Mapping Lung Cancer Epithelial-Mesenchymal Transition States and Trajectories with Single-Cell Resolution

Elucidating a continuum of epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) states in clinical samples promises new insights in cancer progression and drug response. Using mass cytometry time-course analysis, we resolve lung cancer EMT states through TGFβ-treatment...

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Main Authors Karacosta, Loukia G, Benedict Anchang, Ignatiadis, Nikolaos, Kimmey, Samuel C, Benson, Jalen A, Shrager, Joseph B, Tibshirani, Robert, Bendall, Sean C, Plevritis, Sylvia K
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Published Cold Spring Harbor Cold Spring Harbor Laboratory Press 07.03.2019
Cold Spring Harbor Laboratory
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ISSN2692-8205
2692-8205
DOI10.1101/570341

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Abstract Elucidating a continuum of epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) states in clinical samples promises new insights in cancer progression and drug response. Using mass cytometry time-course analysis, we resolve lung cancer EMT states through TGFβ-treatment and identify through TGFβ-withdrawal, an MET state previously unrealized. We demonstrate significant differences between EMT and MET trajectories using a novel computational tool (TRACER) for reconstructing trajectories between cell states. Additionally, we construct a lung cancer reference map of EMT and MET states referred to as the EMT-MET STAte MaP (STAMP). Using a neural net algorithm, we project clinical samples onto the EMT-MET STAMP to characterize their phenotypic profile with single-cell resolution in terms of our in vitro EMT-MET analysis. In summary, we provide a framework that can be extended to phenotypically characterize clinical samples in the context of in vitro studies showing differential EMT-MET traits related to metastasis and drug sensitivity.
AbstractList Elucidating a continuum of epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) states in clinical samples promises new insights in cancer progression and drug response. Using mass cytometry time-course analysis, we resolve lung cancer EMT states through TGFβ-treatment and identify through TGFβ-withdrawal, an MET state previously unrealized. We demonstrate significant differences between EMT and MET trajectories using a novel computational tool (TRACER) for reconstructing trajectories between cell states. Additionally, we construct a lung cancer reference map of EMT and MET states referred to as the EMT-MET STAte MaP (STAMP). Using a neural net algorithm, we project clinical samples onto the EMT-MET STAMP to characterize their phenotypic profile with single-cell resolution in terms of our in vitro EMT-MET analysis. In summary, we provide a framework that can be extended to phenotypically characterize clinical samples in the context of in vitro studies showing differential EMT-MET traits related to metastasis and drug sensitivity.
Author Benedict Anchang
Bendall, Sean C
Karacosta, Loukia G
Benson, Jalen A
Shrager, Joseph B
Tibshirani, Robert
Ignatiadis, Nikolaos
Kimmey, Samuel C
Plevritis, Sylvia K
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Cites_doi 10.1007/11561071_34
10.1158/1078-0432.CCR-15-0876
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Snippet Elucidating a continuum of epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) states in clinical samples promises new insights...
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SubjectTerms Computer applications
Cytometry
Lung cancer
Mesenchyme
Metastases
Systems Biology
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Title Mapping Lung Cancer Epithelial-Mesenchymal Transition States and Trajectories with Single-Cell Resolution
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https://www.biorxiv.org/content/10.1101/570341
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