Kir6.1‐dependent K ATP channels in lymphatic smooth muscle and vessel dysfunction in mice with Kir6.1 gain‐of‐function - Davis, Michael J.; Kim, Hae Jin; Zawieja, Scott D.; Castorena‐Gonzalez, Jorge A.; Gui, Peichun; Li, Min; Saunders, Brian T.; Zinselmeyer, Bernd H.; Randolph, Gwendalyn J.; Remedi, Maria S.; Nichols, Colin G. (The Journal of physiology, 2020)
Keywords: Adenosine Triphosphate; Animals; Gain of Function Mutation; Humans; Hypertrichosis; KATP Channels - genetics; Mice; Mice, Inbred C57BL; Muscle, Smooth; Sulfonylurea Receptors - genetics; cantú syndrome; lymphatic dysfunction; pressure myography; electrophysiology; lymphedema