Higher dietary cholesterol and ω-3 fatty acid intakes are associated with a lower success rate of Helicobacter pylori eradication therapy in Japan

Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake...

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Published inThe American journal of clinical nutrition Vol. 106; no. 2; pp. 581 - 588
Main Authors Ikezaki, Hiroaki, Furusyo, Norihiro, Jacques, Paul F, Shimizu, Motohiro, Murata, Masayuki, Schaefer, Ernst J, Urita, Yoshihisa, Hayashi, Jun
Format Journal Article
LanguageEnglish
Published United States Elsevier Inc 01.08.2017
Oxford University Press
American Society for Clinical Nutrition, Inc
Subjects
Online AccessGet full text
ISSN0002-9165
1938-3207
1938-3207
DOI10.3945/ajcn.116.144873

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Abstract Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline. We investigated the influence of food and nutrient intake on H. pylori eradication therapy. In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to H. pylori (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a 13C-urea breath test (UBT). These 389 subjects received 1-wk H. pylori eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful H. pylori eradication therapy. The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age (P = 0.02), higher CRP concentrations (P < 0.01), higher dietary cholesterol (P < 0.01) or egg intake (P < 0.01), higher ω-3 (n–3) fatty acid (P = 0.02) or fish intake (P = 0.01), and higher vitamin D intake (P = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of H. pylori. Our results indicate that higher egg and fish intake may be negatively correlated with successful H. pylori eradication therapy in H. pylori–positive subjects with gastritis and/or duodenal ulcers.
AbstractList Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline.Objective: We investigated the influence of food and nutrient intake on H. pylori eradication therapy.Design: In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to H. pylori (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a 13C-urea breath test (UBT). These 389 subjects received 1-wk H. pylori eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful H. pylori eradication therapy.Results: The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age (P = 0.02), higher CRP concentrations (P < 0.01), higher dietary cholesterol (P < 0.01) or egg intake (P < 0.01), higher ω-3 (n-3) fatty acid (P = 0.02) or fish intake (P = 0.01), and higher vitamin D intake (P = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of H. pyloriConclusions: Our results indicate that higher egg and fish intake may be negatively correlated with successful H. pylori eradication therapy in H. pylori-positive subjects with gastritis and/or duodenal ulcers.Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline.Objective: We investigated the influence of food and nutrient intake on H. pylori eradication therapy.Design: In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to H. pylori (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a 13C-urea breath test (UBT). These 389 subjects received 1-wk H. pylori eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful H. pylori eradication therapy.Results: The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age (P = 0.02), higher CRP concentrations (P < 0.01), higher dietary cholesterol (P < 0.01) or egg intake (P < 0.01), higher ω-3 (n-3) fatty acid (P = 0.02) or fish intake (P = 0.01), and higher vitamin D intake (P = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of H. pyloriConclusions: Our results indicate that higher egg and fish intake may be negatively correlated with successful H. pylori eradication therapy in H. pylori-positive subjects with gastritis and/or duodenal ulcers.
Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline. We investigated the influence of food and nutrient intake on H. pylori eradication therapy. In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to H. pylori (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a 13C-urea breath test (UBT). These 389 subjects received 1-wk H. pylori eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful H. pylori eradication therapy. The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age (P = 0.02), higher CRP concentrations (P < 0.01), higher dietary cholesterol (P < 0.01) or egg intake (P < 0.01), higher ω-3 (n–3) fatty acid (P = 0.02) or fish intake (P = 0.01), and higher vitamin D intake (P = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of H. pylori. Our results indicate that higher egg and fish intake may be negatively correlated with successful H. pylori eradication therapy in H. pylori–positive subjects with gastritis and/or duodenal ulcers.
ABSTRACT Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline. Objective: We investigated the influence of food and nutrient intake on H. pylori eradication therapy. Design: In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to H. pylori (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a 13C-urea breath test (UBT). These 389 subjects received 1-wk H. pylori eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful H. pylori eradication therapy. Results: The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age (P = 0.02), higher CRP concentrations (P < 0.01), higher dietary cholesterol (P < 0.01) or egg intake (P < 0.01), higher ω-3 (n–3) fatty acid (P = 0.02) or fish intake (P = 0.01), and higher vitamin D intake (P = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of H. pylori. Conclusions: Our results indicate that higher egg and fish intake may be negatively correlated with successful H. pylori eradication therapy in H. pylori–positive subjects with gastritis and/or duodenal ulcers.
Background: Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline. Objective: We investigated the influence of food and nutrient intake on H. pylori eradication therapy. Design: In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to H. pylori (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a 13C-urea breath test (UBT). These 389 subjects received 1-wk H. pylori eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful H. pylori eradication therapy. Results: The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age (P = 0.02), higher CRP concentrations (P < 0.01), higher dietary cholesterol (P < 0.01) or egg intake (P < 0.01), higher ω-3 (n-3) fatty acid (P = 0.02) or fish intake (P = 0.01), and higher vitamin D intake (P = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of H. pylori. Conclusions: Our results indicate that higher egg and fish intake may be negatively correlated with successful H. pylori eradication therapy in H. pylori-positive subjects with gastritis and/or duodenal ulcers.
infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline. We investigated the influence of food and nutrient intake on eradication therapy. In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a C-urea breath test (UBT). These 389 subjects received 1-wk eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful eradication therapy. The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age ( = 0.02), higher CRP concentrations ( < 0.01), higher dietary cholesterol ( < 0.01) or egg intake ( < 0.01), higher ω-3 (n-3) fatty acid ( = 0.02) or fish intake ( = 0.01), and higher vitamin D intake ( = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of Our results indicate that higher egg and fish intake may be negatively correlated with successful eradication therapy in -positive subjects with gastritis and/or duodenal ulcers.
Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful in treating these disorders; however, the success rate of eradication therapy is declining. There may be an interaction with nutrient intake to account for this decline. Objective: We investigated the influence of food and nutrient intake on H. pylori eradication therapy. Design: In this study, 4014 subjects underwent endoscopy, were tested for serum antibodies to H. pylori (2046 positive; 51.0%), and had their food intake assessed with the use of a food-frequency questionnaire (FFQ). Of the positive subjects, endoscopies showed that 389 (19.0%) had gastritis and/or duodenal ulcers and were also positive for a ¹³C-urea breath test (UBT). These 389 subjects received 1-wk H. pylori eradication therapy with lansoprazole, amoxicillin, and clarithromycin and a second UBT 8 wk after treatment. Complete demographic characteristics, serum lipid, insulin, glycated hemoglobin, C-reactive protein (CRP), and creatinine concentrations as well as complete FFQs were available for 352 subjects. Multivariate logistic regression analyses were performed to determine factors that were associated with successful H. pylori eradication therapy. Results: The success rate of eradication therapy was 60.4% (235 of 389). Factors associated with the failure of eradication therapy included increased age (P = 0.02), higher CRP concentrations (P < 0.01), higher dietary cholesterol (P < 0.01) or egg intake (P < 0.01), higher ω-3 (n–3) fatty acid (P = 0.02) or fish intake (P = 0.01), and higher vitamin D intake (P = 0.02). Moreover, the higher vitamin D intake was strongly linked to higher fish intake. A limitation of the study is that we did not assess the antibiotic resistance of H. pylori. Conclusions: Our results indicate that higher egg and fish intake may be negatively correlated with successful H. pylori eradication therapy in H. pylori–positive subjects with gastritis and/or duodenal ulcers.
Author Ikezaki, Hiroaki
Urita, Yoshihisa
Schaefer, Ernst J
Shimizu, Motohiro
Jacques, Paul F
Hayashi, Jun
Furusyo, Norihiro
Murata, Masayuki
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  givenname: Hiroaki
  surname: Ikezaki
  fullname: Ikezaki, Hiroaki
  email: hiroaki.ikezaki@tufts.edu
  organization: Department of General Internal Medicine, Kyushu University Hospital, Fukuoka, Japan
– sequence: 2
  givenname: Norihiro
  surname: Furusyo
  fullname: Furusyo, Norihiro
  organization: Department of General Internal Medicine, Kyushu University Hospital, Fukuoka, Japan
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  givenname: Paul F
  surname: Jacques
  fullname: Jacques, Paul F
  organization: Nutritional Epidemiology Program
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  surname: Shimizu
  fullname: Shimizu, Motohiro
  organization: Department of General Internal Medicine, Kyushu University Hospital, Fukuoka, Japan
– sequence: 5
  givenname: Masayuki
  surname: Murata
  fullname: Murata, Masayuki
  organization: Department of General Internal Medicine, Kyushu University Hospital, Fukuoka, Japan
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  givenname: Ernst J
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  fullname: Schaefer, Ernst J
  organization: Cardiovascular Nutrition Laboratory, Human Nutrition Research Center on Aging at Tufts University and Tufts University School of Medicine, Boston, MA
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  givenname: Yoshihisa
  surname: Urita
  fullname: Urita, Yoshihisa
  organization: Department of General Medicine and Emergency Care, Toho University School of Medicine Omori Hospital, Tokyo, Japan
– sequence: 8
  givenname: Jun
  surname: Hayashi
  fullname: Hayashi, Jun
  organization: Department of General Internal Medicine, Kyushu University Hospital, Fukuoka, Japan
BackLink https://www.ncbi.nlm.nih.gov/pubmed/28659302$$D View this record in MEDLINE/PubMed
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Copyright 2017 American Society for Nutrition.
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ID FETCH-LOGICAL-c2882-f5d63d4ee969b5d4bf52f5ab1d28653ea12b99d57979c9117a1d2ac1c373c0563
ISSN 0002-9165
1938-3207
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Sun Sep 28 07:36:19 EDT 2025
Mon Oct 06 17:59:03 EDT 2025
Wed Feb 19 02:25:36 EST 2025
Wed Oct 01 04:43:18 EDT 2025
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Wed Sep 11 04:48:15 EDT 2024
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IsDoiOpenAccess true
IsOpenAccess true
IsPeerReviewed true
IsScholarly true
Issue 2
Keywords CRP
FFQ
Helicobacter pylori
KOPS
eradication therapy
omega 3 fatty acids
UBT
PPI
cholesterol
HUFA
dietary factors
Language English
License http://www.elsevier.com/open-access/userlicense/1.0
2017 American Society for Nutrition.
LinkModel OpenURL
MergedId FETCHMERGED-LOGICAL-c2882-f5d63d4ee969b5d4bf52f5ab1d28653ea12b99d57979c9117a1d2ac1c373c0563
Notes ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
content type line 14
content type line 23
OpenAccessLink https://dx.doi.org/10.3945/ajcn.116.144873
PMID 28659302
PQID 1963430848
PQPubID 41076
PageCount 8
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PublicationDate August 2017
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  text: August 2017
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PublicationTitle The American journal of clinical nutrition
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American Society for Clinical Nutrition, Inc
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Snippet Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful...
ABSTRACT Background:Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is...
infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of is successful in treating these disorders; however, the...
Background: Helicobacter pylori infection is a known risk factor for duodenal ulcers, gastritis, and gastric cancer. The eradication of H. pylori is successful...
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SubjectTerms Age Factors
Aged
Amoxicillin
Animals
Anti-Bacterial Agents - pharmacology
Anti-Bacterial Agents - therapeutic use
Antibiotic resistance
Antibiotics
Antibodies
blood lipids
blood serum
breath tests
C-reactive protein
C-Reactive Protein - metabolism
Cholesterol
Cholesterol, Dietary - administration & dosage
Cholesterol, Dietary - adverse effects
Clarithromycin
Clinical outcomes
Creatinine
Demographics
demography
Diet
dietary factors
Duodenal Ulcer - drug therapy
Duodenal Ulcer - microbiology
duodenal ulcers
Eggs
Endoscopy
Eradication
eradication therapy
Fatty acids
Fatty Acids, Omega-3 - administration & dosage
Fatty Acids, Omega-3 - adverse effects
Feeding Behavior
Female
Fish
fish consumption
Fishes
Food
food frequency questionnaires
Food intake
Gastric cancer
Gastritis
Gastritis - drug therapy
Gastritis - microbiology
glycohemoglobin
Gram-negative bacteria
Helicobacter Infections - drug therapy
Helicobacter Infections - microbiology
Helicobacter pylori
Helicobacter pylori - drug effects
Hemoglobin
Humans
Insulin
Japan
lansoprazole
Male
Middle Aged
nutrient intake
Nutrients
omega 3 fatty acids
Regression analysis
Risk factors
Seafood
stomach neoplasms
Therapy
Treatment Failure
Ulcers
Urea
Vitamin D
Vitamin D - administration & dosage
Title Higher dietary cholesterol and ω-3 fatty acid intakes are associated with a lower success rate of Helicobacter pylori eradication therapy in Japan
URI https://dx.doi.org/10.3945/ajcn.116.144873
https://www.ncbi.nlm.nih.gov/pubmed/28659302
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https://www.proquest.com/docview/2176347509
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