,11,906-913(1992). – reference: 19) 松下祥:花粉症の遺伝要因,Pharma Medica, 12, 19-23 (1994). – reference: 8) Ishizaki, T., Koizumi, K., Ikemori, R., Ishiyama, Y. and Koshibiki, E: Studies of prevalence of Japanese cedar pollinosis among the residents in a densely cultivated area, Ann. Allergy, 58, 265-270 (1987). – reference: 21) 宮崎恭一,野上浩志,大島明,中村正和:わが国の職場の分煙・禁煙状況,日本公衛誌,39(10,特別付録),44(1992). – reference: 34) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清ダニIgE抗体と居住環境との関係の研究(第1報)住宅環境との関係について,日本公衛誌,32,731-737(1985). – reference: 35) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清スギIgE抗体と居住環境との関係について,アレルギー,36,72-80(1987). – reference: 27) Ownby, D. R. and McCullough, J.: Passive exposure to cigarette smoke does not increase allergic sensitization in children, J. Allergy Clin. Immunol., 82, 634-638 (1988). – reference: 6) 鈴木修二,千葉響子,玉川鐡雄,西田〓太郎,坂口雅弘,井上栄,宮本昭正:都内事業所3集団における抗スギ花粉IgE抗体値と花粉症症状,アレルギー,36,587(1987). – reference: 23) Ronchetti, R., Bonci, E., Cutrera, R., DeCastro, G., Indinnimeo, L., Midulla, F., Tancredi, G. and Martinez, F. D.: Enhanced allergic sensitisation related to parental smoking, Arch. Dis. Child, 67, 496-500 (1992). – reference: 25) Bloom, J. W., Halonen, M., Dunn, A. M., Pinnas, J. L. and Burrows, B.: Pneumococcus-specific immunoglobulin E in cigarette smokers, Clin. Allergy, 16, 25-32 (1986). – reference: 16) 前田裕二,安枝浩,剱田幸子,信太隆夫:飛散アレルゲンの量とアレルギー患者の感作状態との関連について,アレルギー,37,919-925(1988). – reference: 28) 中村晋:大学生の入学時と4年次における杉花粉症有病率の推移に関する調査成績,アレルギー,42,101-106(1993). – reference: 1) 堀口申作,斉藤洋三:栃木県日光地方におけるスギ花粉症Japanese cedar pollinosisの発見,アレルギー,13,16-18(1964). – reference: 26) Arshad, S. H. and Hide, D. W.: Effect of environmental factors on the development of allergic disorders in infancy, J. Allergy Clin. Immunol., 90, 235-241 (1992). – reference: 24) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童の血清ダニIgEと母の喫煙習慣との関係の研究,日衛誌,40,1789-1794(1985). – reference: 9) 九嶋敦,大橋裕二,小泉一裕,井上栄,坂口雅弘:栃木県日光地区におけるスギ花粉症の出現頻度,アレルギー,36,588(1987). – reference: 30) 雨皿亮,鵜飼幸太郎,坂倉康夫:モルモット鼻粘膜非特異性過敏性および反応性に及ぼす上気道ウイルス(HVJ)感染の影響,アレルギー,37,140-146(1988). – reference: 2) 奥田稔:鼻アレルギー(第2版),pp.113-137,金原出版,東京(1992). – reference: 5) 井上栄,坂口雅弘,森田盛大,庄司俊雄,金田誠一,木村英二,山本保男,井上博雄,小野哲郎,道家直,平川浩資:一般住民のスギ花粉特異IgE抗体保有率の地域差,医学のあゆみ,145,121-122(1988). – reference: 7) 宮沢博,坂口雅弘,井上栄,鈴木修二:一般成人におけるスギ花粉特異的IgE抗体保有と症状との関係,アレルギー,38,890(1989). – reference: 22) 白川太郎,森本兼曩:ライフスタイルとアレルギー反応,(森本兼曩:ライフスタイルと健康),pp.83-100,医学書院,東京(1991). – reference: 36) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清ダニIgE抗体と居住環境との関係の研究(第2報)室内環境との関係について,日本公衛誌,33,3-11(1986). – reference: 3) 中村晋:大学生並びに大学職員における杉花粉症の頻度調査成績,アレルギー,39,476-482(1990). – reference: 12) Rothman, K. J.: Modern epidemiology, pp. 7-21, pp. 250-274, Little, Brown and Company, Boston (1986). – reference: 33) 高木学:小児期におけるスギ花粉感作の研究,第1編小児気管支喘息におけるスギ花粉感作に影響を及ぼす環境因子の検討,アレルギー,37,1065-1071(1988). – reference: 20) 厚生省編:喫煙と健康(第2版),pp.268-269,保健同人社,東京(1993). – reference: 14) SAS Institute Inc. SAS/STAT User's Guide, Version 6, Fourth edition, Volume 2, pp. 1122-1125, SAS Institute Inc., Cary (1990). – reference: 29) 雨皿亮,鵜飼幸太郎,坂倉康夫:亜硫酸ガス暴露モルモット鼻粘膜のヒスタミンに対する過敏性および反応性の検討,アレルギー,37,40-46(1988). – reference: 17) 小川保,石井譲治,三谷一憲,今井昌雄,今岡浩一,井上栄:スギ花粉症患者における血中特異抗体濃度の経時変化,Bull. Inst. Public Health, 41, 404-406 (1992). – reference: 10) 奥田稔:鼻アレルギー(第2版),pp.138-208,金原出版,東京(1992). – reference: 11) 宗像恒次:新版行動科学からみた健康と病気,p.9,メヂカルフレンド社,東京(1990). – reference: 15) 斉藤憲治:スギ花粉飛散と症状について,耳鼻臨,76,補冊(印刷中). – reference: 37) 逢坂文夫,春日斉,杉田稔,松木秀明,三宅健:学童における血清総IgE抗体と暖房器具との関係について,日衛誌,41,593-600(1986). – reference: 31) 横山榮二,山内巌雄:自動車排出ガスの健康リスク,公衆衛生研究(Bull. Inst. Public Health), 41, 324-334 (1992). |
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| Title | A Case Control Study of Risk Factors for Japanese Cedar Pollinosis |
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